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Stack #33803

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Question
Answer
Herniation of intracrainal contects   Encephalocele  
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Only herination of meniges   meningocele  
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Herination of Meninges and brain   memingoencephalocele  
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Meninges,brain,and venticular   meningohydroencephalocele  
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Defect in posterior neuropore   myeloschisis  
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Myeloschisis always results in   Spina Bifida  
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Spina Bifida   failure of vertebral arches to form and cover spinal cord  
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Spina pinda bifida occulta   skin closed over it--unseen  
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Spinda Bifida aperta   skin not closed over it  
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As with occipilta encephoceles,   a cystic mass may accompany spina bifida  
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Caudal eminence   a cell mass cadual to neural tube, and enlarfes and cavites  
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Cudal eminence corresponds to   Secondary neurulation  
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What Secondary Neurlation process joins neural tube   caudal eminence  
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Defect of Secondary Neurulation   Myelodysplasia  
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Myelodysplasia chacteristics   usually covered with skin, and marked with unusal pigmentation, hair, telangiectases or dimple  
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Tethered cord syndrome   where the conus medullaris and filum terminale are fixed to vertebral column  
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Tethered cord syndrome causes   loss of sensation from the legs adn feets and problems with bladder control  
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3 Primary Brain Vesicles   1 Prosencephalon 2 Mescencephalon 3. Rhombencephalon  
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Where cervial flexure is   Rhombencephalon-Spinal cord region  
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Cephalic flexure   2nd bend in neural tube @ mesencephalon  
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Secondary Brain vesicles   5th---3 primarys dived into 5, and 2 more flexures  
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Secondary Brain vesicles   1. Pontine Fleuxure, and 2. Telencephalic flexure  
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Main structure of forebrain develop   2nd month of gestation  
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Why abnormalitis of forebrain associate with facial defects   mesoderm during 2nd month forming facial features  
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Central induction   process of froebrain development  
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End of 5th week Telencephalon gives rise to   2 lateral expansion called Telencephalic vesicles  
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Diencephalon develops into   Thalamic nuclei AND optic cup  
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Prosencephalization   where prosencephalon develops into diencephalic and telencephalic vesicles  
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Holoprosencaphly   failure of prosencephalon to undergo cleavage  
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Alobar holoprosencephaly results in   no lobes develop, most severe large single forebrain ventricles, thalamus poorly developed, and many structures are missing  
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Hyprotelorism   close set eyes  
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Cyclops   only sinlge midline eye  
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End of 1st trimester spinal cord is   fully formed  
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Ancencephaly results in   the brain not forming, skull may be absent, and facial abnormalities  
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Types of Defects of Primary Neulation   encephalocele, myleoschisis  
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Telangiectases   large superficial cappillaries  
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Anterior neuropores closes   4th week  
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Pontine flexure divides   divides MESO-into Myelencephalon, and metencephalon (rostral)  
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After pontine flexure what does not divide further   Mescencephalon  
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Telencephalic flexure divides the   Proencep--into diencephalon, and telencephalon (rostal)  
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Telencephalon expands into   complex lobes, and b/c largest part of brain  
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The Telencephalic VESICLE are adult derivatives of   cerbral cortex, internal capsule, ofactory bulb and tract, postions of balsal ganglia, and the amygdala, and hippocampus  
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Optic cup forms   optic nerve and retina  
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Week 10--   major structures of CNS are recognizable  
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Immature versions of all strcutres are present by   end of 1st trimester  
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Most infants born with holoprosencephaly also have   facial abnormalities  
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Semilobar holoprosencephaly   some separtion of forebrain into 2 lobes, and partial develop of falx cerebri  
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Disease that has rudimetary but enlarged lateral and 3rd ventricles   semilobar holoprosencephaly  
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Telencephalic vesicle is AKA   cerebral hemisphers  
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The diencephalon and cerebral hemispheres forms @   end of week 5  
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