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Fluid & Electrolytes

Medical Surgical Nursing in Canada - Ch. 19

TermDefinition
Body weight an excellent indicator of overall fluid volume loss or gain
Cations positively charged molecules
Anions negatively charged molecules
Potassium prevalent cation in intracellular fluid
Sodium prevalent cation in extracellular fluid
Diffusion Movement of molecules from high to low concentration
Facilitated Diffusion Movement of molecules from high to low concentration without energy
Active Transport Process in which molecules move against concentration gradient (an area of lower concentration to an area of higher concentration)
Osmosis Movement of water between two compartments by a membrane permeable to water but not to solute
Osmotic Pressure Amount of pressure required to stop osmotic flow of water
Hydrostatic Pressure Force within a fluid compartment
entering the capillary If our hydrostatic pressure is less than the oncotic pressure we will have fluid
leave the capillary If our hydrostatic pressure is greater than our oncotic pressure we will have fluid
Edema When Plasma-to-interstitial fluid shifts
Water deficit (increased ECF/decreased ICF) Associated with symptoms that result from cell shrinkage as water is pulled into vascular system
Water excess (decreased ECF/increased ICF) Develops from gain or retention of excess water
Atrial natriuretic factor This hormone causes vasodilation and increased urinary excretion of sodium and water when there is increased atrial pressure (from increased volume)
Insensible Water Loss Invisible vaporization from the lungs and the skin, cannot be measured and the individual is unaware that the loss of water occurred
sodium This electrolyte is controlled by kidneys and action of aldosterone and ADH
Sodium Normal values 136-145 mmol/L
hypernatremia Excessive sodium intake with inadequate water intake can lead to
Hyponatremia Results from loss of sodium-containing fluids or from water excess
hyponatremia Manifestations are due to cellular swelling and first appear in CNS
osmotic demyelination syndrome with permanent damage to nerve cells in brain If you correct sodium too quickly it can lead to
Potassium Normal values 3.5-5.1 mmol/L
Potassium If the kidneys are not working, this electrolyte will accumulate at high levels
Hyperkalemia common in clients with massive cell destruction
Hyperkalemia Cardiac disturbances are most clinically significant in this electrolyte
Hypokalemia <3.5 mmol/L
Ionized Calcium Normal Values are 1.15-1.35mmol/L
Total Calcium Normal Values are 2.10-2.50 mmol/L
Phosphorus increases If calcium decreases
Hypercalcemia leads to reduced excitability of both muscles and nerves
Hypocalcemia increased nerve excitability and sustained muscle contraction (tetany)
Trousseaeu’s sign Carpal spasms induced by inflating a blood pressure cuff above the systolic pressure
Chvostek’s sign Contraction of facial muscles in response to a tap over the facial nerve in front of the ear
Phosphate Normal values 1.0-1.50 mmol/L
Hyperphosphatemia Neuromuscular irritability and tetany (related to low serum calcium levels associated with high phosphate levels)
Magnesium Normal Value 0.65-1.05 mmol/L
Hypermagnesemia Deep tendon reflexes lost (as levels increase) followed by muscle paralysis and coma
Hypotonic These solutions provides more water than electrolytes which dilutes the ECF and causes a movement of water from the ECF into the ICF
Isotonic Expands only the ECF and is ideal for fluid replacement
Hypertonic Raises the osmolality of ECF, the higher osmotic pressure causes water to shift out of cells and into ECF
Created by: KadduonoLU
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