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Cardiovascular

TermDefinition
Signs and Symptoms of cadrio Disease Chest/Neck/UE Pain or Discomfort, Palpitations, SOB/Dyspnea, Syncope, Fatigue, Cough, Cyanosis, Claudication
What is Claudication? Symptom of vascular component consisting of edema & LE pain. Peripheral edema hallmark of right ventricular failure Skin discoloration, trophic changes (thin, dry, hairless skin) cool skin = vascular obstruction warm skin = inflammation/infection
Aging of the cardiovascular system THE HEART:Reduction in myocytes & conduction cells, less capillaries, impaired autonomic control of heart rate ARTERIES: Deterioration of walls. Stiffwalls, increased BP = arterial damage (cycle) Lowered functional capacity, esp. during exercise
Exercise and the cardiovascular system Lowers resting HR, Lowers BP, Improves endurance/cardiac efficiency, Reduces incidence of diseases that are risk factors for CV disease,
Gender differences CAD is the leading cause of death in U.S. women, Higher incidence of bleeding with thrombolytics, Estrogen is protective in heart disease, AMI: 1stc/o is often nausea!
Ischemic Heart Disease( CAD/ CHD) Myocardium not receiving adequate 02, Form of above; atheromas & fatty streaks develop. Fibrous plaque, rupture, & thrombosis can result in coronary arteries Arteriosclerosis, Atherosclerosis
Modifiable Risk factors of heart disease Smoking - #1 preventable cause of CAD, HTN, Blood Lipids, Obesity, Diabetes Mellitus, Metabolic Syndrome (“diabesity”)
Sternal precautions No lifting over 8-10 lbs, No push/pull with arms, Walking – HHA best, cane worst, No reaching behind back, Hug pillow during cough/sneeze. Move in the Tube: Goal = Shortened lever arm for load-bearing movements
PT Rehab of Cardic pt. Great research: improved exercise tolerance without adverse outcome, Imperative to MONITOR VITALS!, Angina, N&V, severe dyspnea, pallor, coldmoist skin, HR over 130bpm or under 40.
Beta Blockers Lopressor (metroprolol), Inderal (propranolol),Tenormin (atenolol) Goal: Lower BP....Relax blood vessels in heart; decline in force of contraction and HR. Side effects: Insomnia, nausea, fatigue, bradycardia, weakness
Calcium-Channel Blockers Norvasc (amlodipine), Procardia (nifedipine) Goal: Lower BP......Dilate coronary arteries to lower BP & suppress some arrthymias Side Effects: fluid retention, palpitations, HA, dizziness.
Diuretics Lasix (furosemide), Hydrodiuril (hydrochlorothiazide Goal: Increase excretion of sodium & water to control HTN and fluid retention Side Effects: drowsiness, dehydration, electrolyte imbalances, muscle cramps & fatigue, dizziness.
Angina Cardiac workload exceeds 02 supply to myocardial tissue Chronic stable angina: Classic, predictable, no pain at rest Unstable angina: Nocturnal, Chronic, Resting/Decubitus PT: Reduce activity or rest with stable angina.
Hypertension/HTN Blood Pressure = Force of Blood exerted on arterial walls. Regulated by 2 factors: 1. Blood flow (determined by cardiac output) 2. Peripheral vascular resistance: diameter of vessels & viscosity of blood
Hypertension blood pressure levels 120/80 mm Hg: normal resting adult 130/80 mm Hg: Stage 1 Hypertension 140/90 mm Hg: Stage 2 Hypertension
Orthostatic Hypotension Gravity causes venous pooling in LE therefore a reduction in the pressure of the heart Cause: Prolonged recumbency, Medication side effects, After physical exertion PT: Testing, Pt. ed:rise slowly, ankle pumps, ESSENTIAL to cool-down after exercise.
Myocardial Infarction Etiology Ichemia with resultant necrosis of myocardial tissue 80-90% from atherosclerotic stenosis & coronary thrombus Goal: reperfusion therapy (thrombolytic; IV heparin) within 1sthour.
Treatment of MI Prognosis –60% mortality rate if over the age of 80 Post-MI Complications: Arrhythmias, CHF, pericarditis. Rehab Implications: Gentle mobility begins within 24 hrs, elemetry, may need 02, No aquatics, WP, hot tubs for 6 week.
Congestive Heart Failure Condition where heart is unable to pump sufficient blood to supply the body’s needs Blood backs up into pulmonary veins→ congestion & pulmonary hypertension Left ventricular failure = CHF•Right ventricular failure = cor pulmonale
S/S of CHF Exertional Dyspnea, Fatigue, Muscular weakness, Renal Changes, Right-sided Heart Failure
CHF Rehab Exercise improves functional capacity, reduces symptoms, Early mobilization in acute care, Monitor vitals; watch for return to baseline (≤ 3 min), High Fowler’s position (HOB ↑)
Cardiomyopathy Group of conditions affecting the heart muscle itself; contraction/relaxation of myocardial muscle fibers impaired, Ischemia from CAD most common cause Prognosis & tx. depend on type & severity Cardiac transplant only cure
Types or Cardiomyopathy Dilated Cardiomyopathy• disease (risks include obesity, smoking, ETOH abuse, systemic HTN); or idiopathic Hypertrophic•Genetic; sudden death in young competitive athletes Restrictive•Myocardial fibrosis, hypertrophy
Arrhythmia Disturbance of rate or rhythm caused by abnormal electrical impulse generation by SA node or abnormal conduction of impulses
Arrhythmia Etiology Classified by:•Origin(ventricular or supraventricular)•Pattern(fibrillation or flutter)•or Rate(tachycardia, bradycardia) Cause – Often Multi-factorial•HTN, h/o MI, valvular heart disease, degeneration of conductive tissue
Types of Arrhythmias Atrial Fibrillation – most common chronicarrhythmia•Blood remains in atria & ventricles don’t fill properly Ventricular Fibrillation (v-fib) •Freq. cause of cardiac arrest; must depolarize quickly
Rehab of Arrhythmias Treatment: cardioversion, meds PT: Most arrhythmias asymptomatic, If symptomatic: syncope, dizziness, chest pain, Increased by exercise (& often during recovery) –monitor MRIs & diathermy CONTRAINDICATED
Aneurysm Abnormal dilation in wall of vessel or heart with diameter ≥ 50% than normal Etiology:injury to tunica media (atherosclerosis, trauma) Pathogenesis: plaque erodes vessel wall, layers stretch & weaken
Types of Aneurysm Dissecting:layers of vessel wall separate AAA:below diaphragmatic border most common Cerebral:CVA Peripheral arterial aneurysms: femoral & popliteal arteries
Aneurysm PT implications Detection usually by accident; many asymptomatic; often monitored if under 5cm Rehab: Invasive sx. – high risk for pulmonary complications, Proper lifting techniques, avoid Valsalva
Peripheral Vascular Disease Pathologic condition of vessels supplying blood to the extremities & major organs (intestines & kidneys) Arteries & veins; LEsmore common than UEs PAD is MOST COMMON reason for AMPUTATION
PVD classifications Arterial occlusive – most common (PAD) Inflammatory (vasculitis) Venous (DVT, PE, CVI) Vasomotor (Raynaud’s)
Peripheral Arterial Disease (PAD) Most common arterial occlusive disease Arteriosclerosis Lumen of artery narrows and then is eliminated Progressive; causes ulcers of LEs, feet Risk Factors: •Smoking #1•Others: same as atherosclerosis
PAD: INTERMITTENT CLAUDICATION Intermittent Claudication– Classic PAD-Begins when vessel ~50% narrowed; generally in calves Predictable Resolves with rest PAD graded by distance person can walk before pain
The 5 P's for PAD 1. Pain 2. Pallor 3. Paresthesia 4. Paralysis 5. Pulselessness
Rehab for PAD Progressive conditioning: intermittent exercise to near-maximal pain, rest until pain subsides Wound healing – complicated by DM
CHRONIC VENOUS INSUFFICIENCY (CVI) aka Venous Stasis Damaged valves = ↓venous return = ↑venous pressure & venous stasis Chronic blood pooling = cell damage Risk Factors: •h/o DVT, LE trauma, varicose veins, immobility, ↓nutrition
CVI S/S Chronic leg ulcers (medial malleolus) Edema Thick, coarse skin; brown pigmentation near ankles Dermatitis & cellulitis can develop
CVI PT implications Avoid prolonged sit/stand, leg crossing, Compression hose during day; LE elevation, Avoid tight garments popliteal area, Compression wraps/devices•ABI First! If CHF must monitor closely Skin assessment, wound care•Whirlpool 1-2 sessions MAX
VENOUS THROMBOSIS aka Thrombophlebitis Partial or complete occlusion of a vein by a thrombus with 2º inflammatory reaction in the wall of the vein •Deep vein thrombosis (DVT) most common 90% found in LEs, 10% UEs Proximal DVTs much more likely to become PEs*
Venous Thrombosis Risk Factors Immobility (venous stasis): bed rest, air travel Trauma (venous damage): sx, trauma, childbirth Lifestyle: pregnancy, smoking, OCs Hypercoagulation: neoplasm, hereditary thrombolytic dz Other: DM, obesity, previous DVT, over 60
DVT Rehab/Prevention Prevention•Early mobilization•Prophylactic use of anticoagulants •TED hose post-op patients Rehab Implications•Often 24 hour PT hold s/p DVT•Watch pt’s with anticoagulant therapy for bleeding:•Hematuria, blood in stool, gum bleeding
Skin care for Vessel Disorders Examine regularly, seek immediate assistance with any breaks Avoid dry skin; no lotion between toes, no scratching NO barefoot, prevent blisters Misc.•No razor blades•Avoid caffeine•Avoid crossing LEs
Created by: SamuelD
 

 



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