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MedSurge Exam 2

QuestionAnswer
S3 vs S4 S3= valvular regurgitation/hypervolmia S4= stenosis that causes hypertrophy, atria contract and fill STIFF ventricle
pericarditis relieving position vs aggravating relief= sit up lean forward worse= lay down supine
forward vs backward hf forward= impaired perfusion, decreased pulses, cyanosis backwards= edem, crackles, jvd
mitral valve prolapse and what assessment finding genetic issue like marfans or rheumatic hd midsystolic clicking
aortic stenosis triad of s/s LATE SAD syncope angina dyspnea
SAD triad syncope, angina, dyspnea aortic stenosis LATE s/s
aortic regurgitation assessment findings water hammer pulse visible neck pulsations
dilated cardiomyopathy s/s= increases preload, fluid overload, crackles, dyspnea, fatigue, JVD, displaced PMI, S3 Gallop causes= genetics, CAD, infections
hypertrophic cardiomyopathy s/s= fatigue, dyspnea, HF like s/s, chest pain, exertional dizzy NO DIURETICS BC CAN WORSEN mostly genetic (young people killed by this a lot bc hard to diagnose) Assess= systolic murmur when SQUAT (esp w kids)
Restrictive cardiomyopathy mostly from amyloidosis or sarcodoisis s/s= PND, nonproductive cough, exercise intolerance, late signs are edema, anasarca (general edema), JVD, S4, liver large, rhf S/S
arrhythmogenic RV cardiomyopathy genetics, muscle replaced with fibrous scar and fatty tissue (HIGH VTACH and cardiac death risk) S/s- asympt until dead
Stress induced cardiomyopathy due to emotions/stress rapid, reversible myocardial weakness Increases troponin but NO blockage s/s= like STEMI, elevated ST
heart transplant important info no vagal innervation so atropine doesnt work resting HR is HIGHER
infective endocarditis s/s and what they are osler nodes= TENDER red/purple bumps on hands and feet janeway lesions= NONtender red spots on feel and hands roths spots= bursts with white dots in eye petechiae splinter hemorrhages= nails have streaks
PCI/cardiac stent balloon expands to flatten plaque go through WRIST mostly through IV CONTRAST
PCI major complication and what to do retroperitoneal bleeding notify provider, admin IV fluid
most common vein used for CABG great saphenous vein
CABG complication possibilities electrolyte imbalance, pneumonia, pulm edema, neuro deficit
peripheral bypass graft prevets peripheral artery disease
PAD s/s intermittent claudication shiny hairless skin decreased pulses
Peripheral bypass graft complications compartment syndrome- tingling/numbness and infection also risk
post-op cardiac/vascular surgery care q15 min for 1 hr, VS mon, assess limb for 6 ps, I and O, s/s of inf, antiplatelet/anticoag
demand vs supply anginas stable= demand (due to exercise usually and incrased myocardial oxygen consumption unstable= supply issue (more occlusion causes decreased blood to area)
ACS vs MI ACS= umbrella term for onset of myocardial ischemia (includes STEMI, NSTEMI, unstable angina)
stable v unstable angina stable= with exertion pain unstable= pain even at rest
unstable angina vs STEMI vs NSTEMI unstable= no troponin, not much EKG change STEMI= ST elevate, troponin elevate, transmural necrosis NSTEMI= no ST elevation, troponin elevate
what are the 3 ACS unstable angina, STEMI, NSTEMI
female MI s/s extreme fatigue, general atypical s/s WORSE outcome due to smaller vessels
elderly s/s MI syncope
if MI suspected EKG, labs (troponin, ckmb, myoglobin), CXR
THROMBINS2 to help MI T- clopidogrel H- heparin R-RAAS O- o2 M- morphine B-beta blocker I-Intervention (PCI) N-nitro S- statin S- aspirin
Pulmonary edema emergency PINK FROTHY SPUTUM AFFFF anxious, tachy, dyspnea, LOC change
Cardiogenic shock what to do o2/intubate QUICK iv morhpine nitro vasopressors hemodynamic mon (Arterial or pulmonary line)
Supraventricular tach- what is it and look like above ventricles (AV node) and caused by stress, caffeiene, etc no T waves bc p waves are so fast it doesn thave time to depolarize
V tach- what is it and look like dangerous ventricles are going too fast, mistly structural abnormalities
premature atrial contraction- what is it and look like p wave normal and then it happens too early and then it pauses to compensate.... too early then chillllllllllll bruh
how to treat SVT first and then second vagal maneuver then adenosine+flush
TEE before cardiovert why and when do you not need one if havent been on anticoag before 3-4 weeks before
how to treat a flutter can vagal iv adenosine cardiovert
cardioversion indications tachyarrhythmias WITH A PULSE
when do we use vagal maneuver SVT helps slow AV NODE conduction
torsades de pointes common cause hypomagnesemia alcoholism
asystole treat CPR Epi intubation
Created by: andywattana
 

 



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