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4120- Arthritis

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Question
Answer
what is arthritis   a general term for conditions of joint flammation, 1+ joints  
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how many forms of arthritis exist   100+  
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what are the diff types of arthritis classified on   degree of joint damage, restriction of movement, functional limitation, pain  
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3 main classifications of arthritis   osteoarthritis rheumatoid arthritis ankylosing spondylitis  
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what is the number one cause of disability   arthritis  
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how does arthritis affect social funcitoning   increased isolation, stress, depression because of pain, leads to decreased QOL  
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what is osteoarthritis   hands, feet, spine, and weight bearing joints have issues  
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secondary affect of osteoarthritis   decreased strength due to decreased movement  
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pathophysiology of osteoarthritis   constant remodeling of joint tissues in abnormal ways  
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what is periarticular atrophy   muscle atrophy around joints  
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what happens to cartilage in osteoarthritis   decreased cartilage, leads to bone on bone rubbing and inflamation, strained ligmanets, weak and pain  
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steps of osteoarthritis degradation   rough, brittle cartilage damage bone thickens to decrease load synovial swelling fluid increase ligaments thicken, bad movement decrease joint space loss of cartilage bc bone on bone weakens ligaments  
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what is rheumatoid arthritis   a chronic autoimmune form  
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which gender is RA more common in   women  
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how is RA infllamation different   systemic inflammation instead of localized to joints  
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symmetrical polyarthritis   multiple joints, both sides of the body, min 4  
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main type of joint/issue with RA   synovial joints, synovitis  
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common comorbities/risks of RA   muscle atrophy, increased fat mass, fatigue, cv/m disease, t2d, osteoporosis  
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pathophysio of RA   immune dysfunction begins attack synovitis pannus, esp w synovial hyperplasia cartilage and bones erode joint detruction/ankylosis (stiffness)  
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two causes of synovitis   excess fluid synovial cell hyperplasia  
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what is pannus   abnormal tissue layers over joints  
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anklyosis   when the joints become stiff and narrow so movement is restricted  
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which type is more daily wear and tear   OA  
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which type is asymmetric   OA  
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which type occurs at younger ages   RA  
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which type occurs more at smaller joints   rA  
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which type has obvious inflammation   Ra  
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which type does not normally need medical treatment besides pain pills   OA  
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ankylosing spondylitis   chronic autoimmune disorder  
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which gender is AS more common in   men  
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which joints is AS more common at   spine, sacroiliac, cervical, peripheral joints  
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pathophysio of AS   autoimmune response attack lower spine ligaments become inflamed bone growths and bone spurs form IN ligaments as response vertebrae bridges/fusion lower back pain, immobility, bad posture  
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where do bone spurs form in AS   in the ligament  
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4 steps of AS response   inflammation erosion syndesmophytes fusion  
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syndesmophytes   bony outgrowths on spinal ligaments  
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secondary effects of AS   decreased: ex tolerance strength aerobic capacity ROM biomechanic efficency proprioreception social functioning  
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3 stages of arthritis   acute/mind chronic/moderate chronic+acute exacerbation of joint symptoms/severe  
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acute arthritis   reversible s/s @ joints, especially decrease synovitis  
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chronic/moderate arthritis   stable s/s but structural damage is irreversible  
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severe arthritis   increase pain, decreased rom and function, increased inflammation  
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s/s/ of affected arthritic joints   pain stiffness joint locking deformity synovitis creptius effusion bone spurs  
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effusion   fluid around joints  
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crepitus   popping and cracking noises  
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how can extent and severity affect testing   ROM, alignment, function, pain mays be impaired  
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functional levels can affect _____   interventions  
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what sign can help diagnose arthritis   extra articular formations  
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common s/s that may display   redness, swelling, pain, heat around inflammed joint  
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what is the diagnostic for arthritis   there is no definitive test or marker  
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two useful screening tools for arthritis   serum/synovial tests joint imaging/mri/ultrasounds to see degradation and abnormalities  
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why would we need ex testing   may be needed for other risks assessments  
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which type has higher comorbidity (especially cv) risk   RA  
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what main symptom increases risk   inactivity  
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what should we use to form baseline for change and guide Rx   muscular skeletal and ROM testing  
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how should intensity be adjusted for rx   small increases  
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how should modes be decided   based on ability level  
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what stage should use treadmill   minimal/mild  
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what stage should use cycle ergo   mild/moderate in lower extremeties  
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what stage should use arm ergo   severe in lower extremities  
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how does ex benefit arthritic indiviuals   control progression minimize symptoms  
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4 main arthritis treatment goals   decrease inactivity manage s/s/improve adl and qol restore/maintain body comp (some may accumate fat mass from inactivity) decrease comorbidities, symptoms, risks  
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non drug treatments for arthritis   education canes PT/OT shoe mods (orthotics) BRACES/BANDAGES ice/heat decrease weight to decrease load no reptitive motion jobs joint irrigation/surgery  
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what is joint irrigation   flush the joint to remove debris  
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common drug treatments for arthritis   nsaids, opiod pain relievers (anagesics), corticiosteroids (anti inflammatory), DMARDs  
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what are DMARDs   disease modifying artirheumatic drugs used to treat RA and ankylysing spondylosis  
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goals of ex rx with arthritis   increase physical function better body comp decreased bw decrease inflammation pain and stiffness prevent deformities and contractures  
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what is a contracture   permanent shortening and tightening of joints that causes movement to be stuck  
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what is the most physically limited clinical population   arthritis  
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6 special considerations for arthritis   avoid high impact morning stiffness/cold Cl in water therapy footwear ankylos spondylitis posture/back brace corticosteroid degradation  
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high impact ex and arthritis   avoid it, prevent injury and may have had previous replacement  
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why underwater therapy is beneficial   increased buoyancy decreases joint pressure  
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chlorine and underwater therapy considerations   Cl can increase rashes and redness w/inflammed joints, may cause respiratory issues  
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orthotics   inserts and supports in the shoe to help w shock support and ADL pain  
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how do corticosteroids affect long term   bone loss and atrophy due to cortsiol mimicking which is bad long term  
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KAATSU study main take away   low load less joint stress but still ex. benefits blood flow restriction lead to decreased inflammation by increasing post ex bfloww to better remove and filter inflammatants  
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