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CP Exam II

PT Associated with Airway Clearance Dysfunction

TermDefinition
Airway Clearance removal of secretions from the airways
Normal Airway Clearance Depends on... patent airways mucocillary escalator expiratory flow
Diseases of Airway Clearance Dysfunction Cystic Fibrosis COPD Bronchiectasis *Any # of conditions that impair normal airway clearance - often in acute care or rehab settings - surgery, chest trauma, weakness, etc.
Secretion Mobilization deep breathing (inspiratory muscle trainer) positive expiratory pressure (PEP) devices oscillatory PEP devices manual airway clearance high-frequency chest wall oscillation
Secretion Removal cough forced expiratory technique suction
Effective Coughing Requires... - large inspiration follow by glottis closure - increased abdominal pressure by contracting expiratory muscles - sudden opening of the glottis allows high expiratory air flow to shear mucus, which expels it toward the mouth
Ineffective Coughing - inability to effectively close glottis (e.g., CVA) - weak inspiratory effort leading to low volume of inspired air or weak expiratory effort (e.g., neuromuscular disease, weakness, pain due to surgery/trauma) - premature airway closure
Outcome Measures for Effective Mobilization/Removal of Secretions - sputum clearance or mobilization - improved gas exchange (SaO2, ABG) - chest x-ray - auscultation (air entry, decreased wheezes & crackles)
Patient Airways...Essential - must have adequate inspiration to get good exhalation - secretion removal may need to begin with deep breathing to open closed airways
Facilitating Inspiration 1. Determine cause (pain, fear, respiratory muscle weakness) 2. Address the cause ( time treatment around pain medications, braching)
Addressing Cause of Low Inspired Volume - incentive spirometers (volume type, flow type) - sustained maximal inspiration (< 3 seconds) - special concerns for COPD - give air time to diffuse into distal alveoli (exhalation helps push blockages out)
Incentive Spirometers must consider... - pts posture (inhale = thoracic ext.) - exhale before attempting inspiration - avoid rapid, high inspiratory flow rate - sustained maximal inspiration - coordinate treatment with pain meds
Inspiratory Muscle Training has a valve you must develop a certain pressure against - forces you to produce a higher pressure to inhale air
Walking & Exercise increases ventilation
Positive Expiratory Pressure (PEP) - positive airway pressure during exhalation created to allow unimpeded inspiration - air comes out at slower rate keeping pressure in airways elevated (back pressure) - rationale: pos. pressure prevents atelectasis (collapse) & helps move secretions
Oscillatory PEP - flutter device creates oscillating PEP for airway patency & collateral ventilation - oscillation loosens airway secretions & decreases sputum viscosity
Acapella similar to oscillatory PEP
High-Frequency Chest Wall Oscillation - intermittent compressive pressure @ certain frequency that is designed to optimize secretion clearance via biased airflow & enhancing cilia action - vibration helps to loosen secretions & then patient coughs them up
Manual Airway Clearance - percussion (chest PT) is rhythmically striking the chest wall over specific lung segments - usually done in a postural drainage position
Postural Drainage - uses different body positions to facilitate gravitational drainage from lung segments (no voluntary effort required) - rationale: gravity pull on secretions, more negative pressure surrounding non-dependent lung segments - maintain position 10-20 min
Postural Drainage Contraindications increase intracranial pressure unstable CV Status GI feeds vomiting or aspiration pulmonary edema positional intolerance no head down in infants with CF
Active Cycle of Breathing Technique BC 20-30 sec normal -> 3-4 deep breath w/ TE -> BC -> 3-4 deep breaths w/ TE -> BC -> huff cough -> effective coughing *goal = mobilize & expel mucous
Forced Expiratory Technique (FET) - glottis remains open - huff at mid to low volumes followed by force expiration using abdominal muscles to facilitate excretion & decrease probability of airway collapse
Forced Expiratory Technique (FET) Indications - people with compromised airway stability (CF, COPD) - people with difficulty coughing due to chest wall or intrathoracic pain
Pursed Lip Breathing - helps control dyspnea - creates small pack pressure (gentle prolonged exhalation) - contributes to: keeping airways open, facilitates air movement of pulmonary system
Oxygen Therapy Physiology - PaO2 is sensitive & non-specific indication of gas exchange - FiO2 is the same at all altitudes, pp O2 changes - normal PaO2 decreases with age - body does not store oxygen - high levels of supplemental oxygen causes tissue damage
Indications for Supplemental Oxygen - hypoxemia - increased work of breathing - increased myocardial demand - pulmonary hyertension
Signs & Symptoms of Hypoxemia - cyanosis - progressive loss of cognitive & motor function - impaired judgement - decreased exercise tolerance - headache - breathlessness or severe dyspnea - palpitations - angina - restlessness - tremors
What is Oxygen Therapy? - Increases PaO2 (increases difference between pressure in alveoli & blood) - decreased hypoxemia (reduces vasoconstriction of pulmonary vasculature) - if pt is on O2: FiO2 x 500 = expected PaO2 *if ABG shows PaO2 = 95 mmHg, assume hypoxemia off O2
Supplemental Oxygen - fraction of O2 is raised by ~4% for each 1 L/min of O2 supplied - typically delivered via Nasal Cannula at rates of 1-5 L/min (FiO2 = 0.24-0.4) - masks are used for high delivery rates (FiO2 > 0.4) or if concerned about reliability of delivery
Oxygen Concentrator - air passes through filer & oxygen is pulled in a reservoir - home concentrators are on wheels but heavy to move - must have max flow rate of 5L - requires electrical power
Types of Oxygen Concentrators: Portable max flow rate 2-3L/min & battery operated
Types of Oxygen Concentrators: Liquid - 99.5% pure oxygen - -297°F - converts back to gas when exposed to warmer temps - needs to be stored in special tank for cold - cheaper/less bulky - not for short term use
Types of Oxygen Concentrators: Compressed Gas E Cylinder - 3 ft tall, 8 lbs, 2200-3000 PSI O2 gas (5-6 hours @ 2 L/min) - 0.25-25 L/min range - made of aluminum rather than steel (lighter)
Types of Oxygen Regulators: Continuous Flow - continuous flow during inspiration & expiration - recommended with higher O2 flow rates
Types of Oxygen Regulators: Pulsed - bolus of O2 upon inhalation - tank lasts longer - requires double lumen nasal cannula to sense inhalation
Low Flow Oxygen - increases FiO2 above room air - O2 is mixed with room air (dilutes amount of O2)
High Flow Oxygen - delivers increase FiO2 @ rates of 40-60 L/min via nasal cannula or mask - constant FiO2 up to 1.0 - requires wall connection - oxygenation not dependent on patients breathing pattern - reduces need for intubation or non-invasive ventilation
Indications for High Flow Oxygen indicated in patients with chronic hypoxia, increased work of breathing, & hypoventilation
Advantages of High Flow O2 decreased O2 dilution washout of waste gases decreased respiratory rate increased tidal volume & end expiratory volume positive end expiratory pressure facilitates secretion clearance decrease dev. of bronchial hyper-response symptoms
Heat Humidification added to most higher flow rates of O2 or pt. is on O2 for longer period because O2 dries out airways (especially mucosa)
Physical Therapist Role in Oxygen Therapy - oxygen considered a drug requiring MD order - APTA reports they are unaware of laws limiting PT's in use of or titration O2 - know your state laws - always check for specific orders - communicate - maintain pt.'s SpO2 > 90%
PT's Role in Oxygen Therapy pt. 2 - assists with titration of O2 - develop exercise prescription with O2 adjustments as needed - educate patient on oxygen adjustments with activity
Medicare Coverage PaO2 < 55 mmHg at rest SpO2 < 88% at rest More lenient criteria if SpO2 < 88% & SpO2 improves w/ supplemental O2
Oxygen Toxicity: Exudative Phase damage to the alveolar-capillary membrane increased permeability to fluid, protein, electrolytes leading to pulmonary edema - capillaries clogged with platelets, interstitial inflammation
Oxygen Toxicity: Proliferative Phase fibroblasts & epithelial cells proliferate collagen deposited in interstitium
Oxygen Toxicity: Recovery Phase can lead to scarring *If severe enough can cause Acute Respiratory Distress Syndrome
Created by: Cummings226134
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