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EENT

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Question
Answer
Cause of blindness, more common in patients >70 y.o. =   Macular degeneration  
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Marcus-Gunn pupils (afferent pupillary defect): due to:   Optic nerve lesion (optic neuritis); or tertiary syphilis  
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Bilateral pinpoint pupils may be 2/2:   Pontine hemorrhage  
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Bilateral DILATED pupils may be 2/2:   Anticholinergics, TCA, anti-parkinsonian drugs, profound hypoxemia  
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Bilateral/bitemporal hemianopia   Optic chiasm lesion  
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Loss of central vision   Ipsilateral optic nerve lesion?  
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Superior contralateral quadrantopia   Temporal optic radiation  
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Eye trauma, diplopia. Exophthalmos, fixed upward gaze, hyphema   Orbital blow-out fracture; Immediate ophthalmology referral  
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Hard, nontender swelling/nodule on upper or lower eyelid. Large lesion may cause astigmatism or distorted vision (2/2 pushing on cornea) =   Chalazion; usually 2/2 blocked meibomian gland  
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Painful swelling of upper or lower eyelid. Visual acuity normal =   Hordeolum; usually 2/2 Staph aureus infection  
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Painless, elevated fleshy yellow nodule on sclera adj to cornea   Pinguecula; 2/2 actinic exposure, trauma, dry windy conditions  
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Transient monocular vision loss   Amaurosis fugax (2/2 TIA, embolus)  
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Vision loss over hours to days, painful EOM   Optic Neuritis (assoc. w/ MS)  
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Recurrent episodes of vision change, diplopia that resolve   MS  
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Red eye, watery discharge, sticky lids, preauricular LAD   Viral conjunctivitis; usu 2/2 adenovirus 3, 8, or 19  
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Red eye, lid edema, copious purulent discharge =   Gonococcal conjunctivitis. Likely concurrent urethritis.Vision not blurred in bac conjunctivitis  
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Red eye, hyperemia, chemosis, nodular conjunctivia   Allergic conjunctivitis  
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Acute painful red eye, halos around lights, hazy cloudy steamy cornea; circumlimbal injection; pupil less reactive & mid-dilated. Onset after being in dark room   Acute angle-closure glaucoma  
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Diplopia, dysarthria, dysphagia   Vertibrobasilar insufficiency  
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Penetrating eye trauma: do not   remove object  
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Rust ring on cornea =   metallic foreign body  
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Chem burns to eye   irrigate with water >30 min; eye shield, ED  
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Blow out fx of eye   cannot look up (infraorbital n. entrapment), cross eyed, exophthalmos, subQ emphysema  
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Corneal abrasion on slit lamp or dye:   epithelial defect but clear cornea (use proparacaine as ocular anesthetic)  
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Corneal abrasion tx   Abx (Erythromycin or Polytrim drops), APAP, cycloplegic agent (cyclopentolate, homatropine), consider patch 24 hrs. If contact wearer: must cover pseudomonas (tobramycin ointment or cipro/ofloxacin drops)  
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Drugs causing macular degeneration   chloroquine, phenothiazine  
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Metamorphopsia=   wavy vision (mac degen), test w/Amsler grid  
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Halo seen around lights; black spots in red reflex; no color vision; pupil whitens = sx of:   cataracts  
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Chronic, Asx until late, increased IOP, peripheral visual field defects, increased cup:disc ratio on fundoscopy =   Open angle glaucoma  
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Ptosis, lid edema, exophthalmos, pus, conjunctivitis; fever, dec eye mx ROM, sluggish pupils:   orbital cellulitis  
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Scurf (seborrhea), collarettes (SA) (red rim); epithelial keratitis =   blepharitis  
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Internal hordeolum location   meibomian gland abscess; deep to palpebral margin  
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External hordeolum location   glands of Moll or Zeis; adjacent to palpebral margin; smaller  
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Slow growing thickening of bulbar conjunctiva   pteryigium; grows from nasal side toward cornea  
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Papilledema causes   malig HTN, hemo stroke, subdural hematoma, pseudotumor cerebri  
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Optic disk swollen, blurred margins, vessels obliterated:   papilledema  
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Lesion anterior to optic chiasm affects:   only one eye  
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Homonymous hemianopia (defect in both R or both L halves of visual fields of both eyes):   lesion of optic tract or lateral geniculate nucleus (R lesion = L side vision loss)  
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Reduced visual acuity not correctable by refractive means=   amblyopia  
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Cyanotic sclera may =   osteogenesis imperfecta  
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Hordeolum tx   May resolve spontaneously. Topical Abx (bacitracin or erythro ointment BID x7-10 days). Warm compress QID. I&D if persistent. Systemic Abx if preseptal cellulitis  
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Chalazion tx   Visual acuity testing. Warm compress QID; refer to Eye Dr if persistent; corticosteroid injection vs I&D if persistent  
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After a VII CN palsy: excess lacrimation, drooping eyelid =   ectropion (lid turned outward). Tx: artificial tears vs lid tightening surgery  
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Blepharitis causative bugs   SA / seborrhea (anterior), S epi, Coag neg Staph. Also meibomian gland dysfn (posterior)  
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Blepharitis mgmt   Anterior: hygiene (lid scrubs, remove scales), bacitracin ointment. Posterior: meibomian gland (MG) expression, Derm, oral Abx (doxy) for MG dysfn  
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Bacterial conjunctivitis: bugs   Chlamydia, gonococci, SA, S pneumo, Hemophilus, Pseudomonas, Moraxella  
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Bac conjunctivitis mgmt   Viral (strict hygiene, toss contacts & case); Bac: topical erythromycin oint or sulta gtt 5-7d.  
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Gono conjunctivitis mgmt   Eye Dr referral ASAP. Hosp for IV & topical Abx: emergency (poss corneal involvement)  
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Dacryocystitis workup   CBC, ANA (to r/o lupus), ANCA Ab (r/o Wegner); consider CT or MRI if uncertain etiology  
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Dacryocystitis Tx   Peds: Aug + topical abx gtt, warm compress. Adults same Abx, dacryocystorhinotomy may be nec  
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Orbital cellulitis: etiology & patho   paranasal sinusitis (most common); eye surg comp, dental infxn, orbit trauma. May cause brain abscess or meningitis  
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Orbital cellulitis: bugs   SA, S pyogenes, S pneumo, H flu  
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Genetic (M>F), HPV, tumor suppressor p53, HLA Ag; triangular conjuctival thickening growing inward to cornea =   pterygium  
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Gray circumscribed opacity that necrosis & forms an excavation of the cornea =   corneal ulcer (refer to Eye ASAP)  
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Fluorescein stain: bright green area (seen under black light) =   corneal abrasion  
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Fluorescein stain: dark, with a surrounding area of bright green (seen under black light) =   corneal foreign body  
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Etio includes MS, viral, SLE, MEtOH, vascular; unilateral pain with any eye movement =   optic neuritis (refer to Eye Dr ASAP)  
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flame-shaped peripapillary hemorrhages may be seen =   optic neuritis  
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2 types of diabetic retinopathy   proliferative; nonproliferative  
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Leading cause of blindness in US =   DM retinopathy (MCC). Most common in patients <70 yo  
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Diabetic retinopathy: capillary microaneurysms, tortuous/dilated veins, flame hemorrhages, cotton wool patches =   non-proliferative  
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Diabetic retinopathy 2/2 ischemia: neovascularization, vitreous hemorrhage   proliferative  
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Etio/pathology of HTN retinopathy   2/2 atherosclerosis (2/2 htn); acute BP elevation -> loss of autoregulation in retinal vessels -> vasoconstriction & ischemia  
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HTN retinopathy: funduscopy   retinal hemorrhage & edema, cotton wool exudates, Cu/Ag wiring, AV nicking  
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Curtain or veil top-to-bottom over eyes, new onset floaters =   Retinal detachment  
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Retinal detachment usually begins at:   superior temporal retinal area; afferent pupillary defect  
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3 types of retinal detachment   rhegmatogenous residual detachment/RRD (most common); exudative/serous (ERD); tractional detachment (TRD)  
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RRD detachment etiology   penetrating or blunt trauma (or spontaneous)  
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ERD detachment etiology   accumulation of subretinal fluid (2/2 inflammation or tumor)  
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TRD detachment etiology   adhesions (2/2 SCDz, trauma, proliferative DM retinopathy)  
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Retinal detachment sx/sx   Photopsia (flashes of light), shower of floaters, wavy distortion of objects. IOP >5-5mmHg.  
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Sudden painless unilateral vision loss. History of TIA, palpitations, arrhythmia, carotid disease, embolic source   CRAO (pale retina, cherry red macula)  
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CRAO causes   emboli, thrombi, vasculitides  
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Arteriolar narrowing, box-car appearance of arterial flow, cherry red spot (perifoveal atrophy), retinal edema (1st 4-6 wks), possible pale retina & optic n., local cotton-wool =   CRAO  
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Sudden painless unilateral vision blurring or loss, often upon waking. History of HTN   CRVO (blood & thunder, retinal hemorrhages); usually 2/2 thrombotic event  
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Neovascular glaucoma can develop in the first 3 months in:   CRVO  
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CRVO mgmt:   W/U to R/O & prevent CVA; panretinal laser photocoag if glaucoma; vitrectomy; tPA?  
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CRAO mgmt:   W/U to R/O & prevent CVA; high-flow O2; IV acetazolamide; IOP reduction, ?anterior chamber paracentesis  
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Painless opacities of the lens (usually bilateral) =   cataracts  
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Dz of optic n. 2/2 abnormal drainage of aqueous humor from trabecular meshwork -> high IOP -> decreased peripheral fields / blindness =   glaucoma  
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Fundoscopy: excavated or enlarged optic disk with pallor. IOP >50 =   Acute angle-closure glaucoma  
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Glaucoma RFs   AA, Asian, Inuit; vascular dz (HTN, migraine, DM, CVD); older pts, myopia, FH  
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Open angle glaucoma Mgmt   Adrenergic blocking eye gtt (timolol, levobunolol, betaxolol), epi eye gtt, beta-agonist gtt, CAI. Laser surg. Cyclophotocoagulation.  
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Acute angle-closure glaucoma mgmt   Decrease IOP via laser; then pilocarpine (after Eye Dr). Drops (timolol, dorzolamide, brimonidine), systemic acetazolamide, isosorbide, IV mannitol. Laser peripheral iridotomy.  
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Hirschberg light reflex test is used to dx:   strabismus (esotropia or exotropia)  
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If abnormal nystagmus on exam, order:   electroretinogram to r/o retinal pathology  
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Unilateral progressive vision loss; painless central vision loss (periphery remains intact) =   macular degeneration  
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macular degeneration, neovascular (wet/exudative) type: characterized by:   confluent, large soft drusen; more rapid loss of vision  
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macular degeneration, atrophic type (dry): characterized by:   bilateral gradual & progressive vision loss  
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macular degeneration mgmt   Stop smoking; exercise; dark green veg & omega 3 FAs; avoid sunlight  
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macular degeneration Dx testing   biomicroscopic funduscopic exam; fluorescein angiography to confirm wet age-related MD  
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Increased intracranial pressure causing swelling of the optic disk =   papilledema  
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papilledema etiologies   meningitis, encephalitis, pseudotumor cerebri, cerebral trauma / ICH, tumor, abscess  
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papilledema funduscopic exam   tortuous retinal veins, optic disk retinal hemorrhages, swollen & hyperemic optic disk  
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orbital blowout fx: upward gaze diplopia signifies entrapment of which muscle?   inferior rectus  
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Medical conditions with higher risk for hyphema (due to elevated IOP)   SCD or anemia  
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treatment of hyphema   Shield eye (no patch), bedrest (with b/r privileges), elevate head of bed to 30 degrees, topical atropine, no aspirin/NSAIDs, consider topical steroids, monitor intraocular pressure  
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infectious keratitis tx   Broad spectrum antibiotic drops  
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endophthalmitis tx   Injection of intravitreal antibiotics (empiric vancomycin and ceftazidime) or surgery ASAP  
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orbital cellulitis mgmt   Emergent CT of the orbits and sinuses, ophthalmologic consultation and admission for cefuroxime IV  
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Mgmt of ruptured globe   Call ophthalmologist immediately. Metallic eye shield, first gen cephalosporin, antiemetic (prevent Valsalva), tetanus update, CT to look for foreign body.  
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How long after the first 2L of irrigation fluid should you wait to check the pH in an eye that has suffered a chemical burn?   5-10 minutes  
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Use to decrease pressure if the IOP is >50 mmHg   Acetazolamide IV  
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Use to decrease IOP if it does not do so with first line agents after 1 hour   Give 1-2g/kg mannitol IV  
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Once IOP is below 40 mmHg in acute angle closure glaucoma, what can be given as long as the patient has an intact lens in place   Pilocarpine drops  
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Tx if there is a strong suspicion of giant cell arteritis   Admit pt for methylprednisolone 250 mg IV every 6 hours  
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chalazion =   inflammatory condition 2/2 foreign body reaction to sebum from meibomian glands  
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Corneal abrasion   severe pain/photophobia; fluorescein: abrasion stains deeper green; tx polymyxin-bacitracin ointment, analgesics  
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glaucoma tx   pressure to <16; prostaglandins (latanoprost); topical BB or CAI; laser trabeculoplasty  
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int/ext hordeolum   internal: meibomian glands; ext: zeis  
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hordeolum tx   topical Abx (bacitracin or erythro)if w/ blepharoconjunctivitis; systemic if preseptal cellulitis (keflex/aug)  
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chalazion =   inflammatory condition 2/2 for body rxn to sebum from meibomian glands  
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Corneal abrasion   severe pain/photophobia; fluorescein: abrasion stains deeper green; tx polymyxin-bacitracin ointment, analgesics  
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hordeolum =   infxs process in eyelid; usu staph aureus  
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glaucoma tx   pressure to <16; prostaglandins (latanoprost); topical BB or CAI; laser trabeculoplasty  
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Homonymous hemianopia with central sparing =   lesion at occipital lobe  
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