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Pharm Parkinson's

QuestionAnswer
Levodopa MOA increase dopamine in brain then stimulates dopaminergic receptors in basal ganglia to improve balance between dopaminergic & cholinergic activity
Levodopa/Carbodopa MOA levodopa plus inhibits peripheral breakdown of levodopa by inhibiting its decarboxylation thereby increasing available levodopa @ bbb
Symmetrol MOA antiviral against influenza A, blocks reuptake of dopamine into presynaptic neurons & causing direct stimulation of post synaptic receptors
Selegline MOA potent MAO-B inhibitor (metabolizes dopamine & increases dopaminergic activity by interfering w/ dopamine reuptake @ synapse
COMT Inhibitors MOA reversibly & selectively inhibits COMT
Comtan MOA inc duration of levodopa by 42-65%, only acts peripherally, no lab monitoring (COMT inhibitor)
Tasmar MOA more potent & longer acting, increases duration of dopamine by 94%, acts peripherally & centrally, need baseline & periodic LFTs (COMT inhibitor)
Parlodel MOA semisynthetic ergot alkaloid derivative w/ dopaminergic properties, inhibits PRL secretion, direct stimulation of dopamine receptors in corpus stratum (dopamine agonist)
Permax MOA semisynthetic ergot similar to parlodel but more potent & longer acting, centrally active dopamine agonist stimulating D1 & D2 receptors (dopamine agonist)
Mirapex MOA nonergot dopamine agonist w/ specificity for D2 receptor & binds to D3 and D4 which stimulates dopamine activity on nerves on striatum & substantia nigra
Requip MOA unknown, stimulant of postsynaptic dopamine D2 w/in caudate
Created by: Marywood
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