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NPTE Neuromuscular

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
Inflammation of membranes of SC or brain   Meningitis  
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Which meningitis is usually sicker with more rapid time course   Bacterial  
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Infection and inflammation of brain   Encephalitis  
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S/S of brain abscess   HA, fever, brainstem compression, Focal signs in CNII & CNVI  
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AIDS dementia syndrome (ADC)   confusion, memory loss, disorientation, ataxia, weak, tremor, hypersensitivity, pain, sensory loss  
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Formation of blood clot or thrombus within cerebral arteries or branches   Cerebral thrombosis  
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Traveling bits of matter that produce occlusion & infarction in cerebral arteries   Cerebral embolism  
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Abnormal bleeding as a result of rupture of blood vessel   Cerebral hemorrhage  
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Risk factors for CVA   atherosclerosis, HTN, CD, DM2, TIA  
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Lack of oxygen to brain   Cerebral anoxia  
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Irreversible cellular damage to brain   Cerebral infarction  
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Accumulation of fluids within brain   Cerebral edema  
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ICA syndromes include   ACA syndrome and MCA syndrome  
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ACA supplies   Supplies anterior 2/3 of medial cerebral cortex.  
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ACA syndrome   CL sensory loss, CL hemiparesis, leg > arm.  
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Occlusions proximal to anterior communicating artery produce   minimal deficits due to Circle of Willis  
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MCA supplies   lateral cerebral cortex, basal ganglia, internal capsule  
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MCA syndrome   CL sensory loss, CL hemiparesis arm>leg, Broca’s aphasia, perceptual dysfunction, homonymous hemianopsia, CL los of conjugate gaze, sensory ataxia  
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Medial medullary syndrome   VBA occlusion – IL tongue paralysis, CL paralysis of arm and leg, decr sensation  
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Lateral medullary syndrome (Wallenberg’s)   IL cerebellar, Horner’s syndrome, dysphagia, impaired speech, decr gag reflex, IL arm/trunk/leg sensory loss, CL pain/temp loss  
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Horner’s syndrome   miosis, ptosis, decr sweating  
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Basilar artery syndrome   brain stem S/S and PCA s/s. Locked in syndrome  
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Locked in syndrome   basilar artery occlusion at level of pons. Preserved consciousness but quadriplegia, anarthria.  
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Medial inferior Pontine syndrome   IL symptoms: cerebellar, conjugate gaze paralysis, diplopia. CL symptoms: hemiparesis, decr sensation.  
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Lateral inferior pontine syndrome   IL: cerebellar, facial paralysis, conjugate gaze paralysis, deafness, tinnitus. CL: pain/temp sensation  
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PCA syndrome   CL homonymous hemianopsia, CL sensory loss, thalamic syndrome, involuntary mvmts, CL transient hemiparesis, Weber’s syndrome, visual s/s.  
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Weber’s syndrome   oculomotor nerve palsy with CL hemiplegia  
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Lesions of parieto-occipital cortex of dominant hemisphere lead to   aphasia  
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Lesions of parietal lobe of non-dominant hemisphere lead to   perceptual deficits  
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Pt with lesion of L hemisphere (R hemi) tend to be   slow, cautious, hesitant, insecure  
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Pt with lesion of R hemisphere (L hemi) tend to be   impulsive, quick, indifferent, poor judgement, overestimate abilities, underestimate problem  
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L hemisphere lesion pts learning guidelines   appropriate communication method, frequent feedback & support, do not underestimate learning ability  
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R hemisphere lesion pts learning guidelines   use verbal cues, demo may confuse, give frequent feedback, focus on slow & control, avoid spatial clutter, do not over estimate ability to learn  
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