4120- Arthritis Word Scramble

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Question	Answer
what is arthritis	a general term for conditions of joint flammation, 1+ joints
how many forms of arthritis exist	100+
what are the diff types of arthritis classified on	degree of joint damage, restriction of movement, functional limitation, pain
3 main classifications of arthritis	osteoarthritis rheumatoid arthritis ankylosing spondylitis
what is the number one cause of disability	arthritis
how does arthritis affect social funcitoning	increased isolation, stress, depression because of pain, leads to decreased QOL
what is osteoarthritis	hands, feet, spine, and weight bearing joints have issues
secondary affect of osteoarthritis	decreased strength due to decreased movement
pathophysiology of osteoarthritis	constant remodeling of joint tissues in abnormal ways
what is periarticular atrophy	muscle atrophy around joints
what happens to cartilage in osteoarthritis	decreased cartilage, leads to bone on bone rubbing and inflamation, strained ligmanets, weak and pain
steps of osteoarthritis degradation	rough, brittle cartilage damage bone thickens to decrease load synovial swelling fluid increase ligaments thicken, bad movement decrease joint space loss of cartilage bc bone on bone weakens ligaments
what is rheumatoid arthritis	a chronic autoimmune form
which gender is RA more common in	women
how is RA infllamation different	systemic inflammation instead of localized to joints
symmetrical polyarthritis	multiple joints, both sides of the body, min 4
main type of joint/issue with RA	synovial joints, synovitis
common comorbities/risks of RA	muscle atrophy, increased fat mass, fatigue, cv/m disease, t2d, osteoporosis
pathophysio of RA	immune dysfunction begins attack synovitis pannus, esp w synovial hyperplasia cartilage and bones erode joint detruction/ankylosis (stiffness)
two causes of synovitis	excess fluid synovial cell hyperplasia
what is pannus	abnormal tissue layers over joints
anklyosis	when the joints become stiff and narrow so movement is restricted
which type is more daily wear and tear	OA
which type is asymmetric	OA
which type occurs at younger ages	RA
which type occurs more at smaller joints	rA
which type has obvious inflammation	Ra
which type does not normally need medical treatment besides pain pills	OA
ankylosing spondylitis	chronic autoimmune disorder
which gender is AS more common in	men
which joints is AS more common at	spine, sacroiliac, cervical, peripheral joints
pathophysio of AS	autoimmune response attack lower spine ligaments become inflamed bone growths and bone spurs form IN ligaments as response vertebrae bridges/fusion lower back pain, immobility, bad posture
where do bone spurs form in AS	in the ligament
4 steps of AS response	inflammation erosion syndesmophytes fusion
syndesmophytes	bony outgrowths on spinal ligaments
secondary effects of AS	decreased: ex tolerance strength aerobic capacity ROM biomechanic efficency proprioreception social functioning
3 stages of arthritis	acute/mind chronic/moderate chronic+acute exacerbation of joint symptoms/severe
acute arthritis	reversible s/s @ joints, especially decrease synovitis
chronic/moderate arthritis	stable s/s but structural damage is irreversible
severe arthritis	increase pain, decreased rom and function, increased inflammation
s/s/ of affected arthritic joints	pain stiffness joint locking deformity synovitis creptius effusion bone spurs
effusion	fluid around joints
crepitus	popping and cracking noises
how can extent and severity affect testing	ROM, alignment, function, pain mays be impaired
functional levels can affect _____	interventions
what sign can help diagnose arthritis	extra articular formations
common s/s that may display	redness, swelling, pain, heat around inflammed joint
what is the diagnostic for arthritis	there is no definitive test or marker
two useful screening tools for arthritis	serum/synovial tests joint imaging/mri/ultrasounds to see degradation and abnormalities
why would we need ex testing	may be needed for other risks assessments
which type has higher comorbidity (especially cv) risk	RA
what main symptom increases risk	inactivity
what should we use to form baseline for change and guide Rx	muscular skeletal and ROM testing
how should intensity be adjusted for rx	small increases
how should modes be decided	based on ability level
what stage should use treadmill	minimal/mild
what stage should use cycle ergo	mild/moderate in lower extremeties
what stage should use arm ergo	severe in lower extremities
how does ex benefit arthritic indiviuals	control progression minimize symptoms
4 main arthritis treatment goals	decrease inactivity manage s/s/improve adl and qol restore/maintain body comp (some may accumate fat mass from inactivity) decrease comorbidities, symptoms, risks
non drug treatments for arthritis	education canes PT/OT shoe mods (orthotics) BRACES/BANDAGES ice/heat decrease weight to decrease load no reptitive motion jobs joint irrigation/surgery
what is joint irrigation	flush the joint to remove debris
common drug treatments for arthritis	nsaids, opiod pain relievers (anagesics), corticiosteroids (anti inflammatory), DMARDs
what are DMARDs	disease modifying artirheumatic drugs used to treat RA and ankylysing spondylosis
goals of ex rx with arthritis	increase physical function better body comp decreased bw decrease inflammation pain and stiffness prevent deformities and contractures
what is a contracture	permanent shortening and tightening of joints that causes movement to be stuck
what is the most physically limited clinical population	arthritis
6 special considerations for arthritis	avoid high impact morning stiffness/cold Cl in water therapy footwear ankylos spondylitis posture/back brace corticosteroid degradation
high impact ex and arthritis	avoid it, prevent injury and may have had previous replacement
why underwater therapy is beneficial	increased buoyancy decreases joint pressure
chlorine and underwater therapy considerations	Cl can increase rashes and redness w/inflammed joints, may cause respiratory issues
orthotics	inserts and supports in the shoe to help w shock support and ADL pain
how do corticosteroids affect long term	bone loss and atrophy due to cortsiol mimicking which is bad long term
KAATSU study main take away	low load less joint stress but still ex. benefits blood flow restriction lead to decreased inflammation by increasing post ex bfloww to better remove and filter inflammatants

Created by: isabellamulet