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TEST #5 F&E

Patho

QuestionAnswer
what is the purpose of fluids in the body? transportation, electrical conductions, energy production, nad maintaining homeostatis
where are fluids in the body found? intra cellular fluid, extracellular fluid, intravascular and interstitial
pressure of H2O against the membrane osmotic pressure
the pressure produced by or associated with osmosis and dependent on molar concentration and absolute temperature osmotic pressure
amoutn of the concentration of the solution osmolarity, osmolality
directional movement of the fluid tonicity
no movement of fluid and replaces lost fluid isotonic
shifts fluid out of the vessles into cells or tissue hypotonic
shifts fluid from the cells and tissue into the vessels hypertonic
what does a high osmolarity/osmolality mean? more concentration with less fluid
What does a low osmolarity/osmolality mean? a lower concentration with more fluid
describe the fluid movement of the infusion of isotonic solution into veins no fluid movement
describe the movement of the infusion of hypertonic solution into the veins fluid movement into veins
describe the fluid movement of the infusion of hypotonic solution into the veins fluid movement out of the veins
when fluid is not in vascular space or in the cells. this causes ____ third space...edema
> pressure of the capillary in which fluid begins to leak out capillary filtration pressure
looking from the vascular side and the plasma proteins keep fluids from shifting into weird places capillary colloidal pressure
pressure outside of the vessel, pushing back into the capillary system interstitial hydrostatic pressure
looking from the cell side and plasma protins keep fludis from shifting into wierd places tissue colloidal osmotic pressure
where does the biggest water fluid intake come from? water beverages
water chases ____ and ___ glucose and Na
what is the normal concentration of Na? .9%
if fluid was hypotonic, what % of saline would be given to a pt? 0.45% or 1/2 of Normal Saline
If fluid levels where hypertonic, what % of saline would you give to a pt? 3% normal saline ex: edema and burned pt
what part of the loss of water cannot be calculated? insensable water loss (ex: sweating, lung water from talking and coughing)
what is the best way to tell if there is a balance or inbalance of fluids? weigh the pt each day at the same time, with the same clothes on, and the same scale
what causes the biggest loss of water from the body? urination
accumulation of interstitial fluids and can be localized or generalized edema
termed used for water edema hydro edema
term used for water shift to the belly ascites edema
term for fluid in the spaces around membrane effusion edema
water makes up ____ of body fluids 90-93%
Na makes up ____ of extracellular fluid 90-95%
___ regulates the extracellular fluid Na
what are the sources of Na? canned or boxed food, stuff outside cell, and in GI secretions
how is Na secreted? through urine and GI tract
what is the role the kidneys play with Na? kidneys regulate the amount of Na in the blood stream
___ holds in Na. Na holds in ____ aldosterone...water
there is a high concentration of ___ outside of the cell Na
___ triggers dehydration and < volume of water thrist
hypodipsia < thirst
polydipsia excessive thirst (ex: in DM pt)
hormone that prevents you from urination antidiuretic hormone
what is diabetes insipidus? when a pt has to go go go (> urination like in a DM pt)
what does a SIADH (syndrome of inappropriate antidiuretic hormone) do? this is when you hold in urination too much
what are the 3 main causes of hypovoliema (fluid volume deficit)? insufficient intake and inadequate replacement, and excessive fluid loss
abnormal deficiency of protein in the blood hypoproteinemia
fluid volume deficit hypovoliema
fluid volume excess hypervoliema
what are the causes of hypervoliema or fluid volume excess? escessive intake, excessive use of saline edemas, steroid therapy, heart failure, liver failure, stress, remobilization after a burn tx, hypertonic or hyperosmolar solutions
what are some major s/s of hypervoliema (fluid volume excess)? tachycardia, hypertension, wt gain, JVD, tachypnea, dyspnea, crackles, cough, peripheral edema, < HCT, < BUN, < specific gravity
measures osmolarity specific gravity
low blood Na hyponatremia
having too much Na or water in the blood vessels, which causes an > in the ICF pressure hypertonicity
too little Na or water in the blood vessles, causing a < in the pressure of the ICF hypotonicity
euvolemic normal fluid volume
what are the ways to lose Na out of the GI tract? GI suction, vomiting, diarrhea
passage ways the body makes that you were not born with fistulas
what are the early s/s of hyponatremia? n/v/d, abdominal cramps
what are the main s/s of hyponatremia? weakness, fatigue, anorxia
what are the late s/s of hyponatremia? tremors, seizures, lethargy, mental confusion, disorentation
high blood Na hypernatremia
an excessive loss of water will cause hypernatremia b/c ___ Na levels will get HIGH if there isn't enough fluid
what are the major s/s of hypernatremia? tachycardia, weak pulse, postural HTN, thirst, low grade temp, tachypnea, oliguria, > BUN, > osmolarlity, > Na, > HCT, > RBC
reduced secretion of urine oliguria
is vital to the acid-base balance Na
the ___ balance is found inside of the cells K
second most abundant cation K
major cation of the ICF K
___ outside of cell, ____ inside of cell Na....K
critical in osmotic and acid-base balance, kidnesy's ability to concentrate urine, necessary for growth, carb, gluscose, and protein metabolism, and electrical conduction K
what is the usual source of K intake? diet
what are the main sources of excretion of K? kidneys, stool, sweat
describe Na and K relationship inversely proportional= Na up, K down....Na down, K up
____ shifts between ICF and ECF in attempts to maintain balance K
what influences K shift between ICF and ECF insulin, B-adrenergic stimulation, serum osmolarity, acid-base balance, and exercise
low K in blood hypokalemia
what are the main causes of hypokalemia? < intake, diuretics
what are the main s/s of hypokalemia? orthostatic HTN, muscle weakness and cramping, parasthesia,hyperglycemia, metabolic alkalosis, dyspnea, polyuria, polydipia, ECG changes, and cardiac arrest
what are the ECG changes in hypokalemia? flat t wave, presence of U wave, depressed ST segment. prolonged QT and PR interval, dysrhythmiass
what are the most dangerous s/s of hypokalemia? cardiac dysrythmias b/c they are a deadly sign
how should you adm an K IV? slowly and diluted...not as a push
high K in blood hyperkalemia
what are the main causes of hyperkalemia? renal impairment, K sparing diuretics, ACE inhibitors, burns
what are the ECG changes in hyperkalemia? tall, narrow, peaked T wave...wide QRS...prolonged PR interval...flattened to absent P wave...dysrhythmias (life threatening)
most of __ is found in the bones Ca
what are the main functions of Ca? bone formation and metabolism, neural transmission and function, initiates skeletal muscel contraction, and maintains cell membrane integrity
enters through GI tract Ca
must have ___ from Ca to be efficient Vit D
where is Ca excreted? to the kidneys and GI tract
__ and ___ play a big role in the excretion of Ca PTH and calcitonin
what is the relationship between Ca and Ph? inversely proportional= Ca up, Ph down...Ca down, Ph up
Ca and ___ are directly proportional Mg.....Mg up, Ca up.....Mg down, Ca down
what are some main causes of hypocalemia? < intake, < absorption, > secretion
what are the main s/s of hypocalemia? parasthesia, Chvestak's sign, trousseau's sign, > DTR, pathological fracture, skin hair and nail changes, larygospasms, stridor, bruising, bleeding, ECG changes
what are the ECG changes in hypocalcemia? prolonged QT interval,dysrythmias
body doesn't take ___ supplements well Ca
high Ca in the blood hypercalcemia
what are the main causes of hypercalcemia? hyperparathyriodism, malignancy, immoblization, renal impairment
what are the main s/s of hypercalcemia? anorexia, N/V, abd pain, < bowel sounds, constipation, neuromuscular weakness to flaccidity, < DTR, confusion, depression, lethargy. stupor, coma, renal calculi, ECG changes
what are the ECG changes for hypercalcemia? shortened QT interval, inotropic effect, dysrhythmias
second most abundant ICF cation Mg
cofactor for many enzyme activity Mg
essential for ATP synthesis, DNA replication and transcription, cellular metabolism, membrane functions, nerve conduction, ion transports, and Ca channel activity Mg
ingested through diet Mg
how is Mg excreted? through kidneys
what are the ECG changes in hypomagnesemia? prolonged QT interval, dysrhythmias
low Mg in the blood hypomagnesemia
high Mg in the blood hypermagnesmia
what are some causes of hypermagnesmia? > intake, use of Mg antacids and laxatives, renal impairment, endocrine disorders, acidosis
what are 3 endocrine disorders that may cause hypermagnesmia? hypoparathyroidism, hypoaldosteroneism, and hypothyroidism
what are some main s/s of hypermagnesmis? bradycardia, hypotension, muscle weakness, <DTR, resp. impairment, lethargy
indirect measure of H ion concentrations pH
a substance that can give up an H ion; result of cellular metabolism Acid
carbonic acid....exhalable volatile
sulfuric, uric acid...excreted in kidneys nonvolatile
blood pH < 7.35 acidosis (acid)
a substance that can accept an H ion base
___ is the primary base in the body bicarb
blood pH > 7.45 alkalosis (base)
a blood pH between ___ and ___ is incompatible with life <6.8 and >7.8
what are some chemical buffers in the acid-base system? bicarb-carbonic acid...phosphate system...ammonium...some proteins
chemical buffers are an ____ system immediate response
the most important chemical buffer and is generateed int he kidneys and aids in the elimanation of H bicarb-carbonic acid
chemical buffer that aids in excretion of H by the kidneys phosphate system
chemical buffer that is added to ammonida in the renal tubules to form ammonium ammonium
chemical buffer that aids in buffering ECF certain proteins
what are the 3 parts of acid-base regulation? chemical buffers, respiratory system, and renal system
regulates teh excretion or retention of carbonic acid respiratory system
if pH <, the resp rate and depth ___ >
if pH >, the resp rate and depth ___ <
fast but weak acid-base regulator respiratory system (responds within minutes)
slow but powerful acid-base regulator renal system (responds within 48 hrs)
regulates the excretion or retention of bicarb and the excretion of H and nonvolatile acids renal system
if pH <, kidneys ____ bicarb retain
if pH >, the kidneys ____ bicarb excrete
losing bases metabolic acidosis
deficit of bicarb metabolic acidosis
what are the causes of metabolic acidosis? ketoacidosis, renal failure, diarrhea
what are the main s/s of metabolic acidosis? weakness, tremors, tachypnea, hypothension, confusion, lethargy, dysrhythmias
what is Kussmaul's? when pt is panting to get more expiration out (mouth breathing)
how does the body compensate with metabolic acidosis? lungs > rate and depth of ventilation...PaCO2 levels <...change is rapid, usually within minutes to hours
gaining > bases metabolic alkalosis
what are the main causes of metabolic alkalosis? NG suction, K losing thru diuretics
what are the main s/s of metabolic alkalosis? bradypnea, parasthsia, confusion, > muscle irritability, tetany, seizures, coma
what are compenstations for metabolic alkalosis? lung < rate and depth of ventilation...PaCO2 levels >...change is rapid, usually within minutes to hours
excessive retention of CO2 respiratory acidosis
what are the main causes of resp acidosis? airway obstruction and hypoventilation
what are the main early s/s of resp acidosis? tachycardia, tachypnea, diaphoresis
what are the late s/s of resp acidosis bradycardia and hypotension
what are the compensations for resp acidosis? kidneys reabsorb more bicarb or excrete more H...bicarb and base excess levels >...change is slow and may take 2-3 days
excessive elimination of CO2 respiratory alkalosis
what is the main cause of respiratory alkalosis? hyperventilation, anxiety
what are the main s/s of resp alkalosis? tachyardia, palpitations, dry mouth, anxeity, profuse perspiration, parasthseia, inability to concentrate
what are the compensations for resp alkalosis? kidneys excrete more bicarb...bicarb and base excess levels...change is slow 2-3 days
what is a tx for resp alkalosis? breath into paper bag
kidneys will shut down if ___ urine is not made
Created by: TayBay15
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