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Physio 2011 Spring
Ch.16 Blood (ch.24)
| Question | Answer |
|---|---|
| Where is blood made? & ex | Bone marrow:Femur, skull, Sternum, pelvis |
| Proteins in the blood 3 | Albumins, Globulins, Fibrinogen |
| Function of Albumins | Carrier protein |
| Function of Globulins | Antabody |
| Function of Fibrinogen | Blood clotting |
| 5 WBCs | -Monocyte -Neutrophil -Eosinophil -Basophil -Lymphocyte |
| Inactive bone marrow | yellow marrow |
| Active bone marrow | Red |
| What change happen in the air as elevation gets higher? | Partial pressure, decrease |
| Is platelets true cell? | No |
| PCV | Packed Cell Volume |
| Platelets is also called | Thrombocytes |
| Platelets are made from | Megakaryocytes in bone marrow |
| Characteristics of leukocyte | -All have nucleus -Team effect, fight infection |
| Characteristic and Function of Monocyte 2 | -Going to injured site -precursor to Macrophage |
| Characteristic and Function of Neutrophil 2 | -Phagocytic cell -release cytokine |
| Characteristic and Function of Eosinophil 3 | -Acidic -Antiviral -Inflammatory response release contents and result in swelling |
| Characteristic and Function of Basophil 3 | -Inflammatory response -release histamine -Allerge |
| Characteristic and function of lymphocytes 4 | -Has cytoplasm -Protect from disease -Target of HIV -Evolved acquired immunity |
| Life span of WBC | 6-24 hours |
| 3 phagocytic cells | -Neutrophils -Monocytes -Macrophages |
| Define Leukemia | Cancer of WBC |
| Explain ALL | -Acute lymphotic leukemia -Risk of infection increases as Lymphocyte in crease -Due to excess production of WBC, production of RBC is limited -Hematopoiesis(Synthesis of blood cells) |
| Define Anemia | -Decrease ability to carry O2(not RBC) |
| mononucleosis | -Caused by Epstin-Barr virus that affects B-lymphocyte -aka Kissing disease |
| Symptom of mononucleosis | Fatigue, less E, due to suppression of immune system |
| Life span of platelets | 10 days |
| explain the shape of inactive platelet | Disk like cell: to fit in the capirally to pick up O2 & CO2 |
| explain the shape of active platelet | -Spiky surface: to stick together to form plaque |
| Biochemical pathway of clotting 6 | 1.Thromboplastin 2.Prothrombin 3.Thrombin 4.Fibrinogen 5.Fibrin 6.Platelets |
| Thromboplastin | -When I have injury to blood vessel, it is released by epithelial cell. -Convert Prothrombin to Thrombin |
| Thrombin | is converted to Fibrinogen(found in plasma) |
| Prothrombin | -Inactive form |
| Thrombin | -Active form of Prothrombin |
| Fibrinogen | Inactive form |
| Fibrin | -Active form of Fibrinogen -Trap platelets |
| Platelets | Trapped in fibrin |
| How is blood clotting event stopped? | release of Anti- platelets = prosracyclin |
| 3 inactive molecules | -Prothrombin -Fibrinogen -Plasminogen |
| Define plasmin | Activated form of Plasminogen(inactive) by Tissue Plasminogen Activator(used for MI) |
| What can break down blood clotting? | Plasmin |
| MOst numerous cell of the body | Erythrocyte |
| How many heme is in Hb? | 4 |
| Where is Fe located? | in Heme |
| Cause of Anemia | -Fe deficiency -Hemorrhage (uncontrolled bleeding)ex:hemophilia -Reduced Hb -Low Erythroprotein -Sickle cell anemia |
| Hemophilia is 3 | -X-link -Blood clotting disorder -male > female |
| Sickle cell anemia | -Autosomal,Homozygose -Blood is sickle shape that trap in capirally -It causes decrease carrying O2, Blood clots,fatigue |
| Which genotype is affected by Maralia? Ss/ ss | Ss; Sickle cell anemia stop mutation of malaria (protozoa) |
Created by:
MikiI