Term
click below
click below
Term
Normal Size Small Size show me how
BIO 202 Exam 2
Term | Definition |
---|---|
what is the name of the space in a blood vessel where blood flows | lumen |
why do larger veins have valves | to prevent backflow |
from outermost to innermost what are the 3 tunica of an artery | tunica adventitia, tunica media, tunica intima |
what are the functions of arteries and arterioles as well as veins, venules, and capillaries | arteries and arterioles take blood away from the heart and to capillaries, which then flow through venules and veins back to the heart |
what are varicose veins | when the valves in veins collapse and blood begins to pool |
what is pulmonary circulation | blood being carried to and from the alveoli (right side of the heart) |
what is systemic circulation | blood being transported around the entire body |
what bone protects the heart anteriorly | the sternum |
what is the name of the thin tough sac that covers the heart | the pericardium |
what is the muscular layer of the heart called | myocardium |
what type of tissue comprises the bulk of the myocardium | cardiac muscle |
what is the inner lining of the heart called | endocardium |
where would you find the sulci of the heart | grooved areas filled with fat that divide the left and right ventricles and atria |
where is the interatrial septum found | between the walls of the atria |
where can the interventricular septum be found | the thicker wall between ventricles |
what chambers of the heart function to receive blood from the veins | left and right atrium |
what are the two bottom chambers of the heart called | ventricles |
what is the scientific term for "heart strings" | chordae tendinea |
what does prolapse refer to in regards to the heart | when valves open backwards |
what does regurgitation refer to in terms of the heart | when there is a failure of the valves |
what are the papillary muscles and their function | they connect to chordae tendinea in order to help flex and open the valves within the heart |
which side of the heart has a thicker ventricular wall and why | the left side because it needs to pump blood much further through the body |
what is the largest artery in the human body | Vena Cava |
what is angina | a partial obstruction of blood flow which may cause chest pain |
what is an infarction | the complete obstruction of blood flow which may cause death |
what is CABG | Coronary artery bypass graft, bypassing major blocks in the blood vessels (with a vein) to the heart to improve the hearts ability to pump blood |
what does myogenic mean | the heartbeat originates in the heart |
what does auto-rhythmic mean | the heartbeat is regular or spontaneous depolarization |
what region of the heart is referred to as the pacemaker | SA Node |
follow an electric impulse through the heart | Sa node, av node, av bundle (bundle of his), bundle branches, purkinje fibers |
what is the importance of the plateau in a heart cells action potential | it allows the heart to completely empty and refill |
What is an ECG (EKG) | electrocardiogram, checkin the electrical activity of the whole heart |
What does the P-wave signify | atrial depolarization (first small hump) |
What does the P-R interval signify | from start of atrial depolarization to start of the QRS complex |
What does the QRS complex signify | ventricle depolarization |
What does the Q-T interval signify | ventricle depolarization to ventricle repolarization |
What does the T-wave signify | ventricle repolarization (2nd small hump) |
What is PVC? Can this be a normal occurence? | Occurs when purkinje cells or ventricular myocardial cells depolarize to threshold and trigger premature contractions |
What is V-Tac | 4 or more PVC's without intervening normal beats |
what is systole | atrial/ventricular contraction |
what is diastole | atrial/ventricular relaxation |
Cardiac output | heart rate X stoke volume (the amount of blood ejected by the ventricle in 1 minute |
pulse | a surge of pressure within the arteries |
tachycardia | when the resting HR is above 100 |
bradycardia | when the resting HR is below 60 (normal for fit people) |
what is preload | the amount of tension in the ventricular myocardium before it contracts |
predict the effects of chronotropic chemicals: catecholamines | NE and epinephrine (cardiac stimulants), increased HR |
predict the effects of chronotropic chemicals: caffeine | inhibits camp breakdown |
predict the effects of chronotropic chemicals: nicotine | stimulates catecholamine secretions |
predict the effects of chronotropic chemicals: thyroid hormone | increase HR, increases sensitivity to sympathetic stimulation, and increases adrenergic receptors in the heart |
explain contractility | the contraction force for a given preload, contractility can be increased by hypercalcemia, catecholamines, glucagon, and digitalis, or decreased due to hyperklaemia and hypocalcemia |
explain how digitalis works to increase cardiac contractility | increases intracellular concentrations of calcium |
explain afterload | caused by the perssure in arteries above the semilunar valves apposing the opening of valves |
what happens to cardiac output when a person "warms up" during exercise | the HR increases |
symptoms of left heart failure | inability to pump into systemic circulation, the left side tends to fall first, and may be caused by stress or myocardial infarction. Edema may occur in the lungs |
symptoms of right heart failure | inability for the right side to adequately pump venous blood into pulmonary circulation, edema can occur in our hands, feet and rest of our body |
where is majority of blood distributed during exercise | muscular system |
at rest where is majority of blood distribution in the human occurring | veins |
what is blood flow | the rate of blood flowing through a tissue in a given time |
what is perfusion | the rate of blood flow per a given mass of tissue |
mathematical relationship between blood flow, pressure, and resistance a. | |
what is blood pressure | pressure in the arteries |
what is capillary hydrostatic pressure | pressure within the capillary beds |
what is venous pressure | pressure in the venous system |
what is TPR | total peripheral resistance |
what is vascular resistance | friction between the blood vessels and the walls |
what is blood viscosity | thickness or resistance caused by RBC's and albumin as well as other molecules suspended in blood |
what is turbulence | a swirling action that disturbs smooth flow of liquid |
what affect TPR | vascular resistance, turbulence, blood viscosity |
normal BP | 120/80 |
what is pulse pressure | difference between systolic and diastolic pressure |
what is mean arterial pressure (MAP) | diastolic pressure +1/3 pulse pressure |
what factors affect local blood pressure and flow | metabolic theory of auto regulation, vasoactive chemicals, reactive hyperemia, angiogenesis |
where are baroreceptors and how do they restore homeostasis to the body | can be found in the aortic arch and carotid sinus as well as other large arteries restoring homeostasis through negative feedback loops |
what are chemoreceptors | receptors found in aortic and carotid bodies like the aortic arch and subclavian/external carotid arteries. They monitor pH |
How do chemoreceptors respond to hypoxia, hypercapnia, and acidosis | hypoxemia or hypercapnia may cause acidosis in the blood stream, so the pH may lower |
what are the effects of Angiotensin II | increase aldosterone production, increases ADH production, increases thirst, increases cardiac output (BP) |
where does ANP and BNP come from | ANP is produced by cells in the right atrium, BNP is produced by ventricular muscle cells |
what effect do ANP and BNP ave on the cardiovascular system when released | increased BP and blood volume cause release, release then causes a variety of things such as increase Na+ loss in urine, water loss in urine, reduced thirst, etc. to overall cause reduced blood volume |