test 4 level 3
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| show | Gastritis and Peptic ulcer disease
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| show | Gastritis
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| gastritis may be clasified as ? | show 🗑
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| occurs as a result of a breakdown in the normal gastric mucosal barrier. | show 🗑
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| protects the stomach tissue from the corrosive action of HCL acid and pepsin | show 🗑
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| In gastritis when the mucosal barrier is broken, HCL acidm and pepsin can diffuse back into the mucusa resulting in? | show 🗑
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| show | NSAIDS, inlduign aspirin and corticosertiods, as they inhibit prostaglandin(which is protective to GI mucosal wall) and anticoagulants and digoxin
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| Injesting alchol, eating large quanitites of spicy, irriating foods and renal failure can cause? | show 🗑
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| show | Helicobacter pylori
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| Bacterial, viral and funfal infections have been associated with? | show 🗑
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| Intense emotional responses and CNS lesion may produce inflmation of mucosal lining as a result of hypersecretion of HCL acid | show 🗑
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| Anorexia, nausea, vommiting, epigastric tenderness and a feeling of fullness. Hemmorhage is associated with alchol abuse and at time is the ONLY symptom | show 🗑
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| show | Self limiting, lasts a a couple hours or days, complete healing
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| When the parietal cells are lost as a result of atrohy the source of intrinsic factor is lost. | show 🗑
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| A CBC in gastritis may show? | show 🗑
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| show | rest, NPO status and IV fluids, antimetics
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| show | PPI's( azoles) or H2 receptor blockers (tidine)
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| show | Antibitotics for H. pylori, colabalmin therapy, non irritating diet w/ six small meals a day
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| drug and alchol abuse think? | show 🗑
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| show | occult blood
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| show | NG tube for lavage
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| Neutralize acid in stomach and esophagus, rapid onset and short effect | show 🗑
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| show | before a meal
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| show | H2 receptor blockers
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| Inhibits gastric acid secretion has a prolonged effect | show 🗑
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| show | all forms of Gastritis
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| A condition charecterized by errosision of the GI mucosa resulting from digestive action of HCL acid a pepsin | show 🗑
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| show | Any portion of the GI tract that comes into contact with gastric secretions
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| show | acute or chronic
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| show | The acute ulcer
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| eroding throguh the musclular wall with the formation of fibrous tissue | show 🗑
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| show | acidic enviorment
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| histamine in released from the damaged mucosa, resulting in vasodilation and increased cappilary permeability in peptic ulcers results in? | show 🗑
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| show | peptic ulcers
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| alters gastric secretion and produces tissue damage leading to? | show 🗑
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| show | Ulcerogenic drufs
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| Coffe and alchol | show 🗑
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| show | The healing of them
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| show | Antrum
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| show | Obstruction
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| H.Pylori, meds, smoking and bile reflux are R/F for? | show 🗑
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| Most common ulcer? | show 🗑
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| show | H. pylori
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| high epigastrum pain, occurs 1-2 hours after meals, pain is burning, if ulcer has eroded through mucosa food agravates this | show 🗑
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| show | Duodenal ulcers
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| Tendencey to occur continously for a few weeks or months and then disaper for a time then recurs | show 🗑
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| Older adults and ulcers | show 🗑
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| show | Hemorrhafe, perforation and gastric outlet obstruction, EMERGENCIES
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| Hemmorahfe is the more common in | show 🗑
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| show | Perferation
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| show | Perferation
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| show | Perforation
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| show | [erforation
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| if perforaiton is untreaed bacterial periotnitis may occur within? | show 🗑
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| show | edema, inflamation or pylorspasm and fibrous scar tissue formation
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| how can relief be obtained with gastric out obstruction due to PUD? | show 🗑
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| Vomit that contains food particles that were injested hours or days before the vommiting episode signals? | show 🗑
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| show | Endoscopy
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| Eoscopy with biopsy is used in PUD to rule out? | show 🗑
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| no invasive tests for H. Pylori? | show 🗑
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| show | may show anemia
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| show | Pacreatic
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| aim of treatment with PUD? | show 🗑
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| Pain from ulcers disaper in? | show 🗑
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| show | follow up endoscoppy
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| show | Antibiotics and PPI
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| show | smoking
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| Because ulcers frequently recur, interupption or discontinuation of therapy can have? | show 🗑
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| show | the ulcer has healed or used as low dose maintance
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| show | H2 receptor blockers
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| show | Proton pump inhibiros
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| These increase gastric Ph by neutralzaing the HCL acid. magnesium hydroxide or aluminum hydroxide | show 🗑
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| show | after meals so they last a long time
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| If Ph is less than 5 with PUD consider? | show 🗑
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| used for short term treatment of ulcers. It proovides cytoprotection for the esophagus, stomach and duodenum, should be given 30 minutes before an antacid. | show 🗑
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| show | Tricylic
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| show | No caffeine, no aclhol, do bland diet, (no broth tho)
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| PUD vleeding, increased pain and discomfort and N/V | show 🗑
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| show | NG tube for decompression/stop aspiration, volume replacement, antibitoics, pain meds and surgery
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| What to do in gastric outlet obstruction? | show 🗑
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| show | Epigastric pain
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| Treatment of acute phase of PUD? | show 🗑
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| sudden severe abdominal pain, rigid boardlike abdomen, severe generalized abdominal and shoulder pain, fetal position and grunting respirations, bowel sounds ABSENT | show 🗑
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| show | Dumping syndrome
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| what is more common acute or chronic PUD? | show 🗑
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| Pathology of PUD? | show 🗑
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| show | Gastric ulcer
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| pain is burning or gaseous pain is spnontaineous usually 1-2 hours after meals, food can agravate it? | show 🗑
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| show | Duodenal ulcers
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| Duodenum ulcers involves the duodenum and the? | show 🗑
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| This ulcer pain is often relived by food? | show 🗑
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| show | Does not distinguish between current or past PUD but good still
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| show | PUD
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| show | 6 bland small meals a day, no spicy, alchol, carbonated beverage/caffeine stop smoking dont take NSAIDS/aspirin
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| show | Hemmorhage
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| show | Perforation
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| show | medical emergencies
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| acute ulcers that follow a major physiologic insult | show 🗑
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| dx: for physiologic stress ulcer? | show 🗑
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| prevention of physiologic stress ulcer? | show 🗑
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| show | reduction of gastric acid secretion with PPI or h2 receptor blockers
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| physiologic stress ulcers occurs most often in? | show 🗑
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| show | Acute pancreatitis
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| Acute pancreatis is more common in? | show 🗑
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| race for pancreatis? | show 🗑
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| show | Gallbladder, second is chronic alchol intake
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| show | acute pancretitis
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| a mixture of cholesterol crystals and calcium salts found in patients with acute pancreatis? | show 🗑
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| show | staiss
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| show | Acute pancretitis
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| show | autodigestion of the pancreas
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| Activated trypsin in the __________ can digest the ________ and produce bleeding | show 🗑
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| show | Mild pancreaitis
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| show | Severe pacreatitis
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| Predominant manifestation of acute pancreatitis | show 🗑
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| show | Pain in pancreaitis
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| Where is the pain in pancreaittis? | show 🗑
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| The pain has a steady onset and is described as severe, deep, piercing and continous or steady, pain is agravated by eating and occurs when recumbnent | show 🗑
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| show | Pacereatis
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| show | decreased or absent
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| Paralytic illeuls and crackles of lungs may be present in? | show 🗑
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| show | Pancreatisis
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| show | Grey turners, and the periumbical area is cullens
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| show | Grey turner/cullens, discolorations of abdominal walls
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| Two significant local complications of acute pancreaitis? | show 🗑
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| show | A pancreatic pseudocyt
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| Palpable epigastric pain is evident of? | show 🗑
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| Serum amlysase levls in pancreatits is? | show 🗑
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| Pacreatic pseudocysts can perferoate cuasing | show 🗑
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| show | collection of pus
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| upper abdominal pain, abdominal mass, high fever and leuko cytosis | show 🗑
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| show | pulmonary (pneumonia, ARDS, pleural effusion) and cardiovasculare (hyptension) and tetant thanks to hypocalcemia
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| show | atelectasis
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| show | DIC, thrombi
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| show | serum amylase and serum lipase
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| show | amylase is raised in other disoreders
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| show | pancreatits
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| Goals of tx for acute pancreatits? | show 🗑
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| show | antispasmodics
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| To pevent shock in pacreatis | show 🗑
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| show | NPO, NG suction to reduce vommiting/gastric distention and to prevenet gastric acidic contents from entering the duodenum, PPI'S/antacids
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| In patients with acute necrotizing pancreatitis, what is the leading cause of death? | show 🗑
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| show | none
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| When food is allowed, in aucte pancreatits what is the diet? | show 🗑
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| is frequent vommiting seen in acute pancreatis? | show 🗑
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| major focus of care in acute pancreatits is? | show 🗑
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| show | reduction of fat as the stimulare onacreas, carbs are less stimulating
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| show | Call dr.
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| show | Chronic pancreaitis
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| show | Chronic pnacreatis
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| show | Chronic pancreatis
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| show | inflamation of the sphincter of oddi ascociated with gall stones
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| show | most commobn
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| Chronic pancreatits can be caused by those who abuse? | show 🗑
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| abdominal pain that is heavy gnawing feeling or burning and cramplike, not relived with food, come and goes | show 🗑
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| Malabsotption with weight loss, consitpation, mild jaundice with dark urine, steatorrhea and diabetes melitys are signs of | show 🗑
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| Frothey urine and stool, steatorhea can become severe with vommitius | show 🗑
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| show | Chronic pancreatis
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| show | ERCP
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| Deficiency of fat-soluble vitamins, cobalmin, glucose intelerance and dibetes may be found in | show 🗑
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| show | Diet, pancreatic enzyme replacement and control of diabetes, small bland frequent meals, no alchol or caffeine prescribe bile salts to help with fat absorption
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| show | a meal or snack
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| show | acute pancreatis
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| Cavity continous with or surronding outside of pancreas filled with necrotic produts and liquid secretions | show 🗑
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| show | Pancreatic absess
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| show | acute pancreatits
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| show | severe nutritional defict
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| show | jejunal feeeding tube
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| Two major types of chronic pancreatis? | show 🗑
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| show | billary diseae
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| show | Chronic pancreatits
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| Most common disorder of the bilairy sytem is? | show 🗑
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| show | neck of the gallbladder or in the cystic duct.
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| Inflamation of the gall bladder, this is usually associated with choleithiasis (gall stones) | show 🗑
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| show | asymptomatic
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| Cholecystectomy is? | show 🗑
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| show | Women, multipara and over 40, oral contraceptives, sedetary lifestyle and obesity. asins
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| Cause of gallstones? | show 🗑
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| show | Cholelithiasis
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| Conditions that upset the balance of cholesterol, bile salts and calcium in solution | show 🗑
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| show | Gall stones
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| show | Protein, calcium, bilirumbin and bile salts.
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| Most common gallstone? | show 🗑
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| show | gall stones
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| The gallstones may remain in the gallbladder or migrate to the? | show 🗑
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| Gallstones cause pain as they? | show 🗑
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| What size gallstones are more liekly to produce an obstruction | show 🗑
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| show | bile can still get though
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| Choleosytisi is most commonly associated with? | show 🗑
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| show | Just choleocytis
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| The gallblader is edmeatous and hypermic and may be distended with bile or pus | show 🗑
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| show | spasms may result
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| bilarly colic, pain is often steady, pain can be excurtating and accomponied by tachy , diaphoresis, may last up to an hour | show 🗑
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| When the pain of cholethiasis subsides resuldual tenderness is felt in the? | show 🗑
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| The attacks of pain from gallstones occurs 3-6 hours after? | show 🗑
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| show | Total obstrutction by gall stones related to bilary obstruction
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| indgestionand pain / tendeness in right upper quadrant | show 🗑
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| show | Chronic Chlecytutus
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| Most common complications of choleysitis in older or diabetic | show 🗑
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| Commonly used to dx gallstones? grear id allergic to contrast! | show 🗑
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| Allows visualaztion of the gall bladder , the cystic duct, the common hepatic duct and the common bile duct, bile taken during this is sent for culture. | show 🗑
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| show | Percutaneous transhepatic cholangiography
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| WBC in choleothasis? | show 🗑
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| show | ALT, AST, and alkaline phosphate
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| show | Surgical intervention with cholecystectomy
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| show | pain control, control of possible infection with antibiotics and maintece of FE NG insertion of N/V is severe
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| may be used to drain purluent material from the obstructed gallbladder | show 🗑
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| Treatment of choice for symptomatic cholethiasis | show 🗑
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| show | a week
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| show | Injury to common bile duct
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| most common drrugs used in the treatment of gallbladder disease? | show 🗑
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| show | bilary tract is obstructed
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| show | Small frequent meals with some fat at end of meal to increase gallblader emptying
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| show | Eat nutrion foods avoid fat for 4-6 weeks
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| show | Bilary tract obstruction
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| bleeding may result in cholesthiasis from | show 🗑
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| show | Pancreaitis
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| A common complication postop from laparscopic chlecystectomy is? | show 🗑
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| how help with shoulder pain after a lap choleystectomy? | show 🗑
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| show | avoid exess fat
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| inflamation to mucosa lining or entire wall of gallblader, edmea | show 🗑
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| show | Gallbladder disease
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| in gallbladder disease what is a major complication if not treated? | show 🗑
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| antimetics are often given in? | show 🗑
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| show | can use a lithiotripter to break apart a gallstone
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Created by:
rebo14