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Stroke
Question | Answer |
---|---|
inadequate blood flow | ischemia |
Occurs when there is ischemia to a part of the brain, or 2. hemorrhage into the brain that results in death of brain cells. | Stroke |
The severity and loss of function varies acording to the | location and extent of brain damage |
who is more likely to die from a stroke women or men? | women |
what arteries supply the brain with blood? | cartoid arteries and verterbral arteries |
The anterior and posterior circulation is connected at the_______ by the anterior and posterior communicating arteries | Circle of willis |
requires continous supply of oxygen and glucose needed to function | the brain |
how much of CO for optimal functioning? | 20% |
neurologic metabolism is altered in? | 30 seonds |
metabolism stops in | 2 minutes |
cellulatr death occurs in | 5 minutes |
the brain is protected from changes in mean systemic arterial blood pressure over a range from 50-150 known as | cerebral autoregulation |
involves changes in the diameter of cerebral blood vessels in response to changes in pressure so that the blood flow to the brain stays constant | cerbral autoregulation |
carbon dioxide is a | dilator |
increase carbon dioxide | increase cerebral blood flow |
decreased co2 | decreased cerebral blood flow |
very low O2 like less than 50 pao2 then | increase cerebral blood flow. |
Sytemic BP, CO and blood viscosity affect | blood flow to the brain |
Cardiac output has to be reduced by ____ before cerebral blood flow is reduced | 1/3 |
Decreased viscosity | increased flow |
Individual differences in _______ determines the degree of damage and functional loss when a stroke occurs | Collatteral circuation |
alternative route | colateral circulation |
blood streams of the internal basilar system meet in the posterior communicating arterres | in normal situations |
Increased Intracrainial pressure causes brain compression and | reduced cerbral blood flow |
age, gender, ethnicity or race and family history or heredity | Nonmodifable risk factors |
2/3rds of all stokes occur in | people over 65 |
higher incidence of stokes | African americans |
can be potentially altered through lifestyle changes and medical treatment, thus reducing the risk of stoke | modifiable risk factors. |
hypertension, heart disease, smoking, excessive alchol, diabetes, obesity, sleep apnea, metabolic syndrome, lack of exercise, poor diet and drug abuse | modifiable risk factors |
metabolic syndrome | modifiable |
sleep apnea | modifiable |
heart disease | modifiable |
diabetes | modifiable |
stroke can be reduced by ____ with treatment of hypertension | 50% |
Heart disease including a fib, MI, cardiomyopathy, cardiac valve abnormalities and cardiac congenital defects. | risk for stroke |
responsible for about 20% of stokes | atrial fib |
increased cholesterol | modifiable risk for stroke |
how much activitiy is needed to have beneficial impact | even light to moderate |
what kind of diet increases risk for stroke? | high in fat and low in fruits and vegetables |
Hyperhomocystemia | modifiable risk for stroke |
migraine headahces and inflamatory conditions and sickle cell | modifiable risk for stroke |
ischemia without infarction | TIA |
a treansient episode of neurologic dysfunction caused by focal brain, spinal cod or retinal ischemia but without acute infarction of the brain | Transient ischemic attack |
clinical syptoms typically last less than ___ in TIA | 1 hour |
symptoms lasting less than ____ in TIA in the past | 24 hours |
may be due to microemboli that temporarily block blood flow | TIA |
Warning sign of progressive cerbrovascular disease | TIA |
loss of vision in one eye, hemiparesis, numbness or loss of sensation or a sudden inability to speak | In a TIA if the cartoid system is involved |
Amaurosis fugax | loss of vision in one eye |
tinitus, vertigo, darkened or blurred vision, diplopia, ptosis, dysarthria, dysphagia, ataxia, and unilateral or bilateral numbness or weakness | If TIA effects verterbrobasilar system |
results in infarction and cell death | Stroke |
results from inadequate blood flow to the brain from partial or complete occlusion of an artery. | ischemic |
80% of strokes are | ischemic |
occurs from injury to a blood vessel wall and formation of a blood clot | Thrombotic stroke |
develops readily where atherosclerotic plaques have already narrowed blood vessels | thrombosis |
results of narrowing of the blood vessel | Thrombotic stroke |
most common cause of stoke | thrombosis |
why are hypertension and diabetes risks for stroke? | it accelerates atherosclerosis |
30-50% of thrombotic strokes are | preceded by TIA |
Many patients with ischemic stroke ______ have a decreased level of concousness in first 24 hours | DO NOT |
Ischemic stroke symptoms mat progress in the first ______ as infarction and cerbral edema increase | 72 hours |
occurs when an embolus larges in and occludes a cerbral artery, resulting in infarction and edema of the area supplied by the involved vessel | Embolic stroke |
atrial fibrilation, MI, infective endocardities, rheumatic heart disease, valvular prosstheses and atrial septal defects | Heat conditions associated with emboli |
air and fat fro long bone fracutres | Can cause embolic stroke but is less common |
Severe clinical symptoms that occur suddenly | Embolic stroke |
second most common cayse of stroke | Embolism |
Where does most emboli originate? | the endocardal (inside) layer of the heart, with plaque braking off from endocardium and entering circulation |
Warning signs are ____ common with embolic stroke than with thromotic stroke | less |
What generally happens during an embolic stroke | patient usually remains consious, although he or she may have a headache. |
prognosis of an embolic stroke is r/t | the amount of brain tissue deprived of its oxygen supply. |
The effects of emboli are initally charecterized by | severe neurologic defects |
T OR F recurrence of emboli are common | T |
results from bleeding into the brain tissue itself (intracerbral or intraparenchymal) or into the subarachnoid space of ventricles (subarachnoid or intraventricular) | Hemorraghic stoke |
bleeding within the brain caused by rupture of a vessel | Intracerebral hemmorhage |
Prognois of intracerebral hemmorrhage | poor |
most common cause of intracerbral hemmorage | Hypertension |
commonly occurs during periods of activity | hemmorhage |
sudden onset of symptoms with progression over minutes to hours | intracerebral hemmorhage |
neurologic deficts, headache, nausea, vommiting, decreased LOC and hypertension | manifestations of intracerbral hemorhage |
a blood clot within the closed skill can result in a mass that causes pressure on brain tissue, displaces brain tissue and decreases cerbral blood flow, leading to iscemia and infarction | Intracerbral hemmorhage |
initially what is expereinced in an intracerbral hemmorhage? | Severe headache with nausea and vomiting |
Weakness of one side, slurred speech, deviation of the eyes | putaminal and internal capsule bleeding |
hemiplegia, fixed and dilated pupils, abnormal body posturing and coma | Severe hemmorahge |
more sensory than motor loss | thalmamic hemmorhage |
problems with vision and eye movement | subthalmic hemmorhage |
severe headach, vomiting, loss of ability to walk, dysphagia, dysarthria, and eye movement disturbances | cerebelar hemmorhages |
nost serous place of hemmoraghe | pons |
If a hemmorhage occurs here then life functions such as breathing are effected | pons |
hemiplegia, complete paralysis, coma, abnormal body posturing, fixed pupils, hyperthermia and death | hemmorage in pons |
occurs when there is intracranial bleeding into the cerebrospinal fluid-filled space between the arachnoid and pia mater membranes of the brain | Subatachnoid hemorrhage |
Where does bleeding in subarachnoid hemmorhage occur? | CSF between arachnoid and pia mater mebranes of the brain |
commonly caused by rupture of a cerebral aneurysm | Subarachnoid hemmorrhage |
congential aquired weakness and ballooning of vessels | aneurysm |
The majority of aneurysms are in the | circle of Willis |
Besides anurysms what are other causes of subarachnoid hemmorhage? | trauma and illicit drug use |
who has a higher incidence of subarachnoid hemoragic strokes | Women |
in general cerebral aneusyms are viewed as | silent killers but can hace warning signs |
worst headache of ones life | ruptured aneuysm and this subarachnoid hemmorrhage stroke |
LOC may change or not, neurologic defict including crainial nerve deficts,nausea, vommtiting, seizures and stiff neck. | SAH |
In SAH peak time of vasospasm is | 6-10 days after inital bleed |
narrowing of blood vessels | cerebral vasospasm |
cerebral vasospasm is a complication of | SAH |
The neurologic manifestations________ significantly differ between ischemic and hemmorhagic stroke | DO NOT |
Clinical manifestations are related to the | location of the stroke |
The functions affected in a stroke are r/t the | artery involved and area of brain it supplies |
most important thing to ask? | time of onset of symptoms |
impairment of mobility, respiratory function, swallowing and speech, gag reflex, and self care abilities | motor defict |
What causes motor deficts? | the destruction of motor neurons in the pyramidal pathway |
nerve fibers from the brain that pass though the spinal cord to the motor cells | pyramidal pathway |
loss of skilled voluntary movement | akinesia |
loss of skilled voluntary movement, impairment of integration of movements, alterations in muscle tone and alterations in reflexes | charecteristic motor deficts |
progression of reflexes for stroke patients, | hyporeflexes then hyperreflexes |
A lesion on one side of the brain affects motor function on the opposite side of the body | contralateral |
Why is a stroke seen contralateral? | because the pyramidal pathways cross |
What hemisphere is dominant for language skills | Left hemisphere |
loss of comprhension | receptive aphasia |
inability to produce language | Expressive aphasia |
total inability to communicate | global aphasia |
occurs when a stroke damages the dominant hemisphere of the brain | aphasia |
damage to frontal lobe | Brocas |
Damage to temporal lobe | Wernickes |
damage to extensive portions of language areas of the brain | Global |
results from damage to different language areas in the brain | Other |
Types on nonfluent aphasia | brocas, global |
Type of fluent aphasia | Wernickes |
Refers to impaired ability to communicate | Dysphasia |
Minimal speech activity with slow speech that requires obvious effor | nonfluent aphasia |
speech is present but contains little meaningful communication | Fluent aphasia |
a disturbance in the muscular control of speech | Dysarthia |
Impairement may involve pronuciation, articulation and phonation, does not affect the meaning of communication or comprehension of language just mechanics of speech | dysathia |
in a stroke emotional responses may be | exagerated or unpredictable |
What parts of intelectual function may be prepared after a stroke? | memory and judgment |
A left brain stroke is more likely to result in | memory problems related to language |
cautious in making judgements | Left brain stroke |
impulsive and move quickly | Right brain stroke |
Who will have more problems with spatial-perceptual orientation? | right side brain stroke |
may deny their illnesses or own body parts if damage to the | parietal lobe |
Homonymous hemianopsia | blindness occurs in the same half of the visual fields in both eyes |
Agnosia | inability to recognize object by sight, touch, or hearing |
problem with spatial orientation example | judging distances |
Apraxia | inability to carry out learned sequential movements on command |
When stroke affects one hemi of the brain the prognois for bladder control is | excellent |
initially after a stroke a patient may experience what in regards to bladder? | frequeny, urgency and incontinence |
bowel problem r/t stroke | constipation |
What tests can be done to immediatly distinguish between ischemic or hemmoragic stroke | noncontrast CT or MRI |
This can provide an estimate of perfusion and detect filling defects in the cerebral artery | CT angiography |
can detect vascular lesions and blockages similar to CTA | magnetic resonance imaging MRA |
Many strokes are caused by blood clots from the | heart |
can identify cervical and cerebrovascular occlusion, atherosclerotic plaques and malformation of vessels | Angiography |
definitive study to identify the source of SAH | Cerebral angiography |
involves the injection of a contrast agent to visualize blood vessels in the neck and large vessels of the circle of willis | digitial subtraction angiography |
measures the velocity of blood flow in the major cerebral arteries, effective in detecting Microemboli, vasospasm and ideal for SAH | Transcranial doppler |
measures brain oxygenation and temp | LINCOX |
Chosen treatment to prevent stroke for those with TIAs | Antiplatlet, aspirin |
TIA treatment person who has AFIB | anticoagulation |
Surgical interventions for TIA due to cartoid disease include? | cartoid endarerectomy, transluminal angioplasty, stenting, and EC-IC bypass |
Treatment for TIA, the atheromatous lesion is removed from the cartoid artery to improve blood flow | cartoid endarterctomy |
TIA treatment- Insertion of a baloon to open up a stenosed artery in the brain and improve blood flow | transluminal angiplasty |
baloon is threaded up to the cartoid artery via a catheter inserted in the femoral artery. | Transluminal angioplasty |
TIA treatment, attempt to maintain patency of the artery | Stenting |
TIA treatment, this involves anastomosing a branch of an extracranial artery to an intrcranial artery beyond area of obstruction to increase cerebral perfusion | EC-IC bypass |
SINGLE MOST IMPORTANT! point in patients history is? | time of onset of symptoms |
goals of care during acute phase is? | preserving life, preventing further brain damage and reducing disability |
25% of patiients with a stroke have neurologic deficts worsen in the first | 24-48 hours |
Someone after an ischemic stroke, no use of thrombolytics, dont treat BP unless higher than? | 220/120 |
If going to have fibrolytic therapy then the BP needs to be less than | 185/110 and maintained at 180/105 for 24 hours after therapy |
High BP follwing a stroke is? | a good thing! |
IV solutions with what are AVOIDED during acute phase of a stroke? | glucose and water |
should hyperglycemia be treated in acute phase of stroke? | Yes |
How would you manage increased ICP | practices that improve venous drainage such as elevating head of bed, maintaing neck and head alignment, and avoiding hip flexion, managment of hyperthermia |
What stroke would you use TPA? | Ischemic |
produces localized fibrinolysis by binding to the fibrin in the thrombi | Tissue plaminogen activator TPA |
lyses clots | fibrolytics/TPA |
Administered IV to restablish blood flow through a blocked artery to prevent cell death in patients with acute onset of ischemic stroke | TPA |
How long after the clinical signs of iscemic stroke MUST tPA be administered? | 3-4.5 hours of the onset of clinical signs |
if head trauma within last 3 months, major surgery within 14 days then.. | cant give tpa |
Intrarterial tPA can be administered how long after stroke symptoms? | 6 hours |
The use of anticoagulants in the emergency phase of an ischemic stroke is generally | NOT reccomended |
After patient is stabilized and had ischemic stroke | Treated with anticoagulants and aspirin, statins |
A way of opening blocked arteries in the brain by using a removable stent system used in ischemic stroke | Stent retreivers |
The clot seeps into the mesh of the stent | Stent retreiver |
Allows the suregeon to go inside the blocked artery of a patient who is experiencing ischemic stroeks, the corkscrew device reaches the clot in the brain, the device penetrates the clot allowing it to be removed | MERCI retriver |
Platlet inhibitors are contraindicated in | Hemorraghic strokes so are anticoagulatants |
Main drug therapy for patients with hemorrhagic stroke is the managment of | hypertension |
In a hemorrhagic stroke BP is maintained within | normal to high range systolic less than 160 |
immediate evacuation of aneurysm-induced hematomas or cerebellar hemotoms larger than 3 cm | Surgical for hemmorhagic |
Treatmen of anteriovenous malformation seen in hemmorhagic stroke? | Surgical resction or radiosurgery |
Usually caused by ruptured aneurysm | SAH |
treatment of anurysm induced Hemoraghic stroke | clipping or coiling |
aneuysm is sealed off from patent vessel | Coiling |
bleeding into ventricles of brain produces | hydrocephalus so do csf drainage |
decription of current illness with point to onset of symptoms, history of similar experiences, current meds, H/O risk factors and family history | primary assesment |
comprehensive neurologic examination | secondary assesment |
used to avaluate the effect of an acute stroke | National insitutes of health stroke scale |
Primary cause of stroke? | uncontrolled or undiagnosed hypertension |
Place the stroke patient on NPO untill | Dyspagia has been rulled out |
If on mechanical ventilation, oral care every ______ reduces ventilator- assisted pnumonia | 2 hours |
with unclipped or uncoiled anurysm dont.... | DONT suction as it can increase ICP, dont cough |
Measures stroke severity, predictor of both short and long term outcomes of stroke patients | NIHSS |
A decreasing LOC may show | increasing ICP |
Becuase hypertension is seen after a stroke monitor for | othostatic hypotension when ambulating for first time |
Since a patient is at risk for VTE after a stroke what may be given as prophlaxis? | LMWH |
nursing goal for musculoskeletal | prevent joint contracutures and muscular atrophy |
How to position if someone is paralyzed on one side? | each joint higher than the joing proximal to it to prevent dependent edema |
Trochanter roll is used in stroke patients to | prevent external rotation of the hip |
hand roles prevent | hand conracures |
How long can a person be positioned on paralyzed side? | 30 minutes |
in acute stage of stroke the primary urinary problem is? | poor bladder conrol, resulting in incotinence |
associated with communication difficulties, undressing and dressing | Functional incotinence |
Patients may initially recieve | iv infusion to maintain fluids/electrolytes, |
majority of patients experience | dysphasia after a stroke |
How long after feeding should someone with dysphagia remian sitting upright? | 30 minutes |
What diets avoid in stroke patients? | purreed, thin liquids and milk |
How do you know if dieatary program is effective? | maintence of weigh, adequate hydration, a patient satisfaction |
If the patient cannot understand then | use gestures to support verbal cues |
Difficultu judging position, distance and rate of movement | stroke on right side of brain |
Impulseive, impatient and deny problems related to stroke | stroke on right side |
at higher risk for injury because of mobility difficulties | right brain stroke |
What is the best way to give directions for activities for comprhension for someone who had a right sided brain stroke? | Verbally |
one sided neglect is common for people with | right sided brain stroke |
slower in organization and performance of task | left sided stroke |
they tend to have impaired spatial discrimination | Left sided stroke |
The patients admit to defects, are fearful and anxiour | left sided stroke |
What is helpful for comprehension of instructions for those who suffer a left sided stroke? | Non verbal cues |
Persistent disregard of objects in part of the visual field should alert you to this | Homonymous hemianopsia |
neglect sydrome or visual field cut may affect | both left and righ sided stroke |
drooping eylid | ptosis |
Process of maximizing the patients capabilities and resources to promote optimal functioning related to physical, metal and social well being | Rehabilitation |
prevent deformity and maintain and improve function | Goals of rehab |
The first year of recovery after a stroke is where the patient will see | the maxmum benifit of rehab |
If muscles are still _________ several weeks after the stroke the prognois for rehab is poor | flaccid |
Patient has voluntary control of isolated muscle groups | final stage of recovery from stroke |
inital step in recovery | balance training |
encourages the patient to use the weakned extremity by restricting movement of normal extremity | Constraint-induced movement therapy |
exagerated mood swings are especially seen in | stroke on left side of brain |
When the patient does not maintain optimal functioning fro self care, family responsibiliries, secion making or socialization | Maladjusted dependece with inadeqaute coping |
highest incidence of stroke occurs among | older adults |
determines patency of cerebral blood vessels | angiography |
Caused by a ruptured blood vessel | Intracerebral Hemorhagic stroke |
Intracerebral hemmorhage can result in | creation of a mass that compresses the brain |
Impaired judgment is most likely the result of | right sided brain damage |
inability to remeber words is most likely the result of | Left sided brain damage |