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SS #2
Study Stack #2 HP
| Question | Answer |
|---|---|
| Caused by the failure of cells to receive or use oxygen. Deprivation of oxygen may be partial (hypoxia) or total (anoxia) | asphyxiation |
| Compensatory mechanisms for metabolic alkalosis | Respiratory C02 retention (hypoventilation) |
| Major components of the innate response | Natural barriers and inflammation |
| Role of histamine and proinflammatory cytokines | Histamine: induces vasodilation and increased capillary permeability Proinflammatory cytokines: have a key role in the destruction of many pathogenic microorganisms |
| Phases of wound healing: | Hemostasis (coagulation) Inflammation Proliferation/tissue formation Remodeling & Maturation - Fibroblasts |
| Primary vs secondary immune response | Primary Immune Response is the reaction of the immune system when it contacts an antigen for the first time. Secondary Immune Response is the reaction of the immune system when it contacts an antigen for the second and subsequent times. |
| Describe the clinical manifestations of diabetes insipidus | Polyuria, nocturia, continuous thirst, and polydipsia. |
| Role of histamine and proinflammatory cytokines | Histamine: induces vasodilation and increased capillary permeability Proinflammatory cytokines: have a key role in the destruction of many pathogenic microorganisms |
| Systemic manifestations of acute inflammation | Fever, leukocytosis, increased plasma protein synthesis |
| Describe the clinical manifestations of combined deficiencies | Clinical manifestations include bleeding secondary to thrombocytopenia (low platelet counts), eczema, and recurrent infections (e.g., otitis media, pneumonia, herpes simplex, cytomegalovirus). |
| Positive feedback loops: | Enhance or amplify changes; this tends to move a system away from its equilibrium state and make it more unstable. |
| Negative feedback loops: | Dampen or buffer changes; this tends to hold a system to some equilibrium state making it more stable |