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GI Alterations

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Question
Answer
inflammation of the stomach and intestinal tract   gastroenteritis  
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major source of gastroenteritis   contaminated food and water  
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major organisms causing gastroenteritis   E. Coli, Compylobacter, C. dif, Salmonella, Shigella  
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incubation period for microorganisms   6hrs to 3-4days  
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bacteria/virus cause this in the intestine   tissue damage and inflammation  
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antibiotics effect on intestine   depresses action of flora  
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endotoxins effect on intestine   greater water and e-lyte secretion  
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stool may be positive for these in gastroenteritis   leukocytes and the causitive organism  
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labs to monitor for gastro.   K and bicarb  
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pancreatic enzymes   trypsin, amylase, lipase  
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action of insulin   transports glucose to cells and storage  
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action of glucagon   inhibits storage of glucose  
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somatostatin   inhibits insulin  
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type of cell in pancreas that produces insulin   beta cells  
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increases amino acid transport   insulin  
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stimulates protein synthesis   insulin  
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stimulates ATP synthesis   insulin  
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fasting plasma glucose   hyperglycemia >126mg/dl; hypoglycemia <50mg/dl  
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GTT   administration of oral glucose load, blood sugar test after 1 and 2hours. >200mg/dl is abnormal  
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Urine Glucose   presence is abnormal except during pregnancy or renal threshold is lowered  
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Glycosylated Hemoglobin (HbA1c)   percent hemoglobin that is glucose bound. nomal is 4-6% Indicates glucose control over last 2-4mos  
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Serum Amylase   rises with pancreatic pathology  
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hypercortisolemia   stimulation of gluconeogenesis(cause of hyperglycemia)  
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elevated thyroid, prolactin, or growth hormone   cause excess insulin release with eventual resistance  
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manifestations of acute pancreatitis   postprandial epigastric pain; sever nausea and vomiting  
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diagnosis of acute pancreatitis   elevated serum amylase,lipase. hyperglycemia and hyperlipidemia. increased WBC  
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how pancreatic CA metastisizes   via the portal vein or lymphatics  
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manifestations of pancreatic CA   vague; aversion to food, pain with advanced disease  
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diagnosis of pancreastic CA   laparotomy, ultrasound, CT scan  
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Tx of pancreatic CA   surgery  
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liver function with glucose   stores glycogen and stimulates gluconeogenesis  
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liver function with aminon acids   stores and produces  
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liver function with fatty acids   breakdown for ATP  
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liver function with cholesterol   breakdown of to synthesize bile salts  
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bilirubin transformation   byproduct of RBC breakdown  
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hormaone biotransformation   converts hormone to water soluble form  
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ammonia biotransformation   byproduct of protein breakdown, made into urea  
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blood storage   liver function  
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plasma protein synthesis   albumin-plasma oncotic pressure  
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hematologic liver function   clotting factor synthesis; 1,2 and 7 vitamin K synthesis  
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immunologic function of liver   phagolytic macrophages, Kupffer cells  
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liver stores these vitamins and minerals   B12, A, D, and iron  
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test of liver function   bilirubin, enzymes, plasma proteins, PT, ammonia  
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diagnostic tests of liver function   ultrasound, CT, MRI, liver biopsy  
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prodromal stage symptoms of hepatitis   malaise, fatigue, URI, myalgia, aversion to food  
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icteric stage of hepatitis   jaundice, hepatic tenderness and enlargement, splenomegaly, pruritis  
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drug to treat hepatitis   antivirals & interferons  
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