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the central language mechanism and its disorders

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Answer
perisylvian zone   broca's area, wernicke's area, supramarginal gyrus angular gyrus and arcuate fasciculus  
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thalamus   involved in naming and memory, primary language area. Massive fiber tracts connecting to brocas and wernickes  
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border zone   area just beyond broca's and wernicke's  
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aphasia   an impairment due to acquired and recent damage of the central nervous system, of the ability to comprehend and formulate language.  
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4 classifications of aphasia   cortical/perisylvian, transcortical/border zone, subcortical aphasia, nonlocalized  
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cotical perisylvian   within the perisylvian area  
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transcortical   in the border zone, Three recognized: Motor, sensory and mixed aphasias  
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subcortical aphasia   below in thalamus  
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non-localized   the exact site of the lesion is unknown, anomia  
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types cortical aphasia   wernicke's, broca's, conduction, global  
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Wernickes Aphasia can be   sensory aphasia, fluent aphasia and receptive aphasia  
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paraphasic (wernicke's aphasia)   omission of parts of words, incorrect use of correct words, use of neologisms and substitution of incorrect phonemes for correct ones  
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logorrhea (wernicke's aphasia)   speech is fluent but excessive  
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press of speech (wernicke's aphasia)   urgency  
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empty speech (wernicke's aphasia)   lacks meaningful and substantive words  
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Symptons of Wernicke's aphasia   use of jargon, comprehension is poor, repetition is poor, reading is disturbed  
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Broca's aphasia is known as   motor aphasia, expressive aphasia, disfluent aphasia  
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symptons of broca's aphasia   decreased verbal output, nonfluent, repetition is always abnormal, naming objects is poor, oral reading is poor, writing is poor  
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Conduction Aphasia   comprhension and articulation are intact, spontaneous speech is better than repetition, conversational speech is fluent and paraphasic  
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conduction aphasia looks like   mild to moderate broca's aphasia  
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Global aphasia   associated with large lesion in the persylvian area. Left side blowout  
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severe impairment of both understanding and expression of language   global aphasia  
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spontaneous improvement of global aphasia   happens in 6 months if it happens  
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person is mute or or uses repetitive vocalizations   global aphasia is marked by...  
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transcortical motor aphasia   lesion is anterior or superior to broca's area, marked by dysfluency and effort in conversation  
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transcortical sensory aphasia   lesion is found deep to and posterior to Wernicke's area  
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fluent aphasia marked by paraphasias, reading comprehension, writing and naming are poor, symptoms are like those of Wernicke's aphasia except they can repeat   transcortical sensory aphasia  
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subcortical aphasia   left thalamic lesion, marked by verbal paraphasias and neologisms  
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reduced vocal volume, aspontaneity in oral expression and word finding deficits with frequent perseveration (get stuck)   subcortical aphasia  
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anomic aphasia   fluent but word finding difficulties (anonmia) occurs in dementia. can be stressed induced  
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progressive aphasia   adult onset degenerative language disorder, language deteriorates slowly over an extended period of time, anomia early sign  
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alexia   inability to comprehend the written or printed word  
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literal alexia   inability to recognize letters  
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verbal alexia   letters are recognized but words are not  
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pure alexia   reading disorder without a writing disorder  
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alexia without agraphia   loss of the ability ot read, retained ability to write, lesion is in the dominant parietal lobe  
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alexia with agraphia   total reading disorder with limited writing ability often two lesions  
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agraphia   inability to normally produce written language lesions are in the left frontal or parietal lobe  
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agnosia   disorder of recognition due to damage to cortical sensory association areas  
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visual agnosia   inability to recognize objects, colors and pictures  
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auditory agnosia   inability to comprehend speech and or nonspeech sounds  
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tactile agnosia   inability to recognize objects by touch  
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Broca's area   motor programming for articulation  
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motor strip   activation of muscles for articulation  
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arcuate fasiculus   transmission of linquistic information to anterior area from posterior areas  
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wernicke's area   comperhension of oral language, decodes question, encodes the answer  
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angular gyrus   association area that integrates visual auditory and tactile informations and carries out symbolic integration for reading  
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39   angular gyrus  
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40   supermarginal gyrus  
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supermarginal gyrus   symbolic integration for writing  
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corpus callosum   transmission of information between hemisphere  
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subcortical areas   thatlamus, limbic lobe, basal ganglia  
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middle cerebral artery   covers the whole perisylvian artery  
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myoneural   lower motor lesion  
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spastic paralysis   upper tract  
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flaccid paralysis   lower tract  
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too much tone   upper tract  
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not enough tone   lower tract  
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upperlesions   in tracts (corticobulbar or cortical spinal)  
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lower lesions   are in nerves  
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reflex arc   sensory information comes in, mortor information comes out  
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babinski   thumb on baby's foot, toes curl. no babinski normal for adults  
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upper motor neuron disorders   spastic paralysis, hypertonia, hyperefexia  
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clonus, babinski sign, little or no atrophy, no faciculations   upper motor neuron disorders  
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lower motor neuron disorders   flaccid paralysis, hyporeflexia, no clonus, no babinski  
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hypertonia, marked atrophy, fasciculations   lower motor neruon disorders  
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atrophy   diseningration of the muscle  
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fasiculations   little twitches when a muscle dies  
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stroke   upper motor lesion  
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ms   lower motor lesion  
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Created by: dbrinker