Vascular disease, cardiomyopathies, rhumetic fever, and tumors
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| Where do berry anurysms occur | cerebral vessels
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| What can vascular disease be secondary to | injury or autoimmune response
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| True aneurysms have complete or partial wall thickness | full
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| Where is the most common place for an AAA to occur | between renal arteries and bifurcation of iliacs or in illiacs
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| what age and gender has the highest risk of AAA's | men over 50
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| Which stage of syphlysis causes aneurysms and where do they occur | tertiary, in ascending aorta
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| In what age range and gender do aortic dissections occur | men 40-60
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| At what size do anuerysms become extremely dangerous | 5cm
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| Which aoritc dissection is more common and where oes it occur | Type A, involves ascending aorta(Type B, distal to subclavian)
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| Pain radiating to the back and downward is indicative of what | aortic dissection
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| Does giant cell arteritis occur more in women or men | women
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| What is the most common cause of giant cell arteritis | unknown
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| focal granulomatous inflammation of the small and medium vessels of the head are indicative of which pathology | giant cell arteritis
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| headache and facial pain with possible blindness | giant cell arteritis
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| Which vessel disorder responds to steroids | giant cell arteritis and takayasu
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| early non-specific mononuclear cell infiltrate, medial fibrosis, and sometimes granulomas | Takayasu Arteritis
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| Where does Takayasu Arteritis occur | medium to large arteries; aortic arch and major branches
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| Which disease can causea loss of pulse in the upper extremeites | takayasu
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| Disease of young adults showing association with Hep B antigen | Polyarteritis Nodosa
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| Which disease is treated with immune suppresants | polyarteritis nodosa
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| segmented fibrinoid necrosis of arterial wall with nuertophilic infiltrate | polyarteritis nodosa
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| Where can polyarteritis nodosa NOT occur | lung
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| Which disease remebles PAN, but occurs in the small arteries | Microscopic Polyangitis
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| Seen in infants and children; fever, rash, lymphadenopathy, oral/conjunctival erythema | Kawasaki
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| Etiology of Kawasaki | unknwon (but lots of immuno-problems)
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| What is the triad of Wegner Granulomatosis | 1. Focal necrotizing vasculitis of lung and upper airway2. Necrotizing granulomas of upper and lower respiratory tract3. Necrotizing glomerulitis
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| C-ANCA antibodies | Wegner Granulomatosis
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| Most common in smokers | Thromboangiitis Obliterans
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| Cause of Thromboangiitis Obliterans | unknown
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| segmented thrombosis, acute, chronic, inflammation of small and medium arteries AND viens in extremeties | Thromboangiitis Obliterans
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| Which diease is secondary to athlertoslerosis, SLE, scleroderma or Buerger disease | Raynuads
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| What are vericose veins secondary to | intralunminal pressure
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| who gets varicoities more, women or men | women, especially the pregnant
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| where are most thromboplebitis and phlebothrmoviosis likely to occur | deep leg viens, caused by CHF, neoplasia, prgnancy, postop, immobilization, and local infection
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| What is Trousseau Syndrome | moving thrombophlebitis - multiple venous thrombi appearing and disappearing - hypercoaguability?
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| Superior/Inferior Vena Cava Syndrome | Superior - secondary to neoplasisms compressing the vessel INferior - same cause but neoplasms are renal or hepatic
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| What are the clinical sugns of Superior/Inferior VC syndrome | cyanosis and/or edema in the areas behind the blockage
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| Which organism is responisble for lymphaginitis | B-hemolytic strep
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