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ans comparison
| Question | Answer |
|---|---|
| neurotransmitter for PSN | Acetylcholine |
| adrenergic receptors | Alpha, Beta 1, Beta 2 |
| neurotransmitter for SNS | Epinephrine, Norepinephrine |
| another term for "dilate" | mydriasis |
| how does the alpha 1 receptor affect the bladder | contract |
| how does the alpha 2 receptor affect the blood vessels | decrease blood pressure, reduce norepinephrine |
| how does the beta 2 receptor affect the lungs | bronchodilation |
| how does the beta 1 receptor affect the heart | increase heart rate, increase heart contraction |
| cholinergic receptors | muscarinic, nicotinic |
| nicotinic receptors | affect skeletal muscles |
| muscarinic receptors | stimulate smooth muscle, slow heart rate |
| what is the neurotransmitter located at the ganglions and the parasympathetic terminal nerve endings that innervates the receptors in organs, tissues, and glands | acetylcholine |
| stimulation of the PSN or parasympathomimetic drugs have what affect on the pupils and bronchioles | constriction |
| how does cholinergic activity affect GI motility | increase |
| how does adrenergic activity affect the bladder | relaxes |
| how does cholinergic activity affect the urinary sphincter | relaxes |
| by blocking the activity in what receptor group would treat urinary incontinence | cholinergic |
| site of stimulation for beta 1 receptor | cardiac muscle |
| site of stimulation for beta 2 receptor | pulmonary muscle, uterine smooth muscle |
| site of stimulation for alpha receptor | arteries, veins, pupils |
| cholinergic blocker side effects | increased heart rate, vision disturbances, dry mouth, constipation, urinary retention, sedation |
| alpha-adrenergic blocker side effects | cardiac sysrhythmias, flush, hypotension, reflex tachycardia |
| beta blocker side effects | bradycardia, dizziness, hypotension, headache, hyperglycemia |
| muscarinic agonists side effects | hypotension, bradycardia, excessive salivation, diarrhea, bronchoconstriction |
| enzymes that inactivate the metabolism of norepinephrine | monoamine oxidase, catechol-O-methyltransferase |
| what must be done after the transmitter (norepinephrine) has performed its function | action must be stopped to prevent prolonging the effect |