Stack #145910
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| actions of neuroleptic drugs can be antagonized by | Drugs that increase dopamine like levodopa and amphetamine or mimic dopamine like bromocriptine.
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| Which drug is used to counteract prolactin secretion arising pituitary toumors | Bromocriptine
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| thist first generation agent can bind D1, D2, 5HT2, histamine H1 and a2 in the brain. | Haloperidol
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| adverse effects including tachycardia, impotence, and dizziness are due to | non-selective alpha adrenergic stimulation.
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| Weight gain and sedation is due to what receptor stimulation? | Histamine H1
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| this more selective atypical drug binds D2 5HT2 and a2 | Rispiridone
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| these type of agents bind dopamine receptors | typical
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| these agents bind dopamine and serotonin receptors | atypical-these are less sticky and bind then fall off
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| T/F blocking serotonin causes an increase release of dopamine | True
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| T/F serotonin itself will block the release of dopamine | True
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| Delusions, hallucinations, disorganized speech/behavior, agitation are examples of? | positive symptoms
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| passivity, apathetic social withdrawal, stereotyped thinking anhedonia, attention impairment, emotional withdrawal are examples of | Negative symptoms
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| impaired verbal fluency, problems with serial learning, problems with focused attention, concentration are examples of? | cognitive symptoms
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| increase in dopamine in this pathway causes positive symptoms | mesolimbic pathway
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| defecit of dopamine in this pathway causes negative and cognitive symptoms. | Mesocortical pathway
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| part of extrapyramidal system and controls movment | Nigrstriatal pathway
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| increase neuronal activity in this pathway inhibits prolactin release. Blockaed of D2 receptor increases prolactin release. | tuberoinfundibular pathway
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| D2 blockade in this path relieves positive symptoms | mesolimbic
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| D2 blockade in this path worsens negative symptoms | mesocortical
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| D2 blockade in this path produces EPS symptoms like parkinsonian, tardive dyskinisias, or hyperkinetic movement disorders | nigrostriatal
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| D2 blockade in this path casues prolactinemia | tuberinfundibular path
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| low potency meds cause | sedation, anti-ACH, and orthostatic hypotension.
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| Highpotency drugs cause | EPS
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| clinical consequences of D2 blockade | EPS movement disorders, endocrine changes, and sexual dysfunction
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| histamine blockade causes | sedation, drowsiness, weight gain, hypotension
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| Alpha-1 receptor blockade causes | Posteral hypotension, reflex tachycardia, dizziness
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| muscarinic blockade causes (anti-ACH) | blurred vision, dry mouth, sinus tachycardia, constipation, urniary retention, memory dysfunction
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| this activity can cause EPS so drugs with this anti activity will be less likely to cause EPS | cholinergic activity-therefore when high potency antipsychotics are precribed we will give anticholinergics.
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| This drug has affinity for D1, D4, 5HT2, muscarinic, and Alpha adrenergic receptors but is also a D@ antagonist | clozapine
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| These agent blocks 5HT2 receptors to a greater extent than D2 | Rispiridone and olanzapine
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| this agent is a partial agonist at D2 and a partial agonist at 5HT1A and an antagonist at 5HT2A. Also has moderate affinity for H1 and cholinergic muscarinic receptors | aripiprizole
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| this agent blocks D2 more potently than5HT2a receptors but is consideered weak overall. It is low risk for EPS due to short period of time it binds to D2 | quetiapine
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| Which drugs particularly block cholinergic (muscarinic receptors? | thioridazine, chlorpromazine
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| Which drugs particularly block H1 receptors? | Chlorpromazine and clozapine
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| Which drugs particularly block serotonin receptors? | Rispiradone and Clozapine
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| Which drugs particularly block Dopamine | all do but Strongest blockers are haloperidol, fluphenazine, and thiothixene
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| Which drug particularly blocks Alpha receptors | chlorpromazine
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| Possible Clinical consequences of D2 blockade | EPS, endocrine changes, sexual dysfunction
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| This drug is a partial agonist at D2 and 5HT1 and antagonist to 5ht2, it also shows maderate affinity for histamine, and alpha but no real affect of muscarenic | Aripiprazole
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| Quetiapine blocks D2 more potently than 5HT2a receptors but is relatively weak at blocking either receptor and is low risk for EPS. | quetiapine
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| all psych drugs have antiemetic effect except for these drugs . | Aripiprazole and thiordizine
-antiemetic effects occur through blockage of CTZ
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| produce anticholinergic effects including blurred vision, dry mouth, constipation, urine retention | thiordizine, chlorpromazine, clozapine and olanzapine.
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| sedation occurs with D2 blocking of H1 receptors with these drugs | Chlopromazine, clozapine, olanzapine, and quetiapine
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| drug induced nausea can be treated with this. | Prochloperazine
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| nausea from motion should be treated with? | antihistamines, sedatives and anti-cholinergics rather than antipsychotics
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| hiccups and pruritis can be treated with this | chlorpromazine
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| disruptive behavior in autism can be treated with this | rispiradone
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| Noncompliant patients can be treated with | long acting drugs like fluphenazine deconate, haloperidol deconate, and risperidone microspheres (upto 2-4 weeks)
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| these drugs are contraindicated in patients with seizure symptoms. | chlorpromazine and clozapine
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| T/F antipsychotics lower seizure threshold | True
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| T/F Atypical drugs decrease mortality in dementia related behavioral disturbances and psychosis | False
they increase mortality
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