Critical care - shock, sedation, respiratory support etc.
Quiz yourself by thinking what should be in
each of the black spaces below before clicking
on it to display the answer.
Help!
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| normal MAP | show 🗑
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| show | indicates hypoperfusion and the resultant anerobic metabolism.
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| SVR in sepsis vs hypovolemic shock | show 🗑
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| normal pH | show 🗑
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| normal pCO2 | show 🗑
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| normal pO2 | show 🗑
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| show | 22-26 mEq/L
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| Normal SaO2 | show 🗑
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| metabolic acid base disorders are illustrated by changes in what blood gas measurement | show 🗑
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| show | pCO2 - elevated when acidiv, decreased in alkalosis
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| what is the compensatory mechanism for metabolic acid base disorders | show 🗑
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| how does metabolic acid base compensate for underlying respiratory acid base | show 🗑
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| show | causes of anion gap metabolic acidosis:
Methanol, Uremia, DKA, Propylene glycol, Intoxication/Infection, Lactic acidosis, Ethylene glycol, Salycilates/Sepsis
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| show | causes of non-anion gap metabolic acidosis:
Fistula, Uteroenteric conduits, saline excess, Endocrine, diarrhea, carbonic anhydrase inhibitors, arginine/lysine/chlorine, renal tubular acidosis, spironolactone
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| Hypovolemic shock - first line treatment | show 🗑
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| protocol to achieve goals in pts with sepsis | show 🗑
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| Options other than phentolamine for extravasation. | show 🗑
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| preferred vasopressors for septic shock | show 🗑
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| show | milrinone and dobutamine but only if cardiac function is compromised or persistently low CO or MAP after adequate fluid resucitation.
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| monitoring for propofol | show 🗑
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| What is PRIS? what causes it and what are the symptoms. | show 🗑
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| What sedatives should you avoid loading doses with | show 🗑
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| show | 0.2-0.7 mcg/kg/hr but some evidence supports going up to 1.5
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| show | nausea, vomiting, agitation. occur after prolonged use, 1 wk
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| show | CAM-ICU and ICDSC
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| drugs associated with delerium | show 🗑
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| show | haloperidol - but no evidence to support
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| show | intubated pts with persistent hypoxia despite adequate sedation and analgesia. control intracranial hypertension in pts with neurological injury from TBI
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| risks with paralysis | show 🗑
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| show | prolonged muscle weakness or paralysis once paralytic is removed
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| show | a peripheral nerve stimulation tool. an awake and alert pt should have a TOF (4/4) and should get a baseline.
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| electrolyte disorders that potentiate nerve blockers | show 🗑
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| electrolyte disorders that antagonize nerve blockers | show 🗑
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| drugs that potentiate nerve blockers | show 🗑
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| show | aminophylline and theophylline, CBZ, phenytoin.
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| show | <180
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| show | respiratory failure with mechanical ventilation >48 hours, coagulopathy (plt<50,000; INR>1.5; aPTT>2xcontrol; )
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| show | head or spinal cord injury, 35%+ burn, hypoperfusion, acute organ dysfunction, GI bleed in last year, high dose corticosteroid, liver failure with associated coagulopathy, postop transplant, AKI, major surgery, multiple trauma
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| hypertensive urgency - time to controlled blood pressure | show 🗑
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| key difference between hypertensive urgency and emergency | show 🗑
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| show | encephalopathy, ICH, unstable angina/MI, acute decompensated heart failure, pulmonary edema, aortic dissection, retinopathy/papilledema, decreased urine output or acute renal failure, eclampsia
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| show | decrease DBP to 100-110 or decrease MAP by 25% within 30-60 minutes
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| drugs of choice for hypertensive emergency - tailored by disease state | show 🗑
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| warfarin INR goal in patients with PAH for precention of catheter thrombosis and VTE | show 🗑
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