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Burns 7-21-11

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Question
Answer
Burns are direct contact with or exposure to any.   Thermal, Chemical, Electrical or Radiation  
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Severity Of Burns   Risk of Infection, Mortality, And cosmetic or functional disability.  
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Factors that Influence Severity   depth, %TBSA, Location, age, general Health, Mechanism of injury  
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Burn Size   %TBSA, est. helps predict the severity of physiological response  
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Rule of Nines   (burn size) divides body into 11 segments.. Each 9% and 1% for genitals  
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Lund & Browder   Standard table based on the parts of body burned according to age  
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Incidence   Males more than females, except of 70 years  
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Thermal   Flames, Hot liquids, Steams semisolid, hot objects.  
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Chemical   contact with or ingestion, inhalation or injection  
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electrical   electrical energy as it passes through the body  
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radiation   least common, caused by radioactive source  
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Risk Factors for Burns   75% result from actions of injured person usually in home  
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Who is at highest risk?   Children under 3 years, and elderly over 70years.  
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Other Risk factors   rural location, mobile home, occupation, lack of smoke detectors, fireworks, and misuse of cig smoking  
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Amount of skin destructed depends on.   temp to which skin exposed, length of time of exposure, and type of injury  
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A tremendous amount of heat is not required to cause..   Damage  
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below 111*F (44*c)   no damage, unless prolonged exposure  
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111*F-124*F (44*C-51*C)   Rate of cellular death doubles with each degree in temp rise.  
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>124*F (51*C)   exposure time is brief for damage  
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SUPERFICIAL (1ST DEGREE)   Epidermal Burn, Cell damage only to the epidermis skin, mild-severe erythema, blanching, surface is dry, no blisters, minor swelling, painful, skin will heal with out scaring, 3-7days  
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PARTIAL THICKNESS (2nd DEGREE)   Superficial Partial Thickness & Deep Partial Thickness  
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SUPERFICIAL PARTIAL THICKNESS   VERY PAINFUL, INTACT BLISTERS(COMMON SIGN), EPIDERMIS IS DESTROYED, SUPERFICIAL DERMIS MIN DAMAGE, MODERATE EDEMA, BLANCHING, HAIR & SWEAT GLANDS PRESERVED, COMPLETE HEAL 14-21 DAYS, LITTLE OR NO SCARING  
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DEEP PARTIAL THICKNESS   DESTRUCTION OF EPIDERMIS & DOWN TO RETICULAR LAYER OF DERMIS, INJURY TO NERVE ENDINGS, HAIR FOLLICLES, AND SWEAT DUCTS; SENSITIVE TO COLD AIR, MIXED RED OR WAXY WHITE; WET SHINY AND WEEPING BROKEN BLISTERS; EDEMA; MAY REQUIRE SKIN GRAFT; HEAL 27+DAY  
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DEEP PARTIAL THICKNESS-- 2 SCARS   HYPERTROPHIC & KELOID SCAR  
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FULL THICKNESS BURN (3RD DEGREE)   ALL EPIDERMAL AND DERMAL LAYERS ARE DESTROYED, SUBCUTANEOUS FAT LAYER DAMAGED  
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ESCHAR   DEVITALIZED TISSUE CONSISTING OF DRIED COAGULUM OF PLASMA AND NECROTIC CELLS -FEELS DRY, LEATHERY, & RIGID --TAN OR YELLOWISH-BROWN COLOR OR BLACK  
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FULL THICKNESS BURN (CONT)   NO BLANCHING, DESTRUCTION OF HAIR FOLLICLES AND NERVE ENDINGS (NO PAIN), DECREASED SENSATION, DAMAGE TO PERIPHERAL VASCULAR SYSTEM, SKIN GRAFT  
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ZONE OF COAGULATION   IRREVERSIBLE DAMAGE, SKIN DEATH!, SKIN GRAFT NEEDED TO HEAL, INCREASED RISK OF INFECTION  
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ZONE OF STASIS   INJURED CELLS (DILIGENT TREATMENT OR CELL DEATH WITHIN 24-48HOURS) TOO TIGHT SPLINTS/COMPRESSION BANDAGES WILL DAMAGE  
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ZONE OF HYPEREMIA   MINIMAL CELL DAMAGE  
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CLINICAL MANIFESTATIONS   LOCATION INFLUENCES SEVERITY  
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PULMONARY COMPLICATIONS   HEAD, NECK & CHEST  
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CORNEAL ABRASIONS   fACE  
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PERMANENT PHYSICAL/VOCATIONAL DISABILITY   HANDS/JOINTS  
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EMERGENT   TIME OF INJURY UNTIL RESTORATION OF CAPILLARY PERMEABILITY  
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RESUSCITATION PERIOD   INITIATION OF FLUIDS AND ENDS WHEN CAPILLARY INTEGRITY RETURNS TO NEAR-NORMAL & LARGE SHIFTS HAVE DECREASED  
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ACUTE PHASE OF RECOVERY   HEMODYNAMICALLY STABLE, CAPILLARY PERMEABILITY RESTORED & DIURESIS BEGINS  
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REHABILITATION PHASE   FINAL PHASE- MAX FUNCTIONAL RECOVERY  
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MOST COMMON AND LIFE THREATENING COMPLICATION   INFECTION  
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OFTEN FATAL COMPLICATION   INHALATION INJURY WITH MAJOR BURNS AND STAPHYLOCOCCAL SEPTICEMIA  
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COMPLICATIONS OF CARDIOVASCULAR & PULMONARY   MULTIPLE ORGAN DYSFUNCTION SYNDROME AND DEATH  
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WOUND CLEANING GOALS   REMOVE DEAD TISSUE, PREVENT INFECTION (STERILE), PROMOTE REVASCULARIZATION &/OR EPITHELIZATION  
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DEBRIDEMENT   REMOVAL OF DEAD TISSUE  
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SHARP DEBRIDEMENT   USE OF SURGICAL SCISSORS OR SCALPEL & FORCEPS; BLEEDING KEPT TO MINIMAL  
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HYDROTHERAPY WITH DISINFECTANT IN WATER   TEMP 98*f -102*F, TOPICAL MEDS OR DRESSING APPLIED  
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PRIMARY EXCISION   REMOVAL OF ESCHAR  
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AUTOGRAFT   PTS ON SKIN USED TO COVER A BURNED AREA, PERMANENT COVERAGE  
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ALLOGRAFT (HOMOGRAFT)   SKIN TAKEN FROM AN INDIVIDUAL OF THE SAME SPECIES; USUALLY CADAVER SKIN, TEMPORARY  
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XENOGRAFT (HETEROGRAFT)   SKIN FROM ANOTHER SPECIES, USUALLY A PIG, TEMPORARY  
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BIO SYNTHETIC GRAFTS (SKIN SUBSTITUTIONS)   CULTURED AUTOLOGOUS SKIN, GROWN IN A LAB, HIGHLY SUSCEPTIBLE TO INFECTION  
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DERMATOME   INSTRUMENT FOR REMOVAL OF SKIN FOR GRAFTING  
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SPLIT-THICKNESS   CONTAINS EPIDERMIS & ONLY SUPERFICIAL LAYERS OF DERMIS, ADHERES BETTER  
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FULL-THICKNESS   CONSIST OF FULL DERMAL THICKNESS, LOOKS BETTER  
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DONOR SITE   SITE FROM WHICH SKIN GRAFT IS TAKEN, THIGHS, BUTTOCKS AND BACK  
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SHEET GRAFT   WITHOUT ALTERATIONS, FOR FACE, NECK AND HANDS  
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MESH GRAFT   ABLE TO EXPAND BEFORE APPLICATION  
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PROCEDURES TO ELIMINATE SCAR CONTRACTURES   SKIN GRAFTS, Z-PLASTY (LEGTHENS SCAR Y INTERPOSING NORMAL TISSUE IN LINE OF SCAR)  
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POSITIONING   DECREASE EDEMA, PREVENT TISSUE DESTRUCTION, MAINTAIN SOFT TISSUE IN AN ELONGATED STATE  
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SPLINTING   PREVENT CONTRACTURES, MAINTAINANCE OF ROM, CORRECTION OF CONTRACTURES, PROTECTION OF JOINT OR TENDON  
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ACTIVE OR PASSIVE EX   PROGRESS 2 STRENGTHENING  
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AMBULATION   AS EARLY AS POSSIBLE  
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SCAR MANAGEMENT   PRESSURE DRESSING, VASCULAR SUPPORT OF SKIN GRAFTS/DONOR SITES, TO CONTROL EDEMA AND SCARRING; MASSAGE TO ASSIST WITH ROM EX.  
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HIGHER MORTALITY RATE   FOR CHILDREN 4 & UNDER AND ELDERLY OVER 65YEARS  
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SURVIVAL RATE FOR ELDERLY   70%  
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FACTORS THAT INCREASE COMPLICATIONS & MORTALITY IN ADULTS   OBESITY, ALCOHOLISM, CARDIAC DISORDERS (PVD)  
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