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PharmFin

Cardiac Glycosides

QuestionAnswer
Digitalis: Mechanism of action Receptor is membrane bound ATP-ase; inhibition of NA-K pump leads to inc. movement of Ca++ into cells or freeing of intracellularly bound Ca++
Effects of Digitalis? 1. + inotropic effect (inc. contractility)2. - chronotropic effect (dec. HT rate)3. AV conduction slowed, partially blocked due to refractory period (no depolarization)4. Diuresis (indirect effect), primarily a result of more blood going to the KD
Toxicity of Digitalis? flickering and greenish - yellow vision
Effects of Digitalis on the HT? Enhanced hypokalemia and counteracted by hyperkalemia
Side effects of Digitalis Early = anorexia (first sign)Moderate = nausea, fatigue, vision disturbancesSevere = diarrhea, arrhythmia
Examples of Cardiac Glycosides: Digoxin, Digitalis, Lanoxin
What do ACE Inhibitors do? Lower blood pressure by blocking the production of angiotensin II
What are some examples of ACE Inhibitors? Catopril, Enalpril
Therapeutic uses of ACE Inhibitors? Hypertension following MI and CHF
Adverse reactions of ACEs? Cough, HA, rash, hyperkalemia, hypotension
Contraindications of ACEs? People who are allergic to Sulfonamides and women in their 2nd and 3rd Tri.
What is the mechanism of action for Quinidine/Norpace? Decreases permeability of Na+, thus slows down depolarization and repolarization; also increases intracellular K+
Name some antidysrhythmic drugs? Quinidine, Lidocaine, Toprol, Verapamil
Name two therapeutic uses for Quinidine? Atrial fibrillation and tachycardia
What are the toxic effects of Quinidine? Hypotension, fibrillation, tinnitus due to vasoconstriction, blurred vision, vertigo, tremors
What are the effects of Toprol? Negative chronotropic and inotropic
Created by: minggeekmen
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