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OT pediatrics

Childhood diseases

Childhood diseases123456
Cerebral Palsy Non progressive damage to motor portion of brain resulting in tone disorder and affecting movement and onset is before during or after birth up to 3 years not genetic (caused by tetratogens (7-8 months) and other factors) Hypertonic( spastic or rigid), Hypotonia (flopp), Athetosis (mixxed or fluctuationg tone), Ataxia (in normal to low tone) Limited ability to initiate pr move through full ROM,poor ability to control co contraction, (plegias) contractures, postura reflex mechanisms, deficits in all performance areas because of tone, 40% noral intelligence, if not walking by 6 then wont be neurodevelopment treatments, biomechanical, adaptation includes splinting, bracing, and technological devices. , life span around 40 years, 10% of adults entirely self supporting,
Spina Bifida Neural tube birth defect of the backbone and sometimes spinal chord, congenital thought to have genetic gfactor, folic acid reduces risk, Occulta, Meningocele, Myelomeningocele, (cyst hold nerev of spinal chord back needs to be surgically closed, hydrocephalus, paralysis below level of defect, musculoskeletal deformities, bowel and bladder dysfunction visospatial deficits, memory deficits, coctail party speech, 85% survive to adulthood
Muscular dystrophy Genetic resulting in flaws in muscle protien gene. -genetic diseases marked by progressive wekness and degeneration of the skeletal, voluntary muscles that control movement.(most common is Duchenne Myotonic (c19) muscle weakness, cns, (congenital-elayed sucking,swallowing,breathing motor and MR) duchenne-2-6 yrs onset males only (x linked)-starts with frequent falling, can't get up, gowers sighn, some have MR, life span only to 20 death from pneumonia, respiratory failure, cardiac problems (blank) (blank)
spinal chord injury damage to spinal chord that results in loss of function such as mobility or feeling 10,000 incidence a year moslty due to car accidents and violence high tetraplegia c1-t4/ low tetraplegia c5-c7/ paraplegia-lumbar or sacral below t10 high tetraplegia- no function below level of injury, c1-c3 use ventilator/low tetraplegia-c5-shoulder and bicepts but no wrist use, c6- wrist control but no hand use, c7t1-dexterity problems and no function below this spinal level/hand not affec at lumbar c7-independent with adaptations--leading cause of death is pnemonia, pulmonary emboli, septicema ((paralysis and loss ofo sensation below level of leision
juvenile rhumatoid arthritis inflammation involving the joints that may last for a minimum of 6= consecutive weeks( thought to be an autoimmune disease) (blank) polyartricular (5 or more joints- female)painful swelling/Systemic JRa associated with fever and anemia/spondylo arthropathy(males)-early assymetrical involvement of large joints avoid moving, joint contractures, tense rigid, tutoring, (blank) splinting during flare ups,excersize and prevent deformity, joint protection
prematurity small for gestation agebron before 37 weeks, one month or more early multiple fetuses, teenage pregnancy, smoking, diabetes, infections, 11% of all births are preme (blank) child appears apathetic, periods of apnea, waxy covering and fine body hair,not enough strength for feeding (PVL)(periventricular leukomalacia), infection, bleeding, hemorhage, jaundice high chance of LD
osteogenisis imperfecta genetic deficit in which bones break easy often from little or no apperent cause type 1 most common , mildest, type 2 most sever, type 3collagen improperly formed, type 4 3-1 no cure, develop optimum bone mass and muscle strength, adaptive equipment, excersize-especially swimming (blank) (blank) (blank)
arthrogryphosis non progessive contracture of 2 or more joints at birth(rare)more common in males limited movement in uterou may cause abnormal dev of tendons bones and joints amyoplasia ( abnormal dev of muscle fibers)/ Cns and spinal chord can be malformed/can be genetic syndrome limb muscle replaced by fibrous or fatty tissue/multiple joint contractures/normal intelligence (blank) wrst with cns involvement best is amyplasia// Splinting ROM compensation and adaptation
fragile X syndrome more males than females hypotonia, poor eye contact, speech is persverative echolailic, Iq usually moderate but females can be svere like autistic adhd agressive anxiety and unstable mood (blank) (blank) (blank)
retts syndrome (blank) (blank) (blank) (blank) (blank) (blank)
prader-willi hypotonia as baby, failure to thrive infancy mild to moderate MR temper tantrums and other behavior problems, shrt stature, apnea, high pain threshhold, obesity is major cause of morbidity and mortality (blank)
angelman (blank) (blank) (blank) (blank) (blank) (blank)
fetal alcohol (blank) (blank) (blank) (blank) (blank) (blank)
down syndrome related to trisomy 21 gene low muscle tone/ joint hyperflexibility need to be taught stranger awareness--iq (50-60) mild-55-60- academics grade 3-7/moderate -40-54 unlikely to pass 2nd grade/severe-25-39 basic habit training/prfound below 25-caregiver needd for survival heart risks and resperatory problems high risk for alzheimers
MR not diagnosed after age 18/significantly sub average intellectual function- (IQ under 70-75) (blank) (blank) (blank) (blank) (blank)
Created by: natkat on 2005-03-20



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