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H&P Ii-Spring 2012

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Question
Answer
What does the CNS include   brain, spinal cord  
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how many nerves in the peripheral nervous system   12 cranial, 31 peripheral n. originating at cord  
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where are the nurona cell bodies   gray matter  
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what is the greatest mass of brain tissue   cerebrum  
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what is in the white matter   neuronal axons, coated w/ myelin to allow n. impulses to travel quickly  
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what are the lobes inthe cerebral hemisphere   frontal, parietal, occipital, temporal  
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4 regions of the cerebrum   cerebral cortex, diencephalon, cerebellum, brainstem  
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what is the diencephalon comprised of   basal ganglia, thalamus, hypothalamus  
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Processes sensory input and relays to the cerebral cortex   thalamus  
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responsible for movement   basal ganglia  
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4 fxns of the hypothalmus   homeostasis, reulates B/P, HR, temp, emotion like anger and sex drive, hypothalmic hormones act directly on the pit  
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white matter where all parets of the cerbral corte descend into the brainstem   internal capsule  
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what does consciousness depend on   interaction b/w cerebral hemisheres and the reticular activating system  
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3 sections of the brainstem   midbrain, pons, medulla  
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coordinates all movement   cerebellum helps maintin body in upright position  
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LP usually done where   b/w L3-5  
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efferent fibers sending motor signals aways from the cord   anterior nerve roots  
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afferent fibers receiving sensory signals from periphery   posterior nerve roots  
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n. roots converge to form combination nerves where most peripheral n. originate   spinal nerves  
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where are deep tendon reflexes mediated   through spinal nerves  
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how can we touch nose with our eyes closed   cerebellum  
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what mediates voluntary movement and intergreates complicated/delicate movments   corticospinal (pyramidal) tracts  
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what is the anatomical arangement of the pyramidal tracts   originates in motor cortex of brain and travels down the medulla where they form a pyramid and cross to contralateral side of medulla  
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where do upper motor neurons synapse for cranial and peripheral nerves   cranial: brainstem, per: spinal cord  
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where are the cell bodies for lower motor neurons   in the anterior horn of the cord  
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where are UMN and LMN divided   pyramids  
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signs for UMN and LMN lesions   UMP: msucle tone and DTR's are exaggerated, LMN: muscle one and DTRs are decreased or abscent  
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in the corticospinal tract if there is a lesion, what happens   fxns are lost below level of injury  
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helps to maintain muscle tone and control gross body movements like walking   basal ganglia  
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disease in the basal ganglia show what   increased muscle tone, posture an gait disturbances (bradykinesia) involuntary movements: tremor (PD)  
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coordinates motor activity, maintains equilibrium and helps to control posture   cerebellar system  
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damage in the cerebellar system shows what   impaired coordination, gait and equilibrium and causes decreased muscle tone  
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Two sensory tracts   spinothalamic tract, posterior colums  
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crude touch, pain an temperature pass where and synapse where   spinothalamic, synapse w/ secondary sensory neurons  
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where does the spinothalamic tract go   to the thalamus  
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what do the posterior columns sense   position, vibration, fine discriminatng touch: travel to medulla  
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where do the posterior columns cross   at the medulla  
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where do the spinothalamic tract neurons cross   at the level of the nerve  
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a lesion that won't impair perception of pain, touch but will impair finer discrimination   sensory CORTEX lesion (3rd set of sensory neurons)  
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a lesion that will impair position and viration but other sensations are preserved   posterior columns  
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loss of sensation in the legs along w/ paralysis and hyperactive reflexes inicates what   cord transection lesion  
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band of skin innervated by sensory root of a single peripheral sinal nerve   dermatome  
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Grading ot DTR's   0-4 0: absent 4: hyperactive w/ clonus 2: average, normal  
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What are the DTR's and their corresponding spinal nerves   achilles: s1, patellar L234 Brachioradialis C56, biceps C56, Triceps C6,7 Abdoment T9-10 and T10-12 Babinski L5-S1, Anal wink S2,3,4  
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what does hyperactive reflexes suggest   UMN lesion, confirmed w/ clonus  
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involuntary, rhythmic muscle contractions in response to a sudden stretching of that muscle   clonus  
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diminished reflexes and decreased sensation suggusts what   cord segment damage, muscle damage  
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is lightheadedness vertigo?   no must differentiate that and syncope  
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medical conditions that might cause lightheadedness   hypotension, arrhythmia, hypoglycemia, vasovagal stimulation, medications  
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causes of hemiplegic weakness   CVA/TIA  
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cause of focal weakness   ischemic, vascular, mass lesions in the NS or peripheral nervous system or MS disorders  
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bilateral proximal weakness suggests   myopathy  
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bilateral distal weakness suggests   polyneuropathy  
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weakness made worse w/ repeated effort and improved w/ rest suggests   myasthenia gravis  
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numbness/weakness in feet spreading proximally sggests   guillain-Barre syndrome  
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tingling and numbness in hands, feet and peri-oral areas suggests   hyperventilation  
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3rd leading cause of death in the U.S.   cerebrovascular dz  
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best tx for stroke   prevent RF's HTN, BM, obesity, alcohol, dyslipidemia, smoking, sedentary, afib  
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when is the highest risk of stroke post TIA   next 30 days  
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what are cranial nerves   peripheral nerves that originate within the cranium  
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where do cranial nerves synapse   with motor neurons in the brainstem  
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where do cranial nerves arise from   dienceephalon and brainstem  
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what CN control the eye movemets   LR6SO43  
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what muscles does the trigeminal n. innervate   masseter and temporalis  
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how do we assess the facial n.   forehead wrinkling, eyelid closure, whistle/pucker  
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what nerves controls swallowing, rise of soft palate, and gag reflex   glossopharyngeal, vagus  
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what does the spinal accessory innervate   sternocleidomastoid and trapezius  
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if the tongue protrudes to the left where is the lesion (CN?)   hypoglossal and toward the affected sign  
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causes of muscle atrophy   motor neuron dz, peripheral n. compression, RA, protein-calorie malnutrition  
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what is muscle tone   the slight residual tension when reflexxed  
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what is decreased tone called   flaccidity  
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increased tone at the extremes of range   spasticity  
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persistent increased tone throughtout the ROM   lead-pipe rigidity  
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impared strength or weakness   paresis  
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absence of strength or paralysis   plegia  
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tests to wein out subtle weaknesses   heel walk, toe walk, getting out of chair, deep knee bend  
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strengh grading   0-5 3: movement against gravity  
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when does clonus present   in CNS dzs  
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inability to hold wrists and fingers in extension   asterixsis (hepatic encephalopathy-alcoholic)  
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causes of scapular winging   werratus ant. weakness d/t long thoracic n. palsy  
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4 parts of coordination   motor, cerebellar, vestibular system, sensory  
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what do clumsy movements like pronating and supinating poorly suggest   cerebellar dz, not so much basal ganglia or UMN  
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what is cerebellar dz   dysdiadochokinesis  
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doing touch finger to nose test that presents worse w/ closing eyes most likely   cerebellar or vestibular dz  
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heel toe walking may wein out what   distal msucle weakness, and corticospinal tract weakness  
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hopping in place on one leg tests   proximal and distal muscle trength, position sense and cerebellar fxn  
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romberg sign   standing then closing eyes: worse balance, tests cerebellar fxn and proprioception  
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+ pronator drift test suggests   lesions in contralateral cortoospinal tract  
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+ for tapped down and fail to return   propriopecption: posterior columns  
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if affected arm overshoots the resting position w/ tap down test   cerebellar dysfunction  
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sensation of pain and temp   spinothalamic  
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sensation of position and vibration   posterior columns  
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light touch   both spinothalamic and posterior columns  
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some disciminative sensations invovles   post spinothalamic, post. and also sensory cortex  
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innervation to each of these areas: shoulders, inner/outer aspect of forearms, thumbs and am fingers, ant thighs, medial/lat aspect of calves, small toes, medial aspect of each buttock   S: C4 I: C6 O: T1 Thumbs: C6 F: C8, Ant.: L2, Med: L4, Lat: L5, Toes: S1, Med butt: S3  
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identifying an object   sterognosia  
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blunt opject or write on their palm a number   graphesthesia  
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worries persisting over a 6m period suggests   generalzed anxiety d/o  
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recurrenty panic attacks followed by periods of anxiety over a futer attack   panic d/o  
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intrusive thoughts and ritualistic behaviors   obsessive-compulisve d/o  
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when are facial expressions masked   parkinson's and apathetic in depression  
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three componants to the GCS   eye opening, vebal, motor  
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defective artciulation/talking   dysarthria  
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d/o of reception or expression of speech   aphasia (can be confused w/ psychosis)  
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phrse substituted for a word that can't be recalled   circumlocutions (what you write with) for pen  
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malformed, wrong, or invented words   paraphasia (i write w/ a den, bar, dar)  
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location and function of wernicke's area   receptive area, understanding language: posterior superior temporal lobe  
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location and fxn of brocas area   expressive, or forming words, posterior inferior frontal lboe  
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misinterpretations of real external stimuli   illusions  
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perceptions in the abscence of real stimuli   hallucinations  
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what does serial 7s or spelling WORLD backwards asses   attention  
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what is remote memory (birthdays anniversarys) impaired by   late dementia  
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what is recent membory impaired by   dementia and delirum  
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what is new learning ability (giving 3 new words to remember) impaired by   dementia, delirium, depression or anxiety  
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how can we asses b/w mild MR and dementia   informationand vocabulary, current public officers or state capitals (high cog. fxn)  
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what is calculating ability often impaired by   dementia, aphasia, limited education  
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how do we test abstract thinking?   proverbs: don't count cxns b4 they hatch: concrete responses in delirium and dementia, bizarre interpretations in schizohrenia  
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what is contrstructional ability effected by   dementia, parietal lobe dysfxn, poor vision  
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what 5 things should we ask the pt about their vision   an pain, changes, redness, discharge, trauma  
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pain w/o redness ddx   acute glaucoma, retinal hemorrage,  
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where does conjunctivitis occur   over the sclera and conjuctiva not the cornea  
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how do we asses the pupil   round and smooth borders 3-4mm  
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slow central VL   cataracts, mac degeneration  
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swiss cheese vision loss dark spots   macular degeneration  
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3 acute causes of vision loss   rtinal detachment, vitreous hemorrhage, central retinal a. occlusion  
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gradual causes of VL   mac degen, open-angle glaucoma,  
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mc causes of hemianopsia   stroke, brain tumor, trauma  
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moving specks or strans indicate   vitreous floaters  
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slow peripheral VL   open  
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fixed defects (scotomas)   lesions in retina or visual pathways  
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flashing lights or new vitrous floaters suggests   retinal or vitreous detachment  
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douple vision, causes   diplopia, brainstem or cerebellum lesions, weakness or paralysis of CN III or VI in horizontal, CN III or IV in vertical  
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diplopia in one eye with the other closed suggests   pathology in cornea or lens  
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what VA is U.S> legal blindness   20/200  
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partial blindthess in the visual fields of one or both eyes   hemianopsia  
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class of hemianopsia but confined to visual field quadrants   quadrantanopsia  
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implies presence of same defect in both eyes - homonymous    
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5 step optic pathway   eye, optic nerve, optic chiasm, optic tract, lateral genticulate nucleus  
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lesion on one retina   vision completely lost in one eye  
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lesion on the optic nerve L side   lose Right vision of L eye  
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lesion at optic chiasms   lost left vision in left eye and R vision in right eye (peripheral)  
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lesion in the optic tract on L side   loss of right vision both eyes  
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lesion in occipital lobe inL side   loss of R vision both eyes  
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where is the physiologic blind spot   15 degrees temporal to the central line of gaze  
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what causes optic disc. enlargement   papilledema, glaucoma, optic neuritis  
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bilateral disc enlargement   metabolic or bilogical cause, unilateral: space occupying lesions  
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prtorusion of the eye   exopthalmos d/t lesions or hyperthyroidism  
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nl corneal convexity in whites and black   W: 20mm B: 22mm  
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thickened conjunciva that may encroach nasal to temporal   pterygium  
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what do we asses the cornea and lense for   arcus, cataracts, scars, and pterygium  
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puckering of pupillary muscles suggests   increase pressure in ant chamber  
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bowing of cornea w/ slit lamp exam   glaucoma  
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unequal size of pupils   aniscoia ~20% population have it <.5mm is nl  
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how do we say assessment of pupils   equal round reactive to light and accomidation  
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where is nl light reflex   symmetrical and nasal to center of pupils  
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nystagumus   more than a few beats of lateral nystagmus is abnormal  
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what happens with the lid in hyperthyroidism   lid lag an poor convergence  
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CI's to dilate pupils   head injury, suspicion of narrow angle glaucoma  
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what will decrease the red reflex   cataracts, tumor/lesion  
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nl color of disc   yellow-orange  
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nl disc ratio   1:2  
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what will papilledema cause   increased cup:disc ration w/ blurry margins  
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bates 261-267 retinal patholgies    
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how do we inspect anterior structures   increasing diopters to +10 or 12  
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