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| Question | Answer |
|---|---|
| Cholinergic NT's | Acetylcholine |
| Cholinergic Receptors | Nicotinic + Muscarinic |
| Cholinergic Elimination | Degradation by ACH |
| Cholinergic Postsynaptic effect | Nicotinic receptor causes depolarization. Muscarinic receptors can cause both depolarization or hyperpolarization depending on the subtype. |
| Amino Acid NT's | Glutamate, Glycine, GABA |
| Amino Acid Receptors | Glu Receptors, Gly receptors, GABA receptors |
| Amino Acid Postsynaptic effect | Glu receptors cause depolarization. Gly and GABA receptors cause hyperpolarization. |
| Biogenic Amides NTs | Seratonin, dopamine, noroepinephrine, epinephrine |
| Biogenic Amides Receptors | 5-HT receptors, D1 and D2 receptors, Alpha + Beta Adrenergic receptors |
| Biogenic Amides postsynaptic effect | Depolarization or hyperpolarization depends on the specific receptor. For example, D1 receptors cause depolarization and D2 receptors cause hyperpolarization. |
| Neuropeptides NT | Met-enkephalin, beta endorphin, VIP, Substance P |
| Neuropeptides Receptors | There are too many to count! yet they are all specific to peptidase |
| Neuropeptides Elimination | Degredation by enzymes called peptidase |
| Neuropeptides Postsynaptic effect | Depolarization or hyperpolarization depends on the specific receptor. |
| Biogenic Amides Elimination | Reuptake by neurons |
| Amino Acid elimination | Reuptake by neurons or glia |