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PARASCI_L7

Gastrointestinal and Abdominal Disorders

QuestionAnswer
liver, gallbladder, bile duct, kidney, transverse colon, ascending colon RUQ
liver, stomach, diaphragm, spleen, kidney, duodenum, pancreas, pancreatic duct, descending colon LUQ
cecum, veriform appendix, uterus, ovaries, RLQ
small intestine, umbilicus, rectum, bladder anus LLQ
liver, gallbladder, right kidney right hypochondriac region
liver (tip), small intestines, ascending colon, right kidney right lumbar region
small intestines, appendix, cecum and ascending colon right iliac region
stomach, liver, pancreas, left and right kidneys epigastric region
stomach, pancreas, small intestines, transverse colon umbilical region
small intestines, sigmoid colon, bladder hypogastric region
stomach, liver (tip), left kidney, spleen left hypochondriac region
small intestines, descending colon, left kidney left lumbar region
small intestines, descending colon, sigmoid colon left iliac region
transient relaxation of weak sphincter causing backflow of gastric contents into oesophagus gastroeosophagal reflux disease
mucosal injury to oesophagus and inflammation reflux oesophagitis
hyperemia, oedema and erosion of luminal surface in throat barett oesophagus
protrusion of stomach going through oespophagal hiatus of diaphragm hiatus hernia
bell shaped protusion of stomach above the diaphragm sliding/axial hiatus hernia
seperate portion of stomach enters thorax through widened opening paraeosophageal/non-axial (rolling) hiatus hernia
occurs with portal hypertension and gradual obstruction of veinous flow in liver, subject to rupture causing haemorrhage oesophageal varices
inflammation of gastric mucosa gastritis
may be accompanied with emesis, pain, haemorrhage, ulceration, acute gastritis
atrophy of stomach epithelium chronic gastritis
ulcerative disorder of upper GI tract occuring with exposure to acid-pepsin secretions peptic ulcer
an acute illness with primary manifestation of diarrhoea, accompanied by nausea, vomiting, fever & abdominal pain gastroenteritis
variable combination of chronic and reccurent intestinal symptoms, including abdo pain, altered bowel function, flatulence, bloating, nausea, anorexia, constipation, diarrhoea, anxiety or depression. pain relieved with bowel movement. irritable bowel syndrome
slow, progressive and recurrent inflammation of any section of GI tract, extending through layers of the intestinal wall. Causes fistula formations, leading to malabsorption, abdominal abcess and intestinal obstruction Crohn's disease.
non-specific continuous inflammatory condition isolated to rectum and colon, lesions form in base of mucosal layer, inflammation causes pinpoint mucosal haemorrhages which may ulcerate and become necrotic Ulcerative Colitis
single weakened pouch diverticulum
multiple weakened pouches diverticula
when pouches become inflamed or perforate diverticulitis
condition commonly occuring in distal descending sigmoid colon where mucosal layer herniates through surrounding muscle, occuring anywhere from pharynx to anus diverticular disease
diverticular present but asymptomatic diverticulosis
inflamed diverticula, may cause fatal obstruction, haemorrhage or infection diverticulitis
inflammation and obstruction where appendix becomes swollen, gangrenous and perforated if untreated, normally abrupt onset of pain localised in LRQ appendicitis
inability of intestinal tract to allow regular passage of food and bowel contents, bowel/intestinal obstruction
intrinsic or extrinsic factors, typically requires definitive intervention mechanical obstruction
common, self limiting without surgery adynamic ileus
inflammatory response of serous membrane, caused by bacterial invasion or chemical irritation, left untreated may cause toxamea, hypovolaemia and shock peritonitis
end stage of chronic liver disease, much of functional liver replaced by fibrous scar tissue, epigastric/ RUQ dull aching, full pain. cirrhosis
increased resistance of flow in portal veinous system & sustained portal vein pressure portal hypertension
80-90% of hepatic functional capacity must be lost in order to occur liver failure
abnormalities in composition of bile, stasis of bile, & inflammation of gall bladder may result in stone formation Cholelithiasis
80% cholesterol, 20% calcium salts gall stones
pass into common duct with symptoms of indigestion <8mm stones
more likely to obstruct flow & cause jaundice, abrupt onset of pain in URQ and epigastric area, persisting 30min-5h >8mm stones
diffuse inflammation of gallbladder, usually secondary to obstruction of gallbladder outlet acute cholecystitis
occurs with repeat acute cholecystitis or chronic irritation of gallbladder with gallstones chronic cholecystitis
inflammation of the pancreas by prematurely activated pancreatic enzymes (gall stones or alcohol abuse) pancreatitis
fluid accumulation and swelling in pancreas oedematous acute pancreatitis
cell death and tissue damage in pancreas necrotic acute pancreatitis
persistent inflammation of pancreas causing irreversible change to pancreatic structure and funciton, protein precipitates calcify and block pancreatic duct-> pancreatic glands enlarge -> damage islet of langherhans -> diabetes mellitus chronic pancreatitis
pathogen colonises the periurethral area and ascends through urethra towards bladder UTI colonisation stage
fimbria allow bladder epithelial cell attachement and penetration, bacteria continue to replicate and may form biofilms UTI uroepithelium penetration stage
bacteria ascend on ureter towards kidney, fimbria aid in ascension progress, bacterial toxins may inhibit peristalsis UTI ascension stage
infection of renal parenchyma causes inflammatory response UTI pyelonephritic stage
tubular obstruction and damage occurs in the kidneys, leading to interstitial oedema, and nephritis UTI acute kidney injury stage
polycrystalline aggregation in calyces renal caliculi
caused by stones 1-5mm in diameter that move into ureter and obstruct flow, causing pain in UQ radiating into LQ renal colic pain
caused by stones producing distention of renal calcyes or renal pelvis, dull deep ache in flank, varying in intensity non-colicky pain
characterised by diarrhoea, malena, haematemesis and coffe ground vomitus abdominal haemorrhage
dark, tar-like bowel movement indicating high abdominal bleed malena
frank blood in diarrhoea indicating lower abdominal bleed
vomiting of bright red blood indicating bleed proximal to stomach haematemesis
indicates lower GI bleed coffee ground vomitus
implantation of embryo outside uterus, commonly in fallopian tubes, associated with endometriosis or IUD ectopic pregnancy
term used to describe sudden, severe abdo pain likely to neccessitate surgery acute abdomen
Created by: 1092422624234171
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