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bacteria

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Question
Answer
Enterobacteriaceae gram   Gram-negative rods  
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Enterobacteriaceae oxidase and catalase   Oxidase-negative, Catalase-positive  
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Facultative anaerobes   Enterobacteriaceae  
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Ferment glucose, reduce nitrate to nitrite   Enterobacteriaceae  
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Most are motile by peritrichous flagella   Enterobacteriaceae  
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Enteric bacteria which tolerate bile salts in MacConkey agar   Enterobacteriaceae  
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Variety of clinical infections   Enterobacteriaceae  
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E. coli Salmonella Yersenia   Major enteric and systemic pathogens  
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Proteus Enterobacter Klebsiella othe   Opportunistic pathogens  
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Found in the intestinal tract of animals and humans   Enterobacteriaceae  
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Contaminate vegetation, soil and water   Enterobacteriaceae  
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Major pathogens   E. coli, Salmonella sp. and Yersinia sp  
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are involved in localized infections in diverse anatomical locations   Opportunistic pathogens  
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Gram-negative, short rods   E. coli  
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Most strains are motile by peritrichous flagella   E. coli  
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Often fimbriate   E. coli  
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A capsule is often present --- mucoid   E. coli  
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Grows well on a variety of media at 37 oC   E. coli  
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Characteristic growth on EMB (metallic sheen   E. coli  
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  E. coli  
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on-spore-forming   E. coli  
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Ferments lactose (pink colonies in MacConkey agar   E. coli  
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E. coli are serotyped on the basis of   lipopolysaccharide  
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lipopolysaccharide   “O” (Somatic), “H” (Flagellar) and “K” (Capsular  
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possesses non-flagellar appendages called pilli.   E. coli  
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pilli. Important types   K88 or F4, K99 or F5, and 987P or F6  
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are almost always associated with isolates from swine   K88 and 987P  
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associated with isolates from cattle, sheep, swine   K99  
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Occurs due to the colonization of the intestinal tract from environmental sources, shortly after birth   Colibacillosis in mammals  
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Colibacillosis as a primary infection   by shell penetration, inhalation in the hatchery & occurs during the first few days of age  
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Colibacillosis as a secondary infection   complicating agent during the growing period  
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Occurs due to the colonization of the intestinal tract from environmental sources, shortly after birth   Colibacillosis in mammals  
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the mechanism of pathogenesis   Based on (1) Tissue localization of E.coli and (2) Biological activity of E.coli toxin  
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Enterotoxigenic E.coli (ETEC) strain that causes   Enteric colibacillosis and Enterotoxemic colibacillosis  
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strain that cause Local invasive colibacillosis   Enteropathogenic E.coli (EPEC)  
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strain that cause Septicemic colibacillosis   Enteroinvasive E.coli (EIEC)  
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strain that cause Hemorrhagic Uremic Syndrome (HUS) in children   Enterohemorrhagic E.coli (EHEC)  
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Causes diarrhea in animals 2 weeks to 1 month of age   Enteric colibacillosis caused by (ETEC  
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Produce Enterotoxins (Exotoxins): 1. Heat-labile (LT) type (Immunogenic) and 2. Heat-stable (ST) type (Non-immunogenic)   Enteric colibacillosis caused by (ETEC  
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Produce Pilus antigens (K antigens), important for adherence & colonization   Enteric colibacillosis caused by (ETEC  
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Causes Neonatal diarrhea in animals less than 1 week of age   Enterotoxemic colibacillosis caused by (ETEC  
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Produce Enterotoxins (Exotoxins): 1. Heat-labile (LT) type (Immunogenic) and 2. Heat-stable (ST) type (Non-immunogenic   Enterotoxemic colibacillosis caused by (ETEC  
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Produce pilus antigens (K antigens   Enterotoxemic colibacillosis caused by (ETEC  
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K antigens involved in Neonatal diarrhea: K88 (piglets) and K99 (calves   Enterotoxemic colibacillosis caused by (ETEC  
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There is absorption of toxins   Enterotoxemic colibacillosis caused by (ETEC  
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Causes Local invasive colibacillosis   Enteropathogenic Colibacillosis caused by (EPEC  
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Local invasion and destruction of intestinal epithelium by E.coli ( invade beyond epithelium to the lamina propria   Enteropathogenic Colibacillosis caused by (EPEC  
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Not enterotoxigenic (do not produce enterotoxins) and do not become bacteremic or septicemic (do not produce endotoxin)   Enteropathogenic Colibacillosis caused by (EPEC  
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Diarrhea is associated with colonization, attachment and destruction of microvilli   Enteropathogenic Colibacillosis caused by (EPEC  
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Mechanism of invasion not known   Enteropathogenic Colibacillosis caused by (EPEC  
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Associated with bacteremia or septicemia   Septicemic colibacillosis (Colisepticemia) caused by (EIEC  
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Endotoxin-mediated   Septicemic colibacillosis (Colisepticemia) caused by (EIEC  
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May or may not have diarrhea or intestinal lesions   Septicemic colibacillosis (Colisepticemia) caused by (EIEC  
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Enters thru respiratory or intestinal tract   Septicemic colibacillosis (Colisepticemia) caused by (EIEC  
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Multiply in blood or tissue   Septicemic colibacillosis (Colisepticemia) caused by (EIEC  
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Fibrinopurulent systemic lesions in different organs such as pericardium, liver and heart   Septicemic colibacillosis (Colisepticemia) caused by (EIEC  
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are present in E. coli similar to many other Gram- negative bacteria   Endotoxins  
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They are part of the outer layer of the cell wall. Embedded in the outer membrane of the cell   Endotoxins  
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It is a complex phospholipids-polysaccharide-protein macromolecule   Endotoxins  
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Endotoxins are released in soluble form during bacterial growth and liberated when bacteria lyse   Endotoxins  
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They are less toxic than exotoxins   Endotoxins  
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Pyrogenic   Endotoxins  
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Causes leucopenia, hypotension   Endotoxins  
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Complement activation   Endotoxins  
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Intravascular coagulation   Endotoxins  
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Death   Endotoxins  
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also produce EXOTOXINS   Certain strains of E. coli (ETEC  
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Certain strains of E. coli (ETEC) also produce EXOTOXINS Two types   Heat-labile and Heat-stable exotoxin  
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Large immunogenic portion   Heat labile type  
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Non-immunogenic   Heat stable type  
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These exotoxins are produced in the intestines   ENTEROTOXINS  
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They attach to different receptors on the intestinal epithelium   ENTEROTOXINS  
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ENTEROTOXINS activate adenylate cyclase which results in increased   cAMP  
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The increased cAMP causes   hyper secretion of water and chlorides into the gut lumen resulting in fluid loss  
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Very soon after birth a neonate ingests   E. coli  
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may inhibit the sudden and abnormal rate of multiplication of these organisms in the intestines   Colostrum  
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should receive 50 ml to 80 ml (or 5% body weight) colostrum/kg body weight within the first 12 hours of birth. Repeat 18 to 20 hours   Calves  
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can be frozen for several months, with almost no deterioration   Colostrum  
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Thaw in lukewarm water before you use   frozen Colostrum  
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Occurs in calves under 2 weeks but has been seen in calves up to a month old   Enteric colibacillosis (ETEC): E.coli with K99 pili  
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Typically occurs in calves 4 to 5 days old   Septicemic colibacillosis or colisepticemia (EIEC  
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Excess fluid in the intestineDiarrhea for several daysMucus present   Enteric colibacillosis (ETEC): E.coli with K99 pili  
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Septicemic colibacillosis or colisepticemia (EIEC   E. coli infections in Cattle  
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there is no scouring. In most acute cases there may be no temperature as the septicemia is overwhelming   In acute colisepticemia  
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is associated with cases of acute mastitis bovine   E. coli  
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Usually associated with poor sanitation   Bovine Mastitis  
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One or more quarters of the udder become swollen and painful   Bovine Mastitis  
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High temperature, 103 to 108 oF   Bovine Mastitis  
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Milk production falls rapidly and may cease   Bovine Mastitis  
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Vaccines usually contains E.coli, Streptococcus sp. & Staphylococcus sp   Bovine Mastitis  
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Pigs are susceptible to disease during the first 14 weeks or so after birth   E. coli  
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E. coli infection in Pigs Various names have been given to these conditions according to the   age, symptoms and lesions  
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1 to 12 days of age   Neonatal colibacillosis/Piglet scours  
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Diarrhea, dehydration with high mortality - 70%   Neonatal colibacillosis/Piglet scours  
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Edema disease (Post-weaning colibacillosis   E. coli enterotoxemia (ETEC  
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Occurs at about 1 week after weaning   Edema disease (Post-weaning colibacillosis) E. coli enterotoxemia (ETEC  
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An acute, highly fatal neurological disorder   Edema disease (Post-weaning colibacillosis) E. coli enterotoxemia (ETEC  
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The disease is dependent upon colonization of small intestine by E. coli that produces a toxin   Edema disease (Post-weaning colibacillosis) E. coli enterotoxemia (ETEC  
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staggering gait, muscular tremors & spasms, edema of eyelids, subcutaneous sub-serosal edema   Edema disease (Post-weaning colibacillosis) E. coli enterotoxemia (ETEC  
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All edema producing E. coli produce hemolysin and have K88 pili antigens   Edema disease (Post-weaning colibacillosis) E. coli enterotoxemia (ETEC  
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Toxin causes arterial degeneration and increased vascular permeability   Edema disease (Post-weaning colibacillosis) E. coli enterotoxemia (ETEC  
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Yolk sac of embryos is the center of infection   E.coli infection in Poultry  
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Occurs in all types and age groups of poultry   E. coli infection  
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Associated with dusty litter   Airsacculitis  
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Navel infection   Omphalitis  
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A variety of syndromes from which E. coli has been isolated   Enteritis Coligranuloma Synovitis and arthritis  
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A new disease of racing greyhounds ” caused by O157:H7 strain   Alabama Rot  
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Lactose fermenter, non hemolytic   Enterobacter  
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Found widely in nature   Enterobacter  
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is the species of veterinary importance   Enterobacter aerogenes  
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They are opportunistic pathogens   Enterobacter  
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can be associated with mastitis in cows and sows   Enterobacter aerogenes  
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Lactose fermenter, non hemolytic   Klebsiella  
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Opportunistic pathogenHas a large polysaccharide capsule   Klebsiella  
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Associated with mastitis in cattle, cervicitis and endometritis in mares   Klebsiella  
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pneumonia in calves and foals and urinary tract infections in dogs   Klebsiella  
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Lactose non-fermenter   Proteus  
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Motile, characteristic swarming on blood agar and non- hemolytic   Proteus  
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Hydrolyze urea. Deaminate phenylalanine and produce H2S   Proteus  
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Otitis externa in dogs   Proteus  
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are the species of veterinary importance.   Proteus mirabilis and Proteus vulgaris  
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in dogs and horses Urinary tract infections are frequently caused by   Proteus  
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Gram negative rods, oxidase negative   Klebsiella  
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Blood agar: Large, wet mucoid, whitish-grey   Klebsiella  
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MacConkey agar: Pink, slimy coalescing, not surrounded by red haze (D/D: E.coli   Klebsiella  
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Metritis and cervicitis in mares   K. pneumoniae  
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Pneumonia and suppurative lesions in calves and foals; Mastitis in cows on wood shavings and sawdust; Urinary tract infections in dogs   Klebsiella  
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Treatment: Amoxicillin-Clavulanate, enrofloxacin, tetracycline, trimethoprim-sulfonamide   Klebsiella  
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Susceptibility test recommended   Klebsiella  
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Gram negative coccobacilli, non-hemolytic, slow growth in MacConkey   Yersenia  
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are important human and animal pathogens   Y. enterocolitica, Y. pestis and Y. pseudotuberculosis  
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causes enteric red-mouth of fish & infection usually results in hemorrhagic septicemia   Y. ruckeri  
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causative agent of human plague. (Cats are infected most frequently than other domestic animals --- source of infection to humans   Y. pestis  
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human enteric pathogen   Y. enterocolitica  
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enteric (wild & domestic animals & septicemic (cage birds & laboratory rodents   Y. pseudotuberculosis  
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