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Cirrhosis Drugs

QuestionAnswer
non-selective ß-blocker causes splanchnic vasoconstriction nadolol
decreases intestinal NH3 by acting against urease producing bacteria neomycin sulfate
somatostatin analog; inhibits serotonin, gastrin, insulin, glucagon and alters mesenteric/sphlanchnic blood flow promoting hemostasis octreotide
inhibits binding of aldosterone to receptor spironolactone
inhibits sodium-potassium symport in ascending limb furosemide
used in cirrhosis induced hepatic encephalopathy neomycin sulfate
used for bleeding esophageal varicies octreotide and nadolol
used for cirrhosis induced edema spironolactone and furosemide
causes nephro and ototoxicity neomycin sulfate
causes gynecomastia spironolactone
monitor B12 levels, cholelithiasis, hypothyroidism, anemia octreotide
avoid NSAIDs spironolactone and furosemide
aides in hydrolysis of fats, proteins and starches pancrelipase
interacts with iron salts, iron, multivitamins pancrelipase
monitor growth and development pancrelipase
titrate "low and go slow" pancrelipase
how do you monitor the effectivity of neomycin sulfate? clinical improvement
used after removal of >5L paracentesis albumin 25%
how long for diuretics to reach "steady state"? and how often should you adjust dose? 3-5 days
don't use with DM or renal impairment octreotide
which 2 cirrhosis drugs are pricy? octreotide and albumin 25%
Created by: duanea00
 

 



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