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Immunologic Disorder Hangman

 
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Immunologic Disorders 1 2 3 4 5 6
Cellular immunity  T cells or lymphocytes  Helper induce T cell, cytotoxic or killer t cell, supressor t cell, supressor inducer t cell  (blank)  (blank)  (blank)  (blank)  
Humoral immunity  B cells or lymphocytes  IgM, IgA,IgG, IgE, IgD  (blank)  (blank)  (blank)  (blank)  
Tests for cellular immunity  Quantitative ( lymphocyte count, T cell subset (CD4, CD8), NK cell numbers)  Qualitative (DTH skin test, ability to elaborate cytokines ( IL1,2,4, TNF)  Ability to generate MHC-restrict CTL  (blank)  (blank)  (blank)  
Tests for humoral immune response  serum immunoglobulin levels  determination of isohemagglutinin and febrile agglutinin titers  determination of antibody titers following immunization with tetanus toxoids, diphtheria toxoid, or pneumococcal polysaccharide vaccines  protein electrophoreisis( albumin,globulin,fibrinogen)  immunoelectrophoreisis  (blank)  
Disorders of immunoglobulin overproduction  MGUS  multiple myeloma  waldenstrom macroglobulinemia  Amyloidosis  heavy chain disease  (blank)  
Primary Immunologic Difficiency Disorder  selective IgA deficiency  Bruton's X linked, Degeorge thymic hypoplasia  chronic mucocutaneous candidiasis  Wiskott-aldrich syndrome  chronic granulomatous disease (inability tto develope neutrophils  ataxia and aids  
Secondary Immunologic Difficiency Disorders  AIDS  iatrogenic ( cancer chemotherapy)  immunodificiency associated with Sarcoidosis and Hodgkins  (blank)  (blank)  (blank)  
Cell mediated autoimmunity  SLE  (blank)  (blank)  (blank)  (blank)  (blank)  
Antibody-mediated autoimmunity  hemolytic anemia  idiopathic thrombocytopenia  goodpasture's syndrom  grave's disease  myasthenia gravis  type 1 DM  
Immune complex disease  SLE  rhumatoid arthritis  drug induced hemolytic anemia  drug induced thrombocytopenia  (blank)  (blank)  
Diagnosis  agglutination of antigen-coated RBC  Enzyme-linked immunoassays (ELISA)  immunoflourescence microscopy  complement fixation  (blank)  (blank)  
Treatment  supression of autoantibody productions (corticosteroids, cytotoxic agents)  relieve tissue damage from immune complexes (ASA,NSAID,colchine, corticosteroids)  remove offending autoantibodies and circulating immunecomplexes (cytotoxic drugs)  (blank)  (blank)  (blank)