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241 EXAM 1

CHAPTER 12 CARDIO

QuestionAnswer
what is collateral circulation Heart muscle compensates and makes its own new blood vessels to perfuse the area of the heart when an obstruction of the heart occurs.
disystole relaxes
systole contracts
Cardia output amount of blood ejected from heart per miniunit
Stroke volume amount of blood pumped with each contraction
Preload voloum of blood in the ventricle at the end of and disystole before heart contracts
Afterload resistance the left ventral must over come to eject blood to the res of the body
systolic pressure Top number pressure by the blood on the arteries during systole
Diastolic pressure lower number pressure of the blood on the arteries during disystole
Peripheral vascular resistance the force opposing blood flow. the amount of friction the vessel walls encounter
ADH hormone reabsorbs fluid increasing BV increasing the BP by cinstricting
Aldosterone hold on sodium and water and excrete potassium. increading BV increasing BP
EKG or ECG show electrical and conduction activity of the heart
Echocardiogram ultrasound of the heart. showing valve abnormalities, congenial defects, and heart structure
Chest x ray shape and size, pulmonary congestion of the heart
Coronary angiography die is injected into the arteries to visualize blood flow through the arteries
Troponin test see if there is damage to the heart tissue by the level of the troponin proteins. detects heart attacks
coronary artery disease CAD angina pectoris or cardiac ischemia or MI leading to heart failure
Angina pectoris CAD chest pain when there is a deficit of oxygen to the heart meaning it has to work harder. usually when there is increases demands of the heart
Atherosclerosis narrowing of coronary arteries from a build up of plaque. can causes MI
classic angina chest pain with activity with exertion
Unstable angina prolong chest pain at rest , Heart attack
Arteriosclerosis ticking and hardening of the arteries
Treatment of angina pectoris rest and something that vasodilates the arteries. nitroglycerin
Myocardial infarction - Heart attack CAD coronary artery is totally obstructed leading to death of myocardial tissue because of ischemia.
What does ST elevation MI's or non ST elevation of MI's When a blood clot completely blocks a coronary artery, needing immediate treatment to restore blood flow
How much time does blood have to be restored to not have any irreversible damage from an MI? 20-30 minuets
S/S of MI chest pain that radiates to the left arm, shoulder, jaw, or neck. indigestion, pallor, diaphoresis, dizzy and weakness, dyspnea, marked anxiety or fear, hypotension causing skock.
How to diagnosis a MI? EKG changes, elevated troponin
How to treat MI? oxygen, morphine, blood thinners, Coronary artery bypass grafting surgery, balloon stent, Cardiac rehab
Cardiogenic shock develops when? pumping capability of the left ventricle is impaired
Congestive heart failure CHF happens when? the contractibility of the ventricle is reduced and stroke volume declines. can happen a few days or later when activity is resumed from a MI
Congestive heart failure CHF when heart is unable to pump sufficient blood to meat metabolic demands of the body. usually chronic. one side of the heart usually fails first then the other side
What are the 2 basic effects when the heart cannot maintain its pumping ability? -Cardiac output or stroke volume decreases meaning less blood is reaching organs and tissues. -Backup congestion the output from the ventricle is less then the inflow of blood
Left sided congestive heart failure CHF left ventricles can not pump all of the blood into the systemic circulation and blood from the lungs can not enter the left side of the heart causing pulmonary congestion
What backs up in the lungs during left sided heart failure? Fluid
Right sided congestive heart failure CHF the right ventricle cannot maintain its output so less blood can proceed to the left side of the heart. Back up in the systemic circulation. increases BV in legs and feet neck, liver Digestive
What increases the demands of the heart during CHF? Hypertension because left ventricle has to work harder. when it over exerts it self it hypertrophies and fails
Pulmonary disease comes from what sided of CHF? right side CHF because it increases the work load of the right ventricle then hypertrophies and fails. known as cor pulmonate
S/S of congestive heart failure decrease cardiac output/ blood supply, fatigue weakness, dyspnea, exercise intolerance, dizziness.
S/S of Right sided congestive heart failure edema , ascites, jugular vein destination.
S/S of Left sided congestive heart failure dyspnea, orthopnea, cough, nocturnal dyspnea, pulmonary edema, rales and crackles, low oxygen
How to diagnosis CHF? Chest x ray to see fluid in lungs ABG to measure oxygenation
How to treat CHF Treat the underlying problem, fluid and sodium restriction, antihypertensives, diuretics, vasodilators
When do structural congential heart defects develope? first 8 weeks of embryonic life
Congenital heart defects can be? chromosomal abnormalities such as down syndrome. Environmental factors like infection of rubella, maternal alcoholism, maternal diabetes
S/S of Congenital heart defects small- heart murmur. Large- pallor, cyanosis, tachycardia, dyspnea on exertion, squatting position for comfort bc of blood flow, clubbed fingers, intolerance for exercise and cold weather, delayed growth and development.
How to diagnosis Congenital heart defects? diagnostic imaging, cardiac catheterization, echocardiograms, ECG. usually detected before birth
How to treat Congenital heart defects? surgical repair, oxygen and medications, some septal defects close with time
Ventricular septal defect (Congenital heart defects) Hole in the septum that separates the atriums or ventricles
Valve defects Congenital heart defects valve stenosis restricting forward flow of blood. valvular incompetence failure of a valve to close completely allowing regurgitation
Tetralogy of Fallot (Congenital heart defects) Pulmonary valve stenosis, ventricular septal defect, right ventrical hypertrophy, transposition of aorta
Rheumatic fever/ Heart disease (Inflammation and infection in the heart) inflammation that appears after an abnormal immune reaction a few weeks after untreated infection like group A beta hemolytic streptococcus
What becomes inflamed during Rheumatic fever/ Heart disease? Heart, joints, skin, Pericarditis, myocarditis
The most common problem is endocarditis, what is it? Rheumatic fever/ Heart disease Endocarditis effects the valves that become edematous along the outer edges of the valve cusps. Treatment would be penicillin
S/S of Rheumatic fever/ Heart disease Low fever, leukocytosis, malaise, anorexia, fatigue, tachycardia even at rest, heart murmurs, epistaxis, abdominal pain
how to diagnosis endocarditis? (Inflammation and infection in the heart)? transesophageal echocardiogram.
S/S and treatment of endocarditis? Fever, chills, impaired heart function, osler nodules on fingers. antibiotics
Pericarditis (Inflammation and infection in the heart)? inflammation of the pericardium causing friction rub. could decrease cardiac out put. called cardiac tamponade
S/S of pericarditis Tachycardia, chest pain, dyspnea, cough, friction rub, EKG changes
Treatment of pericarditis? pericardiocentesis- needle used to aspirate or drain fluid from pericardial
Hypertension (Vascular disorder) can result it? renal failure, stroke from hemorrhage, loss of vision, or CHF
What areas are more frequently damaged by hypertension? kidneys, brain, retina
S/S of Hypertension? asymptomatic, fatigue, malaise, headache, consistently elevated BP
Treatment of Hypertension? life style changes like weight loss, stress reduction, sodium restriction, excise, medications
Shock (Vascular disorder) decreased circulating blood volume and decreased tissue perfusion and hypoxia.
What happens if shock is not reversed quickley? organs and tissues will no longer recover resulting in degenerate die, could even be multiple organ failure
Hypovolemic shock loss of blood or plasma from circulating blood
Vasogenic or distributive shock Mass vasodilation causes decreased BP
Septic shock develops in person with a sever infection as bacteria produces endotoxins
Anaphylactic shock rapid vasodilation caused by released histamine in sever allergic reaction
S/S of shock cool, moist, pale skin, tachycardia with weak puls, lethargy, hypoxemia, metabolic acidosis, hypotention
Treatment of Shock? treat underlying problem, fluid replacement, blood transfusion, antihistamines, antibiotics for septic shock, oxygen,, vasoconstriction meds to increase BP
Created by: mmarino2
 

 



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