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UCI Physio Test 1

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
equation for how fast the membrane potential can change   dV/dt=I/C  
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equation for how much charge needs to be stored in the membrane to maintain a given membrane potential   V=Q/C  
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C is proportional to what   s/d so dV/dt=Id/s  
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V is proportional to what   Qd/s  
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hyperkalemia   high levels of potassium in serum  
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hypokalemia   low levels of potassium in serum  
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nernst equation for potassium   V(k)=(RT/z(k)F)ln(K(o)/K(i))  
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nernst equation in usefull form for all ions   V=(62/z)log(ion(o)/ion(i))  
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what is the current when Vm=Vion   0  
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electrochemical driving force   V(m)-V(ion)  
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what is G(ion)   conductance of the membrane for that ion G(ion)=NP(o)gamma (N is number of channels, P(o) is the % of channels open, and gamma is the conductance of a single channel  
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I(ion) equation   I(ion)=G(ion)[V(m)-V(ion)] or replace G(ion) with NP(o)Gamma  
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general equation for V(m)   V(m)=[Sum from i to n] G(i)V(i)/[Sum from i to n]G(i)  
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Na/K ATPase pumps what ratio   3 Na in for 2 K out  
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what sets the V(resting) for potassium   leak channels  
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digoxin and ouabain   cardiac glycosides pump inhibitors and increase heart contractility  
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palytoxin   very toxic; binds to Na/K ATPase pump and locks it allowing free Na K transported  
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halothane and isoflurane   general anesthesia gases that activate K+ leak channels, making neurons less excitable  
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lidocaine and procaine   local anesthetics; block Nav channels  
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propofol (divrivan)   short-acting hypnotic agent blocks Nav channels; used to induce and maintain general anasthesia  
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antiepileptic/anticonvulsant drugs   inhibit Nav channels (phenytoin and carbamazepine)  
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antiarrhythmic drugs   block delayed rectifier Kv channels (Dofetilide)  
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TTX and STX   tetrodotoxin and saxitoxin; paralytic toxins; block Na channels  
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channelopathies   diseases caused by altered fxn of ion channel (genetic or acquired)  
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synaptotagmin   Ca++ sensor that links Ca++ channel activation to vesicle fusion  
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BoNT and TeNT action   cleave the SNARE complex so that vesicle fusion can't occur  
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AchE   degrades Ach  
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drugs that treat dementia in patients with Alzheimer target what   AChE  
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Sarin   nerve gas that inhibits AchE causes paralysis  
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ionotropic   ion channel directly gated by the ligand neurotransmitter  
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metabotropic   receptor that, upon neurotransmitter binding, initiates an intracellular signaling cascade  
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nicotinic AchR   postsynaptic receptor at NMJ; made of 5 subunits- 2 alpha, beta, gamma, delta; both alpha sites must be bound for opening  
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reverse potential equation for the ESPS   V(esps)=((Gamma(Na)/Gamma(K))V(Na) + V(K))/((Gamma(Na))/(Gamma(K))+1)  
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equation for Gamma Na/Gamma K   GammaNa/GammaK=(Vepsp-Vk)/(Vna-Vepsp)  
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MEPP   miniature end plate potential which occur randomly  
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probablility of NMJ firing from one AP   100%  
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probability of CNS firing from one AP   low  
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curare   non-depolarizing muscle relaxant that blocks nAChR  
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myasthenia gravis   autoimmune disorder from antibodies that block nAChRs; treated with AchE inhibitors  
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type I CNS synapses   glutamatergic (excitatory)  
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type II CNS synapses   GABA (inhibitory)  
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