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Pharm term 2
Beta Blockers
Question | Answer |
---|---|
What are beta blockers also known as? | Beta adrenergic blocking agents |
Where are beta 1 receptors located? | In the heart |
Where are beta 2 receptors found? | In the lungs |
How do beta blockers work? | By blocking the effects of norepinephrine and epinephrine they reduce heart rate, reduce blood pressure by dilating blood vessels |
How do non selective beta blockers work? ( propranolol /inderal) | l black b1 and b2 receptors therefore affect the heart, blood vessels and air passages |
How do selective beta blockers work? Metoprolol, lopressor | Primarily block B 1 receptors and therefore mostly affect the heart and do not affect air passages |
How does Labetalol /normadyne and Carvedilol/ coreg work? | They block beta and alpha 1 receptors. Blocking alpha receptors adds to the blood vessel dilating effect of labetalol and carvedilol |
What is a side effect of beta blockers that block B 2 receptors? | They may cause shortness of breath in asthmatics. |
What is the beta that beta blockers block? | Beta adrenergic receptors of the SNS |
Which condition might be an indication for beta-blockade? | BP 190 / 101 |
For what purpose are cardiac patients often prescribed beta blockers? | anti aginal |
A patient on lopressor complains of fatigue and slow heart rate. What do you suspect? | Normal effect of beta-blockade |
To reduce myocardial oxygen consumption, angina pectoris patients sometimes take: | Lopressor |
Fatigue and slow heart rates often occur with administration of: | Beta blockers |
What letters do beta blockers tend to end in? | Lol |
The generic name for inderal is: | Propranolol |
The following pathology is a relative contraindication for administering certain beta antagonist: | Status asthmaticus |
What drug when used in therapeutic doses, intentionally depresses the pumping action of the heart? | Propranolol |
Patients receiving beta-blockers will have | Increased R-R interval |
Loss of effectiveness of beta-blockade over a long period of time may be due to: | Receptor up-regulation |
Beta blockers ultimately reduce the coupling of actin and myosin fibrils by their effect on the chemical | Troponin I |
Increase in the number of beta receptors present on cell membranes in response to sustained beta-blockade is known as: | Upregulation |
Beta blockers reduce oxygen demand by decreasing the double product which isv | Heart rate times blood pressure |
Beta blocker dosage is adjusted to attain a resting heart rate ofv | 55 to 60 beats per minute |
Post-infarction, administration of beta blockers | Reduces mortality 35% |
Effects of beta blockers include | Decreasing cardiac muscle contractility |
Which of the following is an indication for beta blocker therapy? | Prevention or treatment of an MI |
Timolol is a non-selective beta antagonist. Therefore its use is contraindicated in patients with: | Asthma |
Which of the following statements best describes one of the mechanism of action of beta adrenergic blocking agents? | The inhibition of the reaction between norepinephrine and the receptor. |
Metoprolol tartrate is used as an: | Anti hypertensive |
Labetalol/normodyne would not be used to treat: | Asystole |
Labetalol/normodyne may be used to treat: | Hypertension, angina, symptomatic mitral valve prolapse, sinus tachycardia caused by hyperthyroidism |