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Stack #4564207
| Question | Answer |
|---|---|
| That is a practical approach to building a large set of flashcards quickly! Creating study materials using the import method you described—where each line is a card and questions and answers are separated by a delimiter (comma, tab, or equal sign)—is very | |
| While the sources do not mention the StudyStack platform or the specific instructions for importing text [Query], they contain a wealth of critical care pharmacology content, nursing program information, and key concepts that can be converted directly int | |
| Below is a block of text structured for importing, designed to provide you with **over 100 flashcards** based exclusively on the detailed information found within the provided sources. I have used the pipe symbol ($ | $) as the delimiter, as it does not appear elsewhere in the source text, making it suitable for parsing. |
| --- | |
| ## Flashcards for Import | |
| **Delimiter:** $ | $ (pipe symbol) |
| **Block of Text for Import (104 Cards):** | |
| **Henderson State University (HSU) Nursing Programs and General Information** | |
| Henderson State University Nursing Pathway Coordinator and Assistant Professor of Nursing $ | $ Miss Alicia Conn [1, 2] |
| Location of Miss Conn's campus $ | $ Hope [2] |
| Miss Conn's initial nursing role $ | $ CNA [2] |
| Duration Miss Conn was in long-term care $ | $ 20 years [2] |
| Miss Conn's educational pathway (order) $ | $ CNA, LPN, Associates Degree, RN to BSN, MSN [2] |
| Original duration of the RN to BSN program at Henderson $ | $ 12 months (crucial) [2] |
| Current duration of the RN to BSN program at Henderson after Miss Conn's suggestion $ | $ Three semesters [2, 3] |
| The first MSN track Miss Conn completed online at Henderson $ | $ MSN to Nurse Administration [2] |
| Miss Conn's current dual MSN degrees at Henderson $ | $ Nursing Administration and Family Nurse Practitioner (FNP) [2] |
| Miss Conn's primary passion in nursing $ | $ The clinical setting [2] |
| Goal of clinical education according to Miss Conn $ | $ To ensure students can critically think, process theory into practice, and be safe and competent [2] |
| The HSU RN to BSN degree format $ | $ Fully online, no clinicals required on site [2] |
| Cost of HSU RN to BSN tuition (flat rate per semester) $ | $ \$3,080 [2] |
| Characteristic of online classes at HSU $ | $ Small classes, instructors know students by name [2] |
| HSU's approach to late assignments for communicating students $ | $ Flexible (e.g., "Sure, let me know.") [2] |
| HSU's philosophy regarding failure $ | $ Setting students up for success, not failure [2] |
| Additional tracks offered in the HSU MSN program $ | $ Nurse Administration, Leadership, Executive, and Family Nurse Practitioner [2] |
| Programs HSU is currently writing for $ | $ MSN in Education and a DNP program [2] |
| Duration of the FNP and Nurse Administration MSN tracks $ | $ Four to five semesters [2] |
| Advanced focus areas with a Bachelor's degree (BSN) $ | $ Leadership, evidence-based practice, public health, and research [2] |
| Semesters in which the BSN online program is offered $ | $ Fall and spring [3] |
| Start time for the FNP and NEA tracks $ | $ August [3] |
| HSU's policy on prerequisite deadlines for the BSN program $ | $ No deadline (they will still accept them if taken a while ago) [3] |
| Example prerequisite courses listed $ | $ Anatomy and Physiology I and II, Chemistry, Micro, College Algebra, General Psych, Sociology or Communication, Comp I and II, Fine Arts, World Theater/World, and a U.S. History (total of 18 hours) [4] |
| **Critical Care Pharmacology Concepts: Neuromuscular Blockers (NMBAs)** | |
| Two categories of neuromuscular blocking agents $ | $ Depolarizing and non-depolarizing neuromuscular blockers [5] |
| Common depolarizing NMBA often used for intubation and certain procedures $ | $ Succinylcholine [5, 6] |
| Example procedures where succinylcholine is commonly used $ | $ Emergent intubation/procedures where rapid muscle paralysis is needed [5, 6] |
| Propofol's (Dipper van's) half-life $ | $ Very short half-life [5] |
| Requirement for an RN titrating a Dipper van drip real-time in Arkansas $ | $ The patient must have a stable airway [7] |
| Method of action for non-depolarizing NMBAs $ | $ Block acetylcholine at the neuromuscular junction [6, 7] |
| Physiological effects of non-depolarizing NMBAs $ | $ Muscle relaxation and hypotension [7] |
| Crucial fact about non-depolarizing NMBAs (consciousness and pain) $ | $ They do not cross the blood-brain barrier, so patients remain conscious and still feel pain [7] |
| Why pain control/sedation must be given with NMBAs $ | $ Paralyzing someone without pain control is "cruel and unusual punishment" [7] |
| When NMBAs are usually used in conjunction with inhaled anesthesia agents $ | $ For induction of anesthesia [7] |
| Emergency situations where NMBAs might be used to paralyze a patient quickly $ | $ Emergency intubation in the ICU or ER (especially if the patient is fighting/combative) [8] |
| Potential procedures NMBAs are used for $ | $ Transesophageal echocardiogram (TEE), bronchoscopy, and treating status epilepticus (continuous seizures) [6, 8] |
| Negative side effect associated with succinylcholine $ | $ Malignant hyperthermia [6, 9] |
| Key drawback of succinylcholine regarding reversal $ | $ There is not an available reversal agent (antidote) [6, 10] |
| Duration of action for succinylcholine $ | $ 5 to 10 minutes [11] |
| Antidote for Cysatracurium (non-depolarizing NMBA) $ | $ Neostigmine [6, 11] |
| Antidote good for reversing Pancuronium, Vecuronium, or Rocuronium $ | $ Sugammadex (Bridion) [6] |
| Most severe complication of NMBAs (due to diaphragm paralysis) $ | $ Respiratory arrest (requires mechanical ventilation) [12, 13] |
| Nursing responsibility when a patient receives NMBAs $ | $ Monitor EKG, respiratory rate/pattern, O2 saturation, blood pressure (continuously/every 5 minutes), and ensure suction/crash cart/pads are ready [13, 14] |
| What happens if succinylcholine and anesthesia gas are given and malignant hyperthermia occurs $ | $ Severe episodes of muscular rigidity [9] |
| Physiological change causing the dangerous heat in malignant hyperthermia $ | $ Rapid contraction and overactivity of muscles [9] |
| Core body temperature patients can reach during malignant hyperthermia $ | $ Upwards of almost 110° F [9] |
| Electrolyte imbalance most concerning in malignant hyperthermia $ | $ Potassium (Hyperkalemia) [15, 16] |
| Muscle type whose activation is tied to potassium, magnesium, and calcium $ | $ Heart muscle [17] |
| Primary treatment goal for malignant hyperthermia $ | $ Rapidly cool the patient down [17] |
| Drug used to decrease metabolic activity in malignant hyperthermia $ | $ Dantrolene [18] |
| Cooling measures for malignant hyperthermia $ | $ Cold 0.9% sodium chloride IV, cooling blanket, ice packs to the groin and armpit [18] |
| Patients who should NOT receive succinylcholine $ | $ Severe burns, multiple trauma, spinal cord injury, or upper motor neuron conditions (Myasthenia Gravis) [16] |
| Patient complaint common after receiving succinylcholine $ | $ Aching in the upper body and back for days [18] |
| **Critical Care Pharmacology Concepts: Sedatives and Hypnotics** | |
| Definition of Sedatives $ | $ CNS depressants that cause relaxation, calm, and decreased anxiety [19] |
| Definition of Hypnotics $ | $ CNS depressants that primarily cause the induction of sleep [20] |
| Drug classes included in Sedatives/Hypnotics $ | $ Benzodiazepines, barbiturates, and benzo-like medications [19] |
| Risk associated with Barbiturates (unlike benzos) $ | $ Cause tolerance, dependence, and are powerful respiratory depressants [19] |
| Caution for dosing benzos in older patients $ | $ Altered liver/renal function means decreased metabolism and excretion, risking over-sedation [21] |
| Endings of many benzodiazepine names $ | $ -lam or -pam [21] |
| Clinical uses for Benzodiazepines $ | $ Anxiety, seizures, alcohol withdrawal, panic disorder, pre-surgical sedation [21] |
| Signs of CNS depression from sedatives $ | $ Drowsiness, lack of coordination, and light-headedness [21] |
| Key instruction for patients discontinuing benzodiazepines $ | $ Taper the dose slowly [22, 23] |
| Adverse effect that midazolam (Versed) is noted for $ | $ Amnesia (patient doesn't remember the event or waking up) [22] |
| Reversal agent (antidote) for benzodiazepine overdose $ | $ Flumazenil (Romazicon) [23] |
| Contraindications for benzodiazepines $ | $ Sleep apnea, organic brain disease (dementia), breastfeeding, substance use disorder, and use with other CNS depressants [24] |
| Specific adverse effect associated with zolpidem (Ambien) $ | $ Amnesia/memory trouble (driving and forgetting) [24] |
| Route consideration for Diazepam (Valium) $ | $ Precipitates in IV lines with "just about anything" [25] |
| Use of Pentobarbital $ | $ To put a patient in a medically induced coma for increased intracranial pressure [26] |
| Propofol's distinctive characteristic $ | $ White milky substance with an oily look [26] |
| Ingredients in Propofol that cause its appearance $ | $ Soybean oil and a form of egg yolk [26] |
| Risk associated with Propofol vial access $ | $ High chance of bacterial overgrowth (should be one-time use) [27] |
| Reason lidocaine is sometimes added to Propofol IV mix $ | $ Propofol burns badly when injected into peripheral IVs [27] |
| Anesthetic properties of Ketamine $ | $ Dissociative anesthetic [28] |
| Psychiatric use of Ketamine that is non-responsive to other treatments $ | $ Depression [29] |
| Contraindications for Ketamine use $ | $ History of mental illness (like schizophrenia), especially in the very young and very old [29] |
| Effect of Adenosine (Adenocard) $ | $ Puts up a roadblock at the AV node, briefly stopping heart conduction to reset the rhythm [30] |
| Rhythm treated by Adenosine $ | $ Supraventricular Tachycardia (SVT) [30] |
| Nursing procedure for giving Adenosine $ | $ Rapid IV push followed immediately by a rapid saline flush [30, 31] |
| **Critical Care Pharmacology Concepts: Opioids and Antidotes** | |
| Opioids used in the hospital setting (IV) $ | $ Fentanyl, Morphine, Hydromorphone, sometimes Meperidine (Demerol) [32, 33] |
| Antidote for opioid overdose (reverses sedative effects) $ | $ Naloxone [33] |
| Reason to choose Fentanyl over Morphine in some cases $ | $ Morphine sulfate can have a renal component and cause renal damage [33] |
| Maximum amount of Fentanyl (in micrograms) that a smaller, elderly patient might receive $ | $ 12.5 micrograms (compared to 50 for a large adult) [33] |
| Common side effect of opioids on the gastrointestinal tract $ | $ Constipation/slowing of the bowels [34] |
| Adverse effect that causes orthostatic hypotension with Morphine $ | $ Relaxation of smooth muscles in vessels (decreased vascular tone) [34] |
| Opioid that should be avoided in patients with biliary colic (gallstones) $ | $ Morphine (can cause spasms in the bile ducts) [35] |
| Opioid often used instead of Morphine for patients with biliary colic $ | $ Meperidine (Demerol) [35] |
| **Critical Care Pharmacology Concepts: Cardiac and Anticoagulants** | |
| Difference between a thrombolytic and an anticoagulant $ | $ Thrombolytic breaks up a clot; anticoagulant prevents a clot from forming [36] |
| Common anticoagulant example $ | $ Heparin [37] |
| Factors needed to calculate Heparin dosing $ | $ PTT and weight (baseline information) [37] |
| Suffix shared by most thrombolytic drugs (clotbusters) $ | $ -Lase [38] |
| Nursing assessments needed for a patient on a diuretic (like Bumex or Lasix) $ | $ I&Os, electrolyte values, blood pressure, hydration status, and daily weight [38, 39] |
| Primary action of Inotropes $ | $ Strengthen the forcefulness of myocardial contraction [40] |
| Unique effect of Dopamine at low doses (2-5 mcg/kg/min) $ | $ Increases renal perfusion and urine output [41] |
| Risk of Dopamine at high doses (upwards of 50 mics) $ | $ Potent vasoconstrictor that can limit blood flow and cause damage to the kidneys [42] |
| Nursing assessment concern for a patient on Norepinephrine (Levophed) $ | $ Circulation/perfusion of the extremities (cold, mottled, cyanosis, skin integrity) [43] |
| Antidote for many antiarrhythmics (especially Class III, e.g., Amiodarone) $ | $ None listed [44] |
| Most frequent antiarrhythmic given nowadays $ | $ Amiodarone [44, 45] |
| Complication of Amiodarone that requires baseline chest x-ray and PFTs $ | $ Pulmonary toxicity (damage to lung tissue) [44, 46] |
| Reason not to give a Beta Blocker (ending in -LOL) to an asthmatic patient $ | $ Causes spasms of the bronchioles (bronchospasms) [47] |
| Maximum acceptable heart rate before holding a Beta Blocker $ | $ Generally less than 50 beats per minute [44, 47] |
| Class of antiarrhythmics that delay the resetting phase (repolarization) of the heart $ | $ Potassium Channel Blockers (e.g., Amiodarone) [44] |
| Side effect of Calcium Channel Blockers (e.g., Verapamil) $ | $ Peripheral edema and potential to cause heart failure [48] |
| Action of Digoxin (Cardiac Glycoside) $ | $ Slows conduction from top to bottom chambers and increases myocardial contractility (used for A-fib/flutter and heart failure) [31] |
| Nursing assessment required before giving Digoxin $ | $ Apical pulse for a full minute [31] |
| Clinical sign of Digoxin toxicity $ | $ Nausea, vomiting, and feeling unwell [31] |
Created by:
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