click below
click below
Normal Size Small Size show me how
MedSurge Exam 2
| Question | Answer |
|---|---|
| S3 vs S4 | S3= valvular regurgitation/hypervolmia S4= stenosis that causes hypertrophy, atria contract and fill STIFF ventricle |
| pericarditis relieving position vs aggravating | relief= sit up lean forward worse= lay down supine |
| forward vs backward hf | forward= impaired perfusion, decreased pulses, cyanosis backwards= edem, crackles, jvd |
| mitral valve prolapse and what assessment finding | genetic issue like marfans or rheumatic hd midsystolic clicking |
| aortic stenosis triad of s/s LATE | SAD syncope angina dyspnea |
| SAD triad | syncope, angina, dyspnea aortic stenosis LATE s/s |
| aortic regurgitation assessment findings | water hammer pulse visible neck pulsations |
| dilated cardiomyopathy | s/s= increases preload, fluid overload, crackles, dyspnea, fatigue, JVD, displaced PMI, S3 Gallop causes= genetics, CAD, infections |
| hypertrophic cardiomyopathy | s/s= fatigue, dyspnea, HF like s/s, chest pain, exertional dizzy NO DIURETICS BC CAN WORSEN mostly genetic (young people killed by this a lot bc hard to diagnose) Assess= systolic murmur when SQUAT (esp w kids) |
| Restrictive cardiomyopathy | mostly from amyloidosis or sarcodoisis s/s= PND, nonproductive cough, exercise intolerance, late signs are edema, anasarca (general edema), JVD, S4, liver large, rhf S/S |
| arrhythmogenic RV cardiomyopathy | genetics, muscle replaced with fibrous scar and fatty tissue (HIGH VTACH and cardiac death risk) S/s- asympt until dead |
| Stress induced cardiomyopathy | due to emotions/stress rapid, reversible myocardial weakness Increases troponin but NO blockage s/s= like STEMI, elevated ST |
| heart transplant important info | no vagal innervation so atropine doesnt work resting HR is HIGHER |
| infective endocarditis s/s and what they are | osler nodes= TENDER red/purple bumps on hands and feet janeway lesions= NONtender red spots on feel and hands roths spots= bursts with white dots in eye petechiae splinter hemorrhages= nails have streaks |
| PCI/cardiac stent | balloon expands to flatten plaque go through WRIST mostly through IV CONTRAST |
| PCI major complication and what to do | retroperitoneal bleeding notify provider, admin IV fluid |
| most common vein used for CABG | great saphenous vein |
| CABG complication possibilities | electrolyte imbalance, pneumonia, pulm edema, neuro deficit |
| peripheral bypass graft | prevets peripheral artery disease |
| PAD s/s | intermittent claudication shiny hairless skin decreased pulses |
| Peripheral bypass graft complications | compartment syndrome- tingling/numbness and infection also risk |
| post-op cardiac/vascular surgery care | q15 min for 1 hr, VS mon, assess limb for 6 ps, I and O, s/s of inf, antiplatelet/anticoag |
| demand vs supply anginas | stable= demand (due to exercise usually and incrased myocardial oxygen consumption unstable= supply issue (more occlusion causes decreased blood to area) |
| ACS vs MI | ACS= umbrella term for onset of myocardial ischemia (includes STEMI, NSTEMI, unstable angina) |
| stable v unstable angina | stable= with exertion pain unstable= pain even at rest |
| unstable angina vs STEMI vs NSTEMI | unstable= no troponin, not much EKG change STEMI= ST elevate, troponin elevate, transmural necrosis NSTEMI= no ST elevation, troponin elevate |
| what are the 3 ACS | unstable angina, STEMI, NSTEMI |
| female MI s/s | extreme fatigue, general atypical s/s WORSE outcome due to smaller vessels |
| elderly s/s MI | syncope |
| if MI suspected | EKG, labs (troponin, ckmb, myoglobin), CXR |
| THROMBINS2 | to help MI T- clopidogrel H- heparin R-RAAS O- o2 M- morphine B-beta blocker I-Intervention (PCI) N-nitro S- statin S- aspirin |
| Pulmonary edema | emergency PINK FROTHY SPUTUM AFFFF anxious, tachy, dyspnea, LOC change |
| Cardiogenic shock what to do | o2/intubate QUICK iv morhpine nitro vasopressors hemodynamic mon (Arterial or pulmonary line) |
| Supraventricular tach- what is it and look like | above ventricles (AV node) and caused by stress, caffeiene, etc no T waves bc p waves are so fast it doesn thave time to depolarize |
| V tach- what is it and look like | dangerous ventricles are going too fast, mistly structural abnormalities |
| premature atrial contraction- what is it and look like | p wave normal and then it happens too early and then it pauses to compensate.... too early then chillllllllllll bruh |
| how to treat SVT first and then second | vagal maneuver then adenosine+flush |
| TEE before cardiovert why and when do you not need one | if havent been on anticoag before 3-4 weeks before |
| how to treat a flutter | can vagal iv adenosine cardiovert |
| cardioversion indications | tachyarrhythmias WITH A PULSE |
| when do we use vagal maneuver | SVT helps slow AV NODE conduction |
| torsades de pointes common cause | hypomagnesemia alcoholism |
| asystole treat | CPR Epi intubation |