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harris final

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Question
Answer
Intoeing gait is caused from   metatarsus adductus (foot deformity) internal tibila torsion ( rotational deformity) excessive femoral anteversion ( rot deformity)  
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roational profile for exam of intoeing gait   foot progression angle evaluation of foot prone hip rot thigh foot rot (angle)  
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morphologci features of MTA   adducted forefoot concave med foot border convex lat foot border  
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MTA coption orrectable tx   stretching reverse last shoes/straight last shoes education  
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MTA not correctable passively tx option   stetching casting reverse last shoes surgical release  
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MTA Surgical options   release adductor hallucis mdial release tarsometrsal capsulotomies multiple metatsal osteomies lateral closing cuboid osteomis opeing cuneform osteotomy  
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Internal Tibial Tosion tx   NONE rotational tibial osteotomy if >10yrs old medial thigh foot angle >10 degrees  
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Excessive femoral anteversion   medial foot progression angle medial hip rot > 70 degrees most resolve by ten yrs of age  
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tx for excessive femoral anteversion   none rotational femoral osteotomy .>10 yrs 80 degrees of prone medial hip rotation  
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genu varum   physilogic bow legs Blount's Disease  
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Genu Valgum   knock knees  
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nomal knee alignment at birth   10-15 degrees varus  
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normal knee alginment at 18 months   neutral  
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30-36 months   genu valgum (outgrow in another 18 months part of normal growth  
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Physiologic bowlegs (varum)   bilateral genu varum in child greater than 2 associated ITT natural hx (spontaneous resolution)  
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tx for physiolocal bow leg   none  
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Tibial vara (BLOUNTs disease   proressive varus and ITT defomity of the proximal tibia  
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etiology of Blount's disease   pathologic compression on the proximal medial tibial results in abnormal growth and development of proximal medial epiphis and pramture physeal closure  
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who get blount's disease   fmale>males overwight children early walkers african americans  
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Knocked knees   genu valgum  
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types of knock knees   physiologic metabolic skeltal dyplasia post traumatic (Cozens adolescence  
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physicologic gen valgum   normal development 3-5 ys old child  
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metabolic knocked knees   x-linked hypophosphatemia renal osteodystophy  
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genu valgu post traumatic (cozen)   s/p proximal tibial metphsis overgorwoth with valgus deformity increasing leg length excellent remoldeling potential  
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genu valgum adolescence   10-11 yr old females 12-13 males knee pain (medial) large stature  
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genu valgum tx   hemiephysiodesis guided growth stapling and 8-plating  
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common foot deformities   calcaneovalgus talipes equinovarus pes planus toe walking  
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calcaneovalgus   present at birth characterized by forefoot valgus and heel df allowing the dorsal suface to touch the anterior tibia  
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etiology fo calcaniovalgus   inuterine molding  
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tx for calcaneovalgus   observation passive stretching seral casting (rarely) rule out tibial bowing  
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talipes equinovarus   clubfoot: present at birth forefoot adduction and supinated heel pf  
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etilogy for clubfoot   multifactoral interuterine  
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morphologic features   dimples oever talus forefoot adduction heel vaur ankle equinus shorter extremitiy smaller calf short smaller foot  
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clinical feature of club foot   cave principle cavus adductus varus equinus  
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tx for club foot   treatable but no correctable nonsurgical-->ponseti method (manipulation serial casting 3 months holding splints )  
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treatment results of clubfoot   abnomal foot decreased size calf atrophy decreased motion pes planus MTA flat top of talus dorsal cubluxation of forefoot  
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treatment for pes plantus   none  
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toe walking causes   idiopathic cp myelodysplaia muscular dystrophy  
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toe walking   bilateral always contracture develops limits df  
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idopathic toe walking clincal feature   cariable toe walking altered choe wear decreased df normal neuro exam  
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ideopathic toe walking rx   non operative ( stretching casting bracing and botox reoccurence common) operative achilles tendon lengthening decreased strength reoccurance rare  
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common hip conditons   transient synovitis and septic arthritis developmetnal dysplasia legg clave perthes slipped capital femoral epiphysis  
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transient synovitis and septic arthritis   pain decreased ROM refusal to bear weight  
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transient synovitis   age 3-8 no/low fever no systemic illness inflammatory  
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septic arthritis   ant greater then 2 fever system ilness infectious  
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tx for transent synovitis   NSAID  
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septic arthrits tx   I & D (incision and drainage) sensitive antibiotic  
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Developmental hip dysplasia   wasterbasket term encompassing all forms of hip instability including subluxation and dilocation  
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developemental hip dysplasia incidence   incidence; 1-6 in 100 births 40-60 in left hip 20 bilateral famales> males  
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developmentall hip dysplasia risk factors   first born female positive familiy hx breech presentatino musculoskeletal abnomality  
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etilogy of developmetn hip dysplasia   multifactorial (genetic disorder, hormonal factors and mechanical factors  
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dx fo hip dysplasia   physical is more important thanradological exam dynamic us  
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DHD physical exam   ortolani barlow galeazzi  
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ortonlani   reduces a dislocated hip  
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barlow   dilocated hip  
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galeazzi   leg length shot leg is dislocated  
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tx for hip dysplasia birth to 6 months   birth to 6 months pavik harness  
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DHD tx 6-18 months   limited abduction galeazzi test telescoping true shortenign gait abnomalities  
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complication with DHD   avascualr necrosis with Leg length discrepancy coxa brevis coxa valga DJD  
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legg clve perthes disease   condition of the immature hip caused by necrosis of al or part of the femoral head which subsequently deforms as necrotic bone is replaced by living bone  
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stage of leff calve perthes   necrosis 5-7 months fragmetnation 7-months reossification 20-38 months remolding ( until maturity  
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clinical feature of legg perthes   painful limp groin pain anteriomedial thigh and knee waddling gait atrophic extremtiy decreased ROM ( mid flexion contracture) limited internal rotation and abduction  
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tx for Legg calve perthes   mainatin ROM head contaitnment (abd orthosis or maybe femoral pelvic osteotomy  
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exercises for legg calve perthes   butterfly knee to chest standing straddle hip extension prone  
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prognosis for legg calve perthes   femoral head deformity jt incongruity impingement 86 percent have symptom of DJD before the age of 65  
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slipped capital femoral epiphysis (SCFE)   posterior and medial displacement of the  
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