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strongyles & trichostrongyles

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Answer
2 teeth in buccal capsule, most pathogenic, causes acute verminous arteritis, cause of colic   S. vulgaris  
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no teeth   S. edentatus  
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3 teeth in buccal capsule   S. equinus  
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– L3’s penetrate the mucosa of the cecum/colon→molt to L4 in the liver and migrate to the peritoneum→molt to L5 in the intestinal wall   S. edentatus  
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L4’s molt in the submucosa of the cecum/colon→migrate to the liver and L5’s molt in the intestinal wall/pancreas.   S. equinus  
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Life cycle - Direct and non-migratory, hypobiosis   Small strongyles (30+ species)  
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Pathology - mucosal lesions during larval encystment and re-emergence contributes to colic (abdominal distress   Small Strongyles  
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LEAF CROWN & TEETHIN BUCCAL CAPSULE   SMALL STRONGYLE  
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Parasiticide resistance   heritable mutation permits survival  
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resistance to all drugs within that action class (e.g., benzimidazoles)   Side resistance  
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Low-dose daily feeding of pyrantel tartrate may lead to resistance of   small strongyles  
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may promote the selection of resistance genes (against pryantel pamoate & oxibendazole)   The mode of administration  
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To slow or stop anthelmintic resistance   administer when actually needed  
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Swine Nodular worm   Oesophagostomum dentatum  
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Common in adults pigs   Oesophagostomum dentatum  
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reduced litter size   “Thin sow” syndrome  
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Nodular worm of cattle (O. radiatum   Oesophagostomum spp  
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May cause diarrhea   Oesophagostomum spp  
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Nodules are seen in the walls of small and large intestines.   Oesophagostomum spp  
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Adults in Large intestine   Oesophagostomum spp  
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sheep and goats)   O. venulosum, O. columbianum  
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Swine Kidney worm   Stephanurus dentatum  
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Complex life cycle, including larval migration through liver   Stephanurus dentatum  
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Adults have “mottled” appearance   Stephanurus dentatum  
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Condemnation of liver at slaughter   Stephanurus dentatum  
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Gapeworm   Syngamus trachea  
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Adults are “in permanent copula   Syngamus trachea  
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Form a “Y” shape   Syngamus trachea  
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Reside in trachea/upper bronchi of avian species   Syngamus trachea  
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Pathology – dyspnea, suffocation & death   Syngamus trachea  
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Brown or Western Stomach Worm   Ostertagia (Teladorsagia)  
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cattle   O. ostertagia  
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sheep and goats   T. circumcinta  
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larval invasion of gastric glands (2 main types of disease):   Ostertagia (Teladorsagia)  
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weanling calves in first grazing season are target animals (late spring); diarrhea and weight loss (low mortality)   Type 1  
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caused by rapid emergence of hypobiotic larvae, yearling calves enter second grazing season (late winter); may be high mortality   Type 2  
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Barber Pole Worm   Haemonchus  
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Most severe in sheep   Haemonchus  
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Adults have small buccal capsule w/lancet to pierce mucosa and suck blood   Haemonchus  
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Gut - uterus and ovaries wind around blood-filled gut resembling a barber pole   Haemonchus  
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Direct life cycle   Haemonchus  
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Hypobiosis - important for larvae to over-winter in host and reemerge when conditions are better   Haemonchus  
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acute haemonchosis (10,000+ worms) in young causes anemia & bottle jaw.   Haemonchus  
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Chronic haemonchosis (100-1000 worms)   Haemonchus  
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Use a scale to measure blood loss (anemia) in sheep & goats   FAMACHA  
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Selective rather than whole-herd treatment   FAMACHA  
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Designed to delay the development of resistance in an unselected population (“refugia” or wild gene pool) which have a pool of sensitive genes.   FAMACHA  
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Thin-necked intestinal worm   Nematodirus  
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Hosts: cattle and sheep   Nematodirus  
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Acute diarrhea in young animals   Nematodirus  
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Eggs – LARGER than typical trichostrongyle egg   Nematodirus  
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Cattle Bankrupt Worm   Cooperia  
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Hosts: cattle, sheep & goats   Cooperia  
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Not highly pathogenic   Cooperia  
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May cause some diarrhea, anorexia & depressed growth   Cooperia  
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Resides in the small intestine   Cooperia  
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Very common species   Trichostronglylus axei  
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Can cross-infect between ruminants, horses, and swine   Trichostronglylus axei  
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Causes gastroenteritis in young animals (“black scours”)   Trichostronglylus axei  
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“Red stomach worm”   Hyostrongylus  
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Larvated eggs found in “fresh” feces   Hyostrongylus  
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Direct life cycle; no migration of larvae   Hyostrongylus  
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Larvae molt in the gastric mucosa   Hyostrongylus  
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Clinical signs: anemia, poor appetite, stomach nodules & ulceration   Hyostrongylus  
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