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medsurg exam 3

peripheral vascular disease

QuestionAnswer
peripheral vascular disease Diseases of the blood vessels (arteries and veins) located outside the heart and brain
What is the cause? mainly a flow problem
types of causes of peripheral vascular disease Damage or obstruction Pump problem Lymph problem
Damage or obstruction arteries: thrombus, plaque, trauma ** Atherosclerosis most common cause veins: thrombus, incompetent valves
pump problem RHF or LHF
lymph problem cancer surgery, radiation
peripheral artery disease most common chronic cause atherosclerosis
peripheral artery disease: atherosclerosis occurs when there are deposits of fat and fibrin that obstructs and hardens arteries
5 Ps of PAD signs and symptoms of acute limb ischemia
PAD is the most common cause of amputations
Peripheral vascular disease (PVD) risk factors smoking, diabetes, HTN, high cholesterol, obesity, physical inactivity, and age over 50
PVD prevalence increases with age for both men and women, higher prevalence in after 70yo
skin appearance: assessment mottled appearance, dependent rudor smooth, shiney skin with no hair, can be necrotic
mottled appearance a patchy, net-like, or marbled pattern of red, purple, or blue discoloration, often on the legs, arms, or trunk **occurs when elevated
dependent rudor a dark, dusky red discoloration of the legs or feet that occurs when they are in a hanging (dependent) position
pain: assessment intermittent claudication
intermittent claudication pain, cramping, or fatigue in the leg muscles at rest: there is good blood flow when exercising: ischemia in legs when sit down again: good blood flow again
blood flow: assessment longer capillary refill hard to palpate pulses, need doppler
ankle brachial index correlates with severity of claudification
how does ankle brachial index work compares blood pressure in the ankle to the arm to detect peripheral artery disease
as ABI number decreases, PAD gets more severe
PAD management reduce risk factors!! promote circulation/vasodilation
how to reduce risk factors for PAD Smoking cessation = Reduced progression and risk of amputation Lipid lowering diet = Regression, less claudication Disease Management Program - Diabetes control, Hypertension control
how to promote circulation/vasodilation for PAD Walking regimen decreased symptoms greater than angioplasty - 50 min/3x week Antiplatelet agent Promoting circulation to lower extremities -
antiplatelet agents for PAD Aspirin – 1st choice (CAD, cost) Plavix Pletal Trental
promoting circulation to lower extremities do not cross legs, position limbs dependently, keep extremities warm
treatment for PAD walking, arterial stenosis, bypass vessel (bypass blockage), amputation (last resort)
varicose veins are irregular torturous veins
varicose veins usually effect veins of lower extremities (saphenous vein)
varicose veins are caused by long standing increased venous pressure, vein valves become incompetent
varicose veins more likely in pregnancy and obesity older adults
varicose veins manifestations visible, dilated aching, heavy, fatigued legs itching, heat thin, discolored skin around ankles
treatments of varicose veins conservative, ablation therapy, sclerotherapy, vein stripping
conservative treatments of varicose veins Compression stocking (augment muscle pumping action of legs) Leg elevation Exercise
ablation therapy treatments for varicose veins go in with laser and burn vein
vein stripping treatments for varicose veins rip out vein under anastesia
vein ablation Laser or radiofrequency Heated catheter creates scare tissue and causes vein to close
chronic venous insufficiency Inadequate venous return over a period of time
chronic venous insufficiency due to valve injury, can't prevent backflow
chronic venous insufficiency symptoms cellulitis, dull ache, worse when standing
venous statsis ulcer common complication of CVI
how frequent are venous stasis ulcer compromise 50-70% of leg ulcers
avg healing time for venous statsis ulcers 6-8 month, need to wrap legs
lymphedema occurs when there is damage or destruction of the lymphatic pathway
lymphatic system function lymphatic system removes macromolecules to large for reabsorption into the circulatory system
etiology of primary lymphedema congenital, rare
etiology of secondary lymphedema due to carcinoma, causes damage to section of lymph nodes, trauma, radiation, chronic infection and chronic venous insufficiency
lymphedema is chronic debilitating disease that requires lifelong management
if lymphedema is untreated, it can progress which causes continued proliferation of fibrotic tissue an increase in size of infected limb chronic infections
lymphedema results in an increase in functional impairment and a decrease in quality of life
treatment of lymphedema complete decongestive therapy
complete decongestive therapy has to be done by PT
complete decongestive therapy is Manual Lymph Drainage Bandaging Exercise Skin and Nail Care Instruction in Self Care
Home Care to Prevent Injury Inspect daily for problems Wash & moisturize feet daily
Inspect daily for problems Change in temp, appearance, edema
Wash & moisturize feet daily Between toes, mild soap, lukewarm water Rinse, pat versus rub dry Apply moisturizer (avoid excess) Do not apply moisturizer between toes
how to prevent injury Trim nails Always wear socks and shoes Protect feet from hot & cold (water, sand, etc) Choose well fitting shoes - check in the evening because feet swell during the day
educate pt when to contact HCP any skin breakdown, redness or pain
surgery for lymphedema Lymphaticovenular Bypass
Lymphaticovenular Bypass Microscopic surgery lymphatic fluid is redirected to drain through small veins
DVT The formation of blood clot in the deep veins of an extremity
DVT can originate in any extremity but 80% originate in deep veins of calf
DVT is the most common complication of surgery and immobility
DVT/PE Venous Thromboembolism Increased risk for surgical patient without DVT prophylaxis Pulmonary embolism major complication Roughly 1 out of 10 hospital deaths related to blood clots in the lungs
who is at risk for DVT Hospitalized, immobile Surgery – 20% increase, 50 % increase for orthopedic surgery Obesity Smokers Oral contraceptives Central Venous Catheters
DVT assessment: symptoms Dull, aching pain, tenderness, warmth, erythema Edema (Increase in extremity circumference) Can be asymptomatic
Venous Thromboembolism Blood Clots may: remain in vein which is Deep Vein Thrombosis (DVT) Can dislodge and travel to lungs: Pulmonary Embolism
management of Venous Thromboembolism (prophylaxis) Early ambulation Sequential compression devices Compression stockings
management of Venous Thromboembolism (medications) LMWH, heparin
diagnosis of Venous Thromboembolism Duplex venous ultrasonography D-Dimer
Duplex venous ultrasonography measures the velocity of flow in veins
D-Dimer Lab test A compound formed after thrombin converts fibrinogen to fibrin **Negative result use to rule out presence of a blood clot!!
management/treatment of Venous Thromboembolism anticoagulants which are 1 of 3 most dangerous classes of meds associated with adverse events
Management -IV Heparin Therapy with PTT (Partial Thromboplastin Time) or Anti-Xa PTT or Anti-Xa drawn frequently
PTT or Anti-Xa drawn frequently every- 2-6 hours as per nomogram All values are drawn STAT
PTT or Anti-Xa pt must achieve 2 consecutive therapeutic PTTs or Anti-Xa to be able to then draw PTT or Anti-Xa once daily
Management -IV Heparin Therapy Anti-Xa Test that measures anti-thrombin activated factor Xa levels in plasma Therapeutic range: 0.3-0.7 IU/mL
Management-IV heparin therapy baseline Baseline PT/PTT, Anti-Xa, H/H and Platelet count required before therapy is initiated
Management-IV heparin therapy plt and H/H every day
Management-IV heparin therapy: assess for HIT report plt count -<150,000 or a 30-50% reduction (any decrease) assess for signs of bleeding - stool guiac, hematuria
reversal agent for heparin protamine sulfate!!!
management for Warfarin (coumadin) Coumadin is given simultaneously with heparin until Coumadin is therapeutic and then Heparin is discontinued
when should coumadin be given every day
how long is pt usually on coumadin 3-6 months
pt on coumadin/heparin needs monitored of INR, INR range therapeutic is usually 2-3
reversal agent for warfarin vitamin K!!!
why would a pt be on warfarin pt can't go home on IV heparin, so they are bridged to warfarin and once that becomes therapeutic (INR 2-3) then they can be d/c
on warfarin, do not take any over the counter medication or herbal supplements without consulting MD first
warfarin info cont Wear a med alert bracelet No smoking No alcohol Obtain blood work as ordered Take precautions to avoid bleeding Report to ED for episode of bleeding
another option for anticoagulants factor Xa inhibitors
surgery for venous thromboembolism inferior vena cava filter thrombectomy
inferior vena cava filter catch blood clots from going to lungs only if the pt cant be on anticoagulants
thrombectomy minimally invasive procedure to remove blood clots from arteries or veins to restore blood flow
layers of artery Tunica Intima Tunica Media Tunica Externa
Thoracic & Abdominal Artery Aneurysms Localized dilation of aorta
common causes of Thoracic & Abdominal Artery Aneurysms Atherosclerosis Hypertension
types of Thoracic & Abdominal Artery Aneurysms True: saccular, fusiform False: dissecting
Thoracic & Abdominal Artery Aneurysms can involve Aortic arch Thoracic aorta Abdominal aorta
who is at risk for Thoracic & Abdominal Artery Aneurysms Male, 6th or 7th decade, increased BP, atherosclerosis, smoker
increased size of Thoracic & Abdominal Artery Aneurysms means increased risk for rupture
symptoms of Thoracic & Abdominal Artery Aneurysms depend on location, size, growth
diagnosis of Thoracic & Abdominal Artery Aneurysms Angiogram Chest X-ray CT MRI Echocardiogram
medical management of abdominal artery aneurysms if asymptomatic: Aggressive BP control Serial imaging Surgery when ≥ 5.5 cm
surgical management of abdominal artery aneurysms Endovascular grafting (EVSG or EVAR) Open approach
mortality of surgical management ≤ 5% elective; 40% emergent
Endovascular grafting (EVSG or EVAR) take pressure off the weakened aortic wall
abdominal aortic aneurysms nursing management Patient teaching – surveillance
what to monitor for in abdominal aortic aneurysms Unexplained back, chest, flank pain Falling BP or hematocrit Smoking cessation
post op abdominal aortic aneurysms Monitor VS, make sure BP is WNL Assess peripheral pulses - distal to where grafts placed Assess bleeding, pain, fever Avoid coughing, sneezing, vomiting
aortic dissection: type A is an Life-threatening emergency Sudden severe excruciating (tearing or ripping) pain located in the back and/or chest
how does a type A aortic dissection occur Tear in the tunica intima of the aorta Hemorrhage into tunica media Splits the vessel wall, forming a blood filled area between the two layers
main cause of type dissections HTN, 70%
other symptoms of dissections Syncope, dyspnea, hypotension, absent peripheral pulses If major arteries effected: ischemia or effect to major organs
type A aortic dissection surgery Emergent surgery High risk for life threatening complications Only contraindication for surgery is if presence of comorbidities impact survival to one year or less
type B aortic dissection surgery Surgery reserved for development of complications related to dissection If uncomplicated generally managed medically Medical management: Blood pressure control, Imaging surveillance
Created by: leh195
 

 



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