OPT Allergies Word Scramble
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Term | Definition |
transient lingual papillitis | enlargement of fungiform papilla on dorsal tongue |
localized form of transient lingual papillitis | one enlarged fungiform papilla on anterior tongue |
diffuse form of transient lingual papillitis | many enlarged, painful fungiform papilla; sometimes with fever and lymphadenopathy |
'papulokeratotic variant' of transient lingual papillits | diffuse and asymptomatic enlargement of fungiform papillae; most likely frictional hyperkeratosis |
recurrent apthous stomatitis/ulceration (RAS/RAU) | oral ulcers mediated by CD8 T-cells |
1. antigenic stimulus 2. immunodysregulation 3. decreased mucosal barrier | 3 pathologic mechanisms of apthous stomatitis |
simple apthosis | most common type of RAS; self-limiting without other disease processes |
complex apthosis | patients that have several apthous ulcers with significant disease and recurrences |
minor apthous stomatitis | mildest form of RAU; a couple of outbreaks each year, usually in adolescents |
major apthous stomatitis | large ulcers with long healing time and a later onset |
herpetiform apthous stomatitis | numerous small ulcers like herpes but not on gingiva or systemic symptoms |
Behcet syndrome | systemic vasculitis with irregular oral ulcers on soft palate, genital ulcers, and ocular problems |
sarcoidosis | multisystem granulomatous disorder, probably autoimmune; lungs and lymphoid tissues are most affected -oral involvement of mass or papules is seen first in 2/3 of patients |
Heerfordt syndrome | acute form of sarcoidosis; parotid gland enlargement, facial nerve paralysis, uveitis and fever |
orofacial granulomatosis | variety of clinical presentations that show non-specific granulomatous inflammation; called Melkersson-Rosenthal syndrome when with facial paralysis and fissured tongue |
Wegener granulomatosis | necrotizing granulomatosis lesions of respiratory tract; effects in kidneys and vasculitis |
classic/generalized Wegener granulomatosis | fatal kidney involvement after initial respiratory infection |
limited Wegener granulomatosis | disease stays localized to respiratory tract without renal lesions |
'superficial' Wegener granulomatosis | only have skin and oral mucosal lesions |
strawberry gingivitis | early manifestation of Wegener granulomatosis |
PR3-ANCA | antibodies detected in 90% of patients with Wegener's and can be used to monitor disease activity |
stomatitis medicamentosa | mucosal reactions to systemic drugs |
anaphylactic stomatitis | IgE mediated erythema and ulceration 24 hours after exposure to drug |
fixed drug eruptions | reactions that reappear at same site after taking drug |
lichenoid drug reactions | mucosal changes from systemic drug that mimic lichen planus |
pemphigus-like drug reactions | mucosal desquamation and bullae formation like pemphigus |
pemphigoid-like drug reactions | mucosa comes off in sheets and mimics pemphigoid |
lupus erythematous-like drug reactions | fever, flu-like symptoms and lupus-like changes |
nonspecific vesiculoerosive or apthous-like lesions | nonspecific mucosal changes due to drug reaction |
allergic contact stomatitis | long list of agents that cause hypersensitivity reaction; {from artificial cinnamon flavoring} |
perioral dermatitis | inflammatory reaction around mouth from antigen or corticosteroid cream |
lichenoid contact reaction to dental restorative materials | reaction adjacent to old, corroding amalgam fillings |
angioedema | diffuse edematous swelling of soft tissue; minor trauma can precipitate reaction |
IgE-mediated type I hypersensitivity reactions | most common cause of angioedema; mast cells degranulate and histamine is released |
angiotensin-converting enzyme (ACE) inhibitors | drug used to treat hypertension that also causes angioedema due to increased levels of {bradykinin} |
angiotensin II receptor blockers | drugs developed to avoid inhibition of bradykinin breakdown and angioedema |
activation of complement pathway | mechanism of causing angioedema; can be hereditary or acquired |
type I complement pathway | inherited from decreased {C1 esterase inhibitor} that enhances complement pathway |
Type II complement pathway | normal C1-INH levels but enzyme does not work |
acquired type of C1-INH deficiency | can be seen in lymphoproliferative diseases that inhibit C1-INH |
Created by:
aharnold
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