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EXAM 2 2460

ICP/TBI Quizlet

QuestionAnswer
ICP Pressure inside the skull
What is ICP based on? Balance of brain tissue, blood, and CSF
Monro-Kellie Doctrine If one increases, the other must decrease to keep ICP stable
Autoregulation Process that maintains the balance (if it fails, = increased ICP)
CPP Pressure needed to ensure blood flow to brain
CPP equation MAP - ICP
CPP normal range 80-100
What does a CPP < 60 mean? Ischemia due to inadequate blood supply
What does a CPP < 50 mean? Tissue death
What does a CPP < 30 mean? Incompatible with life and brain damage
What does increased ICP mean? ↑ ICP (more pressure, bad) = ↓ CPP (blood flow)*CRITICAL to maintain MAP when ICP ↑
Normal ICP range 5-15
What does an ICP > 20 mean? Abnormal AND NEEDS TREATMENT ASAP
Factors that influence ICP - Arterial and venous pressure- Intra abdominal and intrathoracic pressure- Posture- Temperature- Blood gases
How is ICP measured? Ventriculostomy- EVD
Causes for increased ICP - Head trauma- Infections --> swelling of the brain*- Hydrocephalus- Brian tumor- Intracranial bleed- Too much CO2- Suctioning, coughing, tight clothing
- Abnormal CT/MRI- ICP monitoring
Nursing assessment for increased ICP Glascow coma scale --> used to assess LOC
Glasgow coma scale
Assessment based on ability to open eyes, speak, and obey commands
Low score = bad
High GCS (13-15)
Awake, normal (low concern)
Moderate GCS (9-12)
Impaired
Low GCS (8 or less)
Severe decreased consciousness
Early s/s of increased ICP - Decreased LOC- Headache --> worse in AM- Confusion, restlessness- Dizziness- Blurred or double vision- N/V
Late s/s of increased ICP Cushings triad -> sign of impending brain herniation
Cushings triad - Hypertension/widened pulse pressure --> systolic - diastolic = huge difference- Bradycardia- Irregular respirations
End-stage signs of increased ICP - Cheyne Stokes respirations- Neck stiffness- Seizures/coma- Fixed pupils- Posturing
Decorticate posturing
Decerebrate posturing
How to decrease ICP --> nursing interventions - Adequate oxygenation- Positioning- Temperature and environment- Avoid triggers- Meds and fluids
ICP oxygenation -nABGs:--- PaO2: ≥ 100 mm Hg-- -PaCO2: 35-45 mm Hg- Intubation or tracheostomy- Mechanical ventilation:--- Keep PaCO₂: 30-35- Suction PRN: OVER suction ↑ ICP--- Hyperoxygenate before & after suctioning- Hyperoxygenate!
ICP positioning - HOB 30-45- Keep head in midline--- Avoid flexion and/or rotation of the neck--- Avoid hip flexion
ICP temp/environment - CORE temp (rectal, foley, esophageal probe)Quiet/Calm environment
ICP things to avoid - Straining, vomiting, coughing, sneezing- Over suctioning b/c it makes you cough + sneeze--- Valsalva maneuver- Agitation
ICP meds and fluids Mannitol and hypertonic/isotonic saline
ICP nursing interventions - Seizure precautions (these pts are very likely to have)- Neuro checks- Calculate CCP hourly : MAP - ICP = CPP
ICP monitoring - Assess for signs of ↑ ICP- Record ICP waveform changes- Monitor fluid and electrolytes
FRONT BACK
What are the 2 major types of stroke? Ischemic and hemorrhagic.
What percentage of strokes are ischemic in the lecture? 87%.
What percentage are hemorrhagic? 13%.
Primary problem in ischemic stroke Vascular occlusion → decreased cerebral blood flow.
Primary problem in hemorrhagic stroke Blood leaks into brain tissue or subarachnoid space.
Major ischemic stroke causes Large-artery thrombosis, small penetrating artery thrombosis, cardiogenic embolism, cryptogenic embolism.
Major hemorrhagic causes Cerebral aneurysm and AVM.
Common stroke manifestations Unilateral weakness/numbness, aphasia, vision changes, gait problems, confusion, severe headache.
BE FAST: B Balance loss.
BE FAST: E Eye/vision changes.
BE FAST: F Facial drooping.
BE FAST: A Arm weakness.
BE FAST: S Speech difficulty.
BE FAST: T Time to call 911.
LEFT hemisphere stroke RIGHT-sided weakness/paralysis, right visual deficit, aphasia, altered intellectual ability, slow/cautious behavior.
RIGHT hemisphere stroke LEFT-sided weakness/paralysis, left visual deficit, spatial-perceptual problems, distractibility, impulsivity, poor judgment/lack of awareness.
Aphasia Impairment of language.
Expressive aphasia Unable/difficulty expressing oneself.
Receptive aphasia Unable/difficulty understanding language.
Dysarthria Difficulty speaking/articulating words.
Apraxia Inability to perform a previously learned action.
First-line imaging for suspected acute stroke/bleeding Noncontrast CT.
Why obtain CT before thrombolytic treatment Must identify/exclude intracranial hemorrhage.
What does CTA evaluate? Cerebral blood vessels/vascular abnormalities.
Why obtain ECG/telemetry after TIA/stroke? Look for cardiac sources such as atrial fibrillation.
NIHSS purpose Measures neurologic deficits/stroke severity and allows trends to be monitored.
NIHSS changes from 6 → 10 Neurologic deterioration; requires prompt evaluation/provider notification.
Warfarin + AFib purpose Prevent cardioembolic stroke.
Warfarin INR goal in today's lecture 2–3.
Antiplatelet purpose Reduce incidence of cerebral infarction when indicated.
Statin purpose Reduce vascular/coronary events and ischemic stroke risk.
tPA purpose Thrombolytic/clot-dissolving therapy for eligible ischemic stroke; time-sensitive.
What finding excludes tPA in the lecture question? Intracranial hemorrhage on CT.
Carotid endarterectom y Surgical removal of atherosclerotic plaque/thrombus from carotid artery.
Carotid artery stenting Opens narrowed carotid artery.
Thrombectomy Endovascular removal of a clot.
Intracerebral hemorrhage Bleeding into brain tissue.
Important ICH risk factor Hypertension.
Amyloid angiopathy Beta-amyloid deposits in cerebral vessels; associated with older adults and hemorrhage.
AVM Abnormal/tangled blood vessels that can rupture.
Subarachnoid hemorrhage Bleeding into the subarachnoid space.
Classic SAH clue Sudden severe/"worst headache of my life."
Other SAH findings N/V, ↓LOC, seizures, neck pain/rigidity, visual disturbances.
Major hemorrhage complications Cerebral hypoxia/decreased blood flow, vasospasm, increased ICP, rebleeding.
Stroke patient becomes increasingly drowsy and difficult to arouse S uspect neurologic deterioration/increased ICP.
TIA Temporary focal neurologic dysfunction from transient cerebral ischemia; warning for future stroke.
TIA symptoms resolve—is the emergency over? No. It still requires urgent evaluation and secondary prevention.
Before giving food/fluid after acute neurologic symptoms Assess swallowing/aspiration risk.
Most important concept with serial neuro assessments TREND/change from baseline.
Created by: Anmag002
 

 



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