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Antihyperlipidemics

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Question
Answer
Atorvastatin (lipitor)   HMG CoA reductase inhibitors; high potency; CYP3A4 substrate; standard for most dyslipidemic , post MI and diabetic pts  
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Fluvastatin (lescol, lescol XL)   HMG CoA reductase inhibitors; lowest potency; CYP2C9 substrate; standard for most dyslipidemic , post MI and diabetic pts  
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Lovastatin (mevacor, altoprev)   HMG CoA reductase inhibitors; CYP3A4 substrate; standard for most dyslipidemic , post MI and diabetic pts  
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Pravastatin (pravachol)   HMG CoA reductase inhibitors; NOT a CYP450 substrate = least # of interactions; standard for most dyslipidemic, post MI and diabetic pts  
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Rosuvastatin (crestor)   HMG CoA reductase inhibitors; most potent (use for pts that need significant reduction to dec side effects); 90% excreted in feces; interacts w/CYP2C9; standard for most dyslipidemic, post MI and diabetic pts  
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Simvastatin (zocor)   HMG CoA reductase inhibitors; CYP3A4 substrate; standard for most dyslipidemic, post MI and diabetic pts  
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Cholestyramine (questran) powder   Bile acid sequestrant; breaks cholesterol down into bile acid (does NOT dec cholesterol synthesis); ok in women considering pregnancy, young pts w/moderate LDL-C prob, and OK in combination therapy w/statins  
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Colestipol (colestid) powder, tablets   Bile acid sequestrant; breaks cholesterol down into bile acid (does NOT dec cholesterol synthesis); ok in women considering pregnancy, young pts w/moderate LDL-C prob, and OK in combination therapy w/statins  
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Colesevelam (WelChol) tablets   Bile acid sequestrant; *most potent and easier to administer/tolerate;* breaks cholesterol down into bile acid (does NOT dec cholesterol synthesis); ok in women considering pregnancy, young pts w/moderate LDL-C prob, and OK in combination therapy w/statin  
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Niacin (dietary supplement)   Nicotinic acid; *most effective for inc HDL-C* only drug to dec Lp(a); don't switch preparations; advise pt about OTC nicotinic acid; caution with diabetics  
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Niacin IR (niacor) prescription   Nicotinic acid; *most effective for inc HDL-C* only drug to dec Lp(a); don't switch preparations; advise pt about OTC nicotinic acid; caution with diabetics  
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Niacin SR (slo-niacin) dietary supplement   Nicotinic acid; *most effective for inc HDL-C* only drug to dec Lp(a); don't switch preparations; advise pt about OTC nicotinic acid; caution with diabetics  
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Niacin ER (niaspan) prescription   Nicotinic acid; *most effective for inc HDL-C* only drug to dec Lp(a); don't switch preparations; advise pt about OTC nicotinic acid; caution with diabetics  
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Gemfibrozil (lopid)   fibrate; greater reduction of TG in severely hypertriglyceridemics (prevents pancreatitis); treats atherogenic dyslipidemia w/nml LDL (dec non-HDL and inc HDL); inc risk of myopathy when used with statins (but can treat high LDL/atherogenic dyslipidemia)  
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Fenofibrate (antara, lofibra, TriCor, triglide)   fibrate; greater reduction of TG in severely hypertriglyceridemics (prevents pancreatitis); treats atherogenic dyslipidemia w/nml LDL (dec non-HDL and inc HDL); slight risk of myopathy when used with statins (to treat high LDL/atherogenic dyslipidemia)  
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Ezetimibe (zetia)   cholesterol absorption inhibitors; *dec LDL 18%* inhib absorption in gut/reabs from bile; dec hepatic chol stores; inc chol clearance; mono or combo w/statins (add'l 20% LDL dec); tolerated, GI effects, myopathy, don't take w/cholestyramine bc of binding  
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Omega-3 FA Ethyl Ethers (Omacor)   *dec TG 20-50%;* dec major coronary events/mortality; well tolerated, belching/dyspepsia, flu-like, prolonged bleeding time w/anticoagulants; no interactions; Asolute contra: fish allergy; Relative contra: coagulopathy or anticoag therapy  
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Omega-3 FAs (dietary supplements)   *dec TG 20-50%;* dec major coronary events/mortality; well tolerated, belching/dyspepsia, flu-like, prolonged bleeding time w/anticoagulants; no interactions; Asolute contra: fish allergy; Relative contra: coagulopathy or anticoag therapy  
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Niacin/Lovastatin (advicor)   combo therapy; **dec LDL, inc HDL;** used when LDL goal isn't met with statin monotherapy; avoid high doses of statin (side effects); Tx other dyslipidemia in addition to hi LDL; worry about rhabdomyopathies  
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Ezetimibe/simvastatin   combo therapy; **dec absorption and production of chol;** used when LDL goal isn't met with statin monotherapy; avoid high doses of statin (side effects); Tx other dyslipidemia in addition to hi LDL  
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Dyslipidemia   Hypercholesterolemias (inc LDL-C, inc non-HDL-C); Hypertryglyceridemias, HDL-C deficiency  
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What is the primary target of dyslipidemia therapy?   LDL-C  
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Normal levels for cholesterol   Normal (abnormal): TC <200 (>240); LDL <100-129 (>160-190); HDL >60 (<40); TG <150 (>200-500)  
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Therapy Options: Therapeutic lifestyle changes (TLC)   Use in combo with drug therapy to acieve **>40%** LDL reduction; diet (fiber; plant sterols; can dec LDL by 25-30%), wt reduction, inc physical activity,  
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Lipid modifying drugs   GOAL = dose for a 30-40% reduction in LDLs; HMG CoA reductase inhibitors (statins); bile acid sequestrants; nicotinic acid; fibrates; cholesterol absorption inhibitors; omega-3 FA  
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HMG CoA Reductase Inhibitors: STATINS   *most effective for lowering LDL (18-55%)* complete inhibition of rate limiting step in chol biosynth; inc expression of LDL-C receptors (less floating around for plaques); dec CHD (CRP), coronaries, stroke, mortality; stabilize plaques/prevent clots  
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Statins: adverse effects   risk of myopathy/rhabdomyolysis when used w/fibrates or nicotinic acid; inc aminotransferases (monitor LFTs for hepatotoxicity; d/c if >3xUNL); Monitor CK (get baseline; d/c if CK >10x normal)  
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Statins: absolute contraindications   pregnancy (category X); active or chronic liver disease  
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Statins: relative contraindications   concomitant use of certain drugs (ex: fibrates)  
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Bile acid sequestrants   used for LDL reduction (15-30%) NOT HDL elev; cation resins inc conversion of chol to bile acid and expression of LDL-C receptors; dec risk of coronaries and CHD deaths; GI distress; NO systemic toxicity; dec absorption of other drugs  
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Bile acid sequestrants: absolute contraindications   TG > 400mg/dL; may increase TGs more and cause acute pancreatitis  
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Bile acid sequestrants: relative contraindications   TG > 200mg/dL  
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Nicotinic acid   **Elevates HDL (15-35%)** (vit B3); inhibits hepatic secretion of VLDL-C and inc LPL activity; also dec TGs (20-50%); slows arthrosclerotic progression, dec coronary events and mortality; dec effect of oral hypoglycemics and inc myopathy when used with st  
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Nicotinic acid side effects   flushing of skin (resolve by taking it at night with aspirin); pruritis, rashes, dry skin; GI distress; hepatotoxicity (use smaller initial dose); hyperuricemia/gout; glucose intolerance/hyperglycemia  
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Nicotinic acid: absolute contraindications   chronic liver disease, severe gout  
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Nicotinic acid: relative contraindications   hyperuricemia, diabetes, peptic ulcer disease  
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Fibrates (fibric acid derivatives)   **Dec TG (20-50%);** unknown MOA; inc LPL activity; inhibits lipolysis and dec hepatic FA uptake; inhibit hepatic secretion of VLDL-C; GI symptoms, chol gallstones, hepatitis, myopathy (w/statins: gemfibrozil); binds ptns (displaces drugs; ie: warfarin)  
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Fibrates: absolute contraindications   severe renal or hepatic disease  
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Fibrates: relative contraindications   gallbladder disease or biliary cirrhosis  
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Cholesterol absorption inhibitors: absolute contraindication   hypersensitivity to ezetimibe  
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Cholesterol absorption inhibitors: relative contraindication   moderate to severe hepatic impairment  
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