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Neuro core

QuestionAnswer
SE phenobarb sedation, hyperactivity
SE phenytoin gingival hyperplasia, BM suppression
SE primidone sedation, hyperactivity
SE ethosuximide GI upset, HA
SE carbamazepine (tegretol) low Na, low WBC, hepatitis
SE valproate (depakote) low plts, hepatitis, pancreatitis
SE felbamate aplastic anemia
SE gabapentin sleepiness
SE lamotrigine (lamictal) rash (incrsd w VPA)
SE topirimate (topamax) cognitive slowing, renal stones
SE tiagibine dizziness, somnolence
SE levtiracetam (keppra) sleepiness, psych disturb in kids
SE oxcarbazepine (trileptal) low Na
SE zonisamide (zonex) renal stones
which sz meds can be given IV phenobarb, phenytoin, valproate, levetiracetam
tx for tourette psych dopa agonists like haloperidol, pimozide + clonidine
tx for tremor (3) bb, primidone, topamax
which sz med esp good for tuberous sclerosis tigabitran
SE tigabitran VF loss
how bring out breath holding spell for pallid (pain related), press on eye
when does stroke appear on CT after 3 days
when does hemorrhage show up on MRI after 3 days
how does hemorrhage appear on MRI ID, BD, BB, DD (T1/T2 <3d, 3-7d, >7, chronic)
key components orbitofrontal emotion, attn, impulse (assoc limbic)
key components medialfrontal motivation (lesion gives akinetic mutism, apathetic state)
key components dorsolateral frontal attn, working memory, sequencing (spelling WORLD backwards), executive fxn pseudodepressed
key components parietal nondominant=visuospatial (ie drawing), dominant=praxis
key components lateral temporal dominant=speech, nondominant=understanding prosody (emotl content of speech**1 language component handled by non dominant), also auditory perception and vocabulary
where's lesion if trbl pairing things lateral temporal
what are 2 main parts of medial temporal hippocampus and amygdala
key components hippocampus, 2 dz in which affected short term memory storage/retrieval and consolidating short term into long term, affected in AD and temporal lobe epilepsy
key components amygdala emotionally charged memories (overlap w primary olfactory, ie power of smells), aversive conditioning
how is dominance determined where language center is
MC site of epilepsy in adult hippocampus (ie medial temporal)
drawing all animals the same would be injury where lateral temporal
where do sympathetic and parasympathetic tracts start hypothalamus
where's the red nucleus, what is it in midbrain, where Cb synapse
describe pt HM had hippocampus and amygdala removed bilaterally and couldn't form any new memories (hippocampus consolidation short term memory to long term memory), but had procedural memory and would improve on tasks
sources of posterior circulation and anterior circulation and relation to structures anterior circulation from carotid (includes m cerebral), near optic chiasm; posterior circulation from vertebral, near brainstem
which side do Cb lesions affect SAME SIDE (ipsi) bc dbl crossed
s/s of lateral medulla stroke ipsi horner, ipsi face incl dysphagia/gag, ipsi ataxia, contra body
what does MLF connect CN3,6 for horiz gaze
key differentiating feature for INO they can still converge
which CN exits from back of head CN 4
where does CNI synapse in medial temporal lobe (not in brainstem)
where does CNII synpase in thal (LGN)
L SCM turns head which way? to the R
if lesion CN12 which way tongue ipsi
if lesion CN9,10 which way uvula away (contra)
compare spasticity and rigidity spasticity is velocity dependent (resistance incrses the faster you go), rigidity is steady tension that's equal in opposing mscl grps
what are cortical sensory fxn tests? What does it test? stereognosis (ID object by touch), graphesthesia (ID
what is responsible for bringing perceived threat to your attn amygdala and thalamus
what is a 3+ on DTR? 4+? 3+=spread to other motor neurons, 4+=clonus
on mscl power what is 3/5? 4/5? 3=full motion w/o gravity, 4=motion w gravity
if can't adduct leg what nerve? Hamstring? Calf? adduct=obturator, hamstring=sciatic, calf=tibial
2 parts of Cb and what control vermis=axial, flocculonodular=vestibular balance
how is sensory ataxia difft from Cb ataxia Cb ataxia has wide based walk and look drunk, sensory ataxia worse in dark
what's festination walking progressively faster bc falling fwd, ie propulsion
what's steppage gait lift foot high 2/2 foot drop (peroneal n and anterior tibialis mscl)
waddle gait seen in which types of dzs prox mscl wknss
Created by: ehstephns on 2011-09-17



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