Alcohol
Help!
|
|
||||
|---|---|---|---|---|---|
| What are the the manifestations in someone with a blood alcohol of .150%? | Major impairment of physical and mental control. Slurred speech, blurred vision, lack of motor skill
🗑
|
||||
| What are the the manifestations in someone with a blood alcohol of .400%? | Unconsciousness, threshold of coma
🗑
|
||||
| What are the the manifestations in someone with a blood alcohol of .600%? | Death from respiratory failure
🗑
|
||||
| What is the treatment for acute alcohol intoxication? | is aimed at symptomatic support (treating symptoms of bradycardia, bradypnea, and hypothermia)
🗑
|
||||
| Why do you administer thiamine, folic acid and vitamens to patients who are chronic abusers of alcohol? | chronic ingestion will lead to decreased absorption of folic acid, thiamine, niacin, and other vitamins
🗑
|
||||
| How does chronic ingestion of alcohol cause liver damage? | Chronic ingestion leads to induction of hepatic microsomal enzyme system and other enzyme systems. Chronic excessive use leads to fatty changes, infiltration, hepatitis, and hepatic cirrhosis and other biochemical changes
🗑
|
||||
| How does chronic ingestion of alcohol affect the endocrine system? | alcohol produces a diuretic effect, a decrease in testosterone levels, an increase in estrogen levels and an increase in corticotropin secretion
🗑
|
||||
| What is Wernicke-Korsakoff syndrome? | a brain disorder involving loss of specific brain functions caused by a thiamine deficiency which commonly accompanies habitual alcohol use or alcoholism.
🗑
|
||||
| What are the symptoms of FAS? | CNS dysfunction (low IQ, microencephaly), prenatal growth retardation, cluster of facial abnormalities, limb abnormalities brain and anatomical defects
🗑
|
||||
| What is the major cause of death with methanol ingestion? | slow metabolic acidosis produced by formic acid
🗑
|
||||
| Antabuse (disulfiram) MOA? | Inhibition of aldehyde dehydrogenase (which is the most important enzyme for in vivo metabolism of ethanol) leads to increased blood levels of acetaldehyde d/t can't convert to acetic acid. Causes signficant N & V.
🗑
|
||||
| What is the MOA of alcohol? | a. Stimulates GABA receptors-increasing GABA mediated inhibition (mediates sedative and ataxia effects)
b. May also block NMDA (glutamate receptor-excitatory NT) which causes blackouts
🗑
|
||||
| Naloxone MOA in alcohol abuse? | decreases craving for ethanol
🗑
|
Review the information in the table. When you are ready to quiz yourself you can hide individual columns or the entire table. Then you can click on the empty cells to reveal the answer. Try to recall what will be displayed before clicking the empty cell.
To hide a column, click on the column name.
To hide the entire table, click on the "Hide All" button.
You may also shuffle the rows of the table by clicking on the "Shuffle" button.
Or sort by any of the columns using the down arrow next to any column heading.
If you know all the data on any row, you can temporarily remove it by tapping the trash can to the right of the row.
To hide a column, click on the column name.
To hide the entire table, click on the "Hide All" button.
You may also shuffle the rows of the table by clicking on the "Shuffle" button.
Or sort by any of the columns using the down arrow next to any column heading.
If you know all the data on any row, you can temporarily remove it by tapping the trash can to the right of the row.
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.
Normal Size Small Size show me how
Normal Size Small Size show me how
Created by:
psfisher50
Popular Pharmacology sets