OPT Allergies
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| transient lingual papillitis | enlargement of fungiform papilla on dorsal tongue
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| localized form of transient lingual papillitis | one enlarged fungiform papilla on anterior tongue
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| diffuse form of transient lingual papillitis | many enlarged, painful fungiform papilla; sometimes with fever and lymphadenopathy
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| 'papulokeratotic variant' of transient lingual papillits | diffuse and asymptomatic enlargement of fungiform papillae; most likely frictional hyperkeratosis
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| recurrent apthous stomatitis/ulceration (RAS/RAU) | oral ulcers mediated by CD8 T-cells
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| 1. antigenic stimulus 2. immunodysregulation 3. decreased mucosal barrier | 3 pathologic mechanisms of apthous stomatitis
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| simple apthosis | most common type of RAS; self-limiting without other disease processes
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| complex apthosis | patients that have several apthous ulcers with significant disease and recurrences
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| minor apthous stomatitis | mildest form of RAU; a couple of outbreaks each year, usually in adolescents
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| major apthous stomatitis | large ulcers with long healing time and a later onset
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| herpetiform apthous stomatitis | numerous small ulcers like herpes but not on gingiva or systemic symptoms
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| Behcet syndrome | systemic vasculitis with irregular oral ulcers on soft palate, genital ulcers, and ocular problems
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| sarcoidosis | multisystem granulomatous disorder, probably autoimmune; lungs and lymphoid tissues are most affected
-oral involvement of mass or papules is seen first in 2/3 of patients
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| Heerfordt syndrome | acute form of sarcoidosis; parotid gland enlargement, facial nerve paralysis, uveitis and fever
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| orofacial granulomatosis | variety of clinical presentations that show non-specific granulomatous inflammation; called Melkersson-Rosenthal syndrome when with facial paralysis and fissured tongue
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| Wegener granulomatosis | necrotizing granulomatosis lesions of respiratory tract; effects in kidneys and vasculitis
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| classic/generalized Wegener granulomatosis | fatal kidney involvement after initial respiratory infection
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| limited Wegener granulomatosis | disease stays localized to respiratory tract without renal lesions
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| 'superficial' Wegener granulomatosis | only have skin and oral mucosal lesions
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| strawberry gingivitis | early manifestation of Wegener granulomatosis
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| PR3-ANCA | antibodies detected in 90% of patients with Wegener's and can be used to monitor disease activity
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| stomatitis medicamentosa | mucosal reactions to systemic drugs
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| anaphylactic stomatitis | IgE mediated erythema and ulceration 24 hours after exposure to drug
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| fixed drug eruptions | reactions that reappear at same site after taking drug
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| lichenoid drug reactions | mucosal changes from systemic drug that mimic lichen planus
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| pemphigus-like drug reactions | mucosal desquamation and bullae formation like pemphigus
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| pemphigoid-like drug reactions | mucosa comes off in sheets and mimics pemphigoid
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| lupus erythematous-like drug reactions | fever, flu-like symptoms and lupus-like changes
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| nonspecific vesiculoerosive or apthous-like lesions | nonspecific mucosal changes due to drug reaction
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| allergic contact stomatitis | long list of agents that cause hypersensitivity reaction; {from artificial cinnamon flavoring}
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| perioral dermatitis | inflammatory reaction around mouth from antigen or corticosteroid cream
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| lichenoid contact reaction to dental restorative materials | reaction adjacent to old, corroding amalgam fillings
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| angioedema | diffuse edematous swelling of soft tissue; minor trauma can precipitate reaction
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| IgE-mediated type I hypersensitivity reactions | most common cause of angioedema; mast cells degranulate and histamine is released
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| angiotensin-converting enzyme (ACE) inhibitors | drug used to treat hypertension that also causes angioedema due to increased levels of {bradykinin}
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| angiotensin II receptor blockers | drugs developed to avoid inhibition of bradykinin breakdown and angioedema
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| activation of complement pathway | mechanism of causing angioedema; can be hereditary or acquired
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| type I complement pathway | inherited from decreased {C1 esterase inhibitor} that enhances complement pathway
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| Type II complement pathway | normal C1-INH levels but enzyme does not work
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| acquired type of C1-INH deficiency | can be seen in lymphoproliferative diseases that inhibit C1-INH
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Created by:
aharnold
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