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krebs

TermDefinition
State where the Krebs cycle occurs. In the mitochondrial matrix.
State the molecule that enters the Krebs cycle. Acetyl-CoA.
Define acetyl-CoA. A 2-carbon molecule formed from pyruvate during the link reaction.
State the first product formed when acetyl-CoA enters the cycle. Citrate (6-carbon molecule).
Explain what happens to citrate in the cycle. It is gradually oxidized and decarboxylated to regenerate oxaloacetate.
Define decarboxylation. The removal of CO₂ from a molecule.
State how many CO₂ molecules are released per turn of the cycle. Two CO₂ molecules.
State the main function of the Krebs cycle. To generate reduced electron carriers (NADH and FADH₂) for the electron transport chain.
List the high-energy products formed per turn of the cycle. 3 NADH, 1 FADH₂, and 1 ATP (or GTP).
State how many NADH are produced per glucose in Krebs. 6 NADH (because each glucose produces 2 acetyl-CoA).
State how many FADH₂ are produced per glucose in Krebs. 2 FADH₂.
State how many ATP are produced per glucose in Krebs. 2 ATP (one per turn).
Explain substrate-level phosphorylation in Krebs. ATP (or GTP) is generated directly in the cycle without the ETC.
Explain why the Krebs cycle is a cycle. Oxaloacetate is regenerated at the end to react again with acetyl-CoA.
Define oxidative decarboxylation. A reaction that removes CO₂ and reduces NAD+ or FAD simultaneously.
State the importance of NADH and FADH₂. They deliver electrons and hydrogen ions to the electron transport chain for ATP production.
Explain why oxygen is required for the Krebs cycle. Oxygen acts as the final electron acceptor in the ETC; without it, NAD+ and FAD cannot be regenerated.
State what happens if NAD+ is not regenerated. The Krebs cycle stops because NADH accumulates.
Explain why the Krebs cycle increases mitochondrial efficiency. It supplies large amounts of reduced carriers that drive oxidative phosphorylation.
Created by: user-1970252
 

 



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