Help
Options
Didn't know it?
click below
click below
Knew it?
click below
click below
Don't Know
Remaining cards (0)
Know
retry
shuffle
restart
0:00
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.
Normal Size Small Size show me how
Normal Size Small Size show me how
ms shock
| Question | Answer |
|---|---|
| defined as a complex, life-threatening condition or syndrome with a variety of underlying causes characterized by inadequate blood flow to the tissues (Tissue perfusion) and cells of the body. | Shock |
| A condition in which systemic blood pressure is inadequate to deliver oxygen and nutrients to support vital organs and cellular function | Shock |
| Components of Shock | Adequate cardiac pump Effective vasculature or circulatory system Sufficient blood volume |
| What happens in shock? | 1. Inadequate perfusion 2. Anaerobic metabolism 3. Buildup of lactic acid 4. Metabolic acidosis 5. Respiratory rate increases in response to metabolic acidosis. |
| TYPES OF SHOCK | a) Hypovolemic shock b) cardiogenic shock c) circulatory or distributive d) obstructive shock |
| It occurs when a significant amount of fluid is lost from the intravascular space, fluid are may be blood, plasma, electrolytes solution . | HYPOVOLEMIC SHOCK |
| It is the most common type of shock | HYPOVOLEMIC SHOCK |
| occurs when the heart’s ability to pump blood is impaired. | CARDIOGENIC SHOCK |
| This is a condition that results from inadequate perfusion of body tissue with oxygenated blood that is insufficient to sustain life; cardiac output is decreased/ diminished. | CARDIOGENIC SHOCK |
| With this type, the vasculature is dilated, making it difficult for the heart to move blood and fluid to the rest of the body | Vasogenic shock (distributive) |
| Types of Vasogenic shock (distributive) | Septic shock Anaphylactic shock |
| Massive infection leads to sepsis as a result of the release of endotoxins from bacteria; this causes vasodilation and pooling of blood. | Septic shock |
| A group of symptoms or syndrome in response to an infection that can include organ dysfunction related to the infection | Sepsis |
| The main causative organisms in sepsis | gram-negative and gram-positive bacteria |
| Three major effects septic shock has on the body include | 1 vasodilation, 2 maldistribution of blood flow, and 3 myocardial depression. |
| Septic shock interventions, fluid replacement | 30 to 50 mL/kg fluid resuscitation with isotonic crystalloids; albumin may also be administered as a later intervention. |
| administered to clients with inadequate blood pressure readings despite fluid resuscitation and vasopressor therapy | Intravenous corticosteroids |
| what to administer in septic shock if on a mechanical ventilator and venous thromboembolism prophylaxis. | stress ulcer prophylaxis |
| An allergic reaction to substances such as drugs, food, or insect bites leads to anaphylactic shock, which results in an acute and life-threatening hypersensitivity reaction. | Anaphylactic shock |
| A phenomenon that occurs after spinal cord injury. The injury results in massive vasodilation without compensation as a consequence of the loss of spinal nervous system vasoconstrictor tone; this leads to a pooling of blood in the blood vessels. | Neurogenic shock |
| A type of shock that is caused by a physical obstruction that reduces the filling or outflow of blood from the heart that results in a reduced cardiac output. d | Obstructive Shock |
| reflects bld flow reaching the tissues | Cardiac Output |
| Normal Cardiac Output | 4-6 l/m |
| Amount of pressure the blood is placing on the walls of the vessels as the blood leaves the heart | MAP |
| Normal MAP | 70 mmHg- 105 mmHg |
| A measure of pressure in terms of R ventricular preload; measures the pressure of the blood returning from the body to the heart. N CVP 3-8 mm Hg | Central Venous Pressure (CVP) |
| means R ventricular Failure and volume overload | elevated CVP |
| a measure of perfusion to the brain | Cerebral Perfusion Pressure (CPP) |
| Formula of CPP | CPP=MAP-ICP |
| A CPP of <50 mmHg is associated with | ischemia and tissue death |
| Stages of Shock | INITIAL COMPENSATORY PROGRESSIVE IRREVERSIBLE |
| restlessness, inc HR, cool and pale skin, agitation. the cardiac output is insufficient to supply the normal nutritional needs of tissues but not low enough to cause serious symptoms. | Stage 1 INITIAL STAGE |
| CO is <100 mmHg, dec urine output, confusion, and the CPP is less than 70 mmHg | Stage 2 COMPENSATORY STAGE |
| Edema, excessively low blood pressure, dysrhythmias, weak and thready pulses | Stage 3 PROGRESSIVE STAGE |
| This stage is unresponsive to vasopressors; there is profound hypotension, the heart rate slows, and multiple organ failure ensues. Most often, the client will not survive. | Stage 4 IRREVERSIBLE STAGE |
| The primary goal in shock | improve tissue perfusion |
| administered to increase cardiac contractility and induce vasoconstriction. | Cardiotonic medications such as digoxin, dopamine, or norepinephrine |
| Vasoactive medications to improve cardiac contractility | Dopamine, Dobutamine, Noradrenaline. |
| characterized by generalized inflammation in organs separate from the initial affected area | Systemic inflammatory response syndrome (SIRS) |
| it is a serious condition and may be caused by a severe bacterial infection (sepsis), trauma, or pancreatitis. | Systemic inflammatory response syndrome (SIRS) |
| A complication of sepsis; the failure of two or more organ systems as a result of SIRS, in which homeostasis cannot be maintained without intervention | Multiple organ dysfunction syndrome (MODS) |
| Prognosis is poor once three or more organ systems fail. | Multiple organ dysfunction syndrome (MODS) |
Created by:
lessuy