Stack #121119 Word Scramble

 
 

 
 

 
 

 
 
 
 
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Sac encasing heartPericardium
2 layers of pericardiumVisceral pericardium & Parietal pericardium
Visceral vs. Parietal pericardiumsV:attaches to heart's epicardium, P:supports heart mediastinum
3 physiologic characteristics that provide heart's synchronizationAutomaticity, Excitability, Conductivity
Automaticity vs. Excitability vs. ConductivityA:initiate electrical impulse, E:respond to electrical impulse, C:transmit electrical impulse to another cell
SA node locationJunction of superior vena cava & Right atrium
AV node locationRight atrial wall near tricuspid valve
Reason for AV node delayAllow atria to contract and complete ventricular filling
Purkinje fibers stimulateMyocardial cells causing ventricular contraction
Ions that stimulate cardiac cellsNa, K, Ca
Na & K r/t Location during Polarized/resting stateNa is extracellular ion, K is intracellular ion
Cardia action potential d/tRepeated depolarization and repolarization
Phase 0Rapid Depolarization, Positive Na ions move into cells
Phase 1Rapid Repolarization, K exits cells
Phase 2Plateau phase, Repolarization slows, Ca enters cells
Phase 3Final repolarization
Phase 4Resting phase b/f next depolarization
Speed r/t Ca & NaNa uses fast channels, Ca uses slow channels
Cells are incapable of stimulation duringRefractory period
Responsiveness r/t Effective refractory periodUnresponsive to any electrical stimulus
Phases r/t Effective refractory periodPhase 0-mid Phase 3
Phases r/t Relative refractory periodEnd of Phase 3
Stronger than normal electrical impulse during Relative refractory period causesPremature depolarization
Causes closing of AV valvesIncrease in ventricular pressure
Causes semilunar valves to openIncreased ventricular pressure
Causes closure of semilunar valvesDecreased pressure in pulmonary and aortic arteries
Atrial systole synonymAtrial kick
Hemodynamic monitoring measuresChamber pressures
Stroke volumeAmount of blood ejected per heartbeat
Cardiac output vs. Stroke volume r/t Resting adultCO:5 L/min, SV:70 mL
Sympathetic impulses vs. Parasympathetic implusles r/t HRS:Increase HR, P:Decrease HR
Catecholamines & Thyroid hormone r/t HRIncrease HR
Location of baroreceptorsAortic arch, Both internal carotid arteries
3 Factors that determine Stroke volumePreload, Afterload, Contractility
Diuresis, Venodilating agents & Low blood volume r/t PreloadDecrease preload
Example of Venodilating agentNitrates
Systemic vascular resistanceResistance of systemic BP to left ventricular systole
Pulmonary vascular resistanceResistance of pulmonary BP to right ventricular systole
Relationship b/w Afterload & Stroke volumeInverse
Increase in stroke volume r/t Preload, Contractility & AfterloadPreload & Contractility increase, Afterload decreases
Complications r/t Acute Coronary Syndrome (ACS)Dysrhythmias, HF
ACS is due toThrombus in diseased coronary artery
Signs and symptoms of CVD are r/tDysrhythmias, Conduction problems, CAD, HF, Cardiogenic shock
Elderly w/Diabetes may not experience which symptom r/t ACSAngina d/t neuropathy
Symptoms r/t ACS in Elderly w/Diabetes to look forSOB, Fatigue
More at risk for CAD r/t GenderMen
Common elimation symptom r/t HFNocturia
Indicator r/t Dysfunction of heartReduced pulse pressure, Cardiac enlargement, Abnormal heart sounds
Central cyanosis is observedTongue, Buccal mucosa
Normal pulse pressure range30-40 mm Hg
Factors r/t Increased Pulse pressureElevated stroke volume, Reduced systemic vascular resistance, Reduced artery distensibility
Factors r/t Decreased Pulse pressureReduced stroke volume, Obstruction to blood flow during systole
3 most common causes r/t Orthostatic hypotensionReduced blood volume, Vasoconstrictor mechanism insufficiencies, Insufficient autonomic vasoconstricition
Time elapse b/w postural changes r/t Orthostatic hypotension measurement1-3 minutes
Vasoconstrictor mechanism insufficiencies vs. Autonomic insufficienciesHR does not change w/autonomic insufficiencies
Sinus arrhythmia r/t InspirationPulse increases w/inhalation, Decreases w/exhalation
Sinus arrhythmis is common inChildren, Young adults
Palpability r/t Apical pulse in 2 adjacent intercostal spacesLeft ventricle hypertrophy
S1 & S2 r/t Area most audibleS1:Apex, S2:Base
Gallops are d/tVibrations during diastole
Gallops during ventricular filling vs. Gallops during atrial contractionVent filling:S3, Atrial contraction:S4
Inadequate renal perfusion r/t Urine outputDecreases urine output
First enzyme level to increase d/t MICreatine kinase
Peaks 2-3 days after CK increasesLactic dehydrogenase
Myoglobin vs. TroponinM:Can rule out early diasnosis of MI, T:Very early or late MI diagnosis
Necessary before Lipid profile and Homocysteine level may be drawn12-hour fast
Normal cholesterol levelLess than 200 mg/dL
LDL vs. HDL r/t functionLDL:transports cholesterol and triglycerides into cell, HDL:transports away from tissues/cells to liver for excretion
Triglyceride levels r/t LDL & HDL levelsLDL and triglycerides increase, HDL decreases
Brain natriuretic peptide(BNP) excellent in diagnosingHeart Failure
End products of protein metabolismBUN and creatinine
Prothrombin time(PT) and International Normalized Ratio(INR) measureLevel of coagulation and effectiveness of warfarin(Coumadin)
Graphic recording of heart's electrical activityElectrocardiography(ECG)
Lead II vs. Lead VI r/t Monitoring effectivenessII:atrial depolarization(P wave), VI:Ventricular dysrhythmias
Electrode changing r/t TelemetryEvery 1-2 days, Use different locations
3 tests r/t Cardiac stress testExcersise stress test, Pharmacologic stress test, Mental stress test
Nursing interventions r/t Exercise stress testing & Pharmacologic stress testing4-hour fast b/f test, Avoid stimulants
Ultrasound of heartEchocardiography
2 IV drugs used to vasodilate r/t Pharmacologic stress testingadenosine(Adenocard), dipyridamole(Persantine)
Block effects of dipyridamole & adenosineTheophylline & xanthines
Thallium uptake r/t Myocardial perfusion imagingNo uptake:infarcted tissue, Delayed uptake:ischemic myocardium
Computed tomography(CT) scans provideCross-section images of chest
Cardiac catheterization measuresPressure & O2 sat in all heart chambers
Electrophysiology providesDysrhythmia diagnosis & management
Phlebostatic axis vs. Phlebostatic level r/t Hemodynamic monitoringA:4th intercostal space at sternum, L:horizontal line in even w/phlebostatic axis in which stopcock must be level for accurate readings
Must be confirmed b/f Intra-arterial catheters are insertedCollateral circulation
Cardioversion vs. DefibrillationC:synchronized w/Pt's conduction, D:unsynchronized
2 Safety measures r/t PaddlesGood contact b/w paddles and skin, No one in contact w/Pt when defibrillator is discharged
Medium necessary b/w Paddles and skinConductive medium
Called 3x b/f pressing Discharge buttonCLEAR
CPR cycles b/w shocks5
Cardioversion discharge that is not synchronized can causeVentricular fibrillation
Defibrillation is not used on these Pt'sConscious, Have a pulse
Inhibited vs. Triggered r/t Pacemaker functionI:pacemaker functions when heart does not beat, T:pacemaker functions d/t intrinsic activity
Capture vs. Spike r/t PacemakersS:ECG interpretation when pacing begins, C:appropriate rhythm after spiking
Most common complication r/t PacemakersDislodgement of pacing electrode
Magnet-containing objectsEarpiece of phone, Large stereo speakers
Airport searching r/t PacemakersRequest hand search instead of wand
Implantable Cardioverter Defibrillators(ICD) functionDetect/terminate life-threatening tachycardia/fibrillation
Microwaves r/t ICD'sNo concerns