CV Pathology: Leech Lectures
Help!
|
|
||||
|---|---|---|---|---|---|
| What is the most common congenital heart malformation | ventricular septal defect
🗑
|
||||
| What is the second most common congenital heart malformation | atrial septal defect
🗑
|
||||
| True or False: Congenital heart disease is caused solely by genetic influence | False: Strong correlations exist between siblings, but enviornmental factors still come into play
🗑
|
||||
| Why is thalidomide important | known teratogen, causes congenital heart problems
🗑
|
||||
| When is the critcal window in development | 3-8 weeks
🗑
|
||||
| Which type of shunt is more common L -> R or R -> L | L -> R
🗑
|
||||
| Which shunt would cause cyanosis and emboli from venous ciculation to enter systemic circulation | R -> L
🗑
|
||||
| What is the danger behind a L->R shunt becoming R->L | Signifigant pulmonary hypertension can develop and be IRREVERSIBEL
🗑
|
||||
| What are the number one cause of CHILDHOOD heart disease | shunts
🗑
|
||||
| What cardiac myopathy woudl cause digital clubbing and blood deficencies | Shunts
🗑
|
||||
| True or False: Obstructive myopathies usually cause cyanosis | FALSE
🗑
|
||||
| What are the three atrial septal defects in order of occurance | Secundum (Foreman ovale), Primum (Low in septum), Sinus Venosum (where SVC enters)
🗑
|
||||
| Which type of defect is usually silent until adulthood | Atrial Septal L-->R shunts
🗑
|
||||
| Ventricular septal defects are often associated with what other abonormality | tetralogy of Fallot
🗑
|
||||
| Where do 90% of ventricular septal defects occur | in the membranous material next to the aortic valve
🗑
|
||||
| True or False: 85-90% of PAD occurs as an isolated incident | true
🗑
|
||||
| What is a common presentation in a L-->R shunt that is finally picked up in adulthood | Right HEart failure or pulmonary hypertension
🗑
|
||||
| Which defect has a machine like sound to it | PDA
🗑
|
||||
| What are the requirements/components of Tetrolagy of Fallot | Ventircular septal defect. Overriding Aorta. Right ventricular Hypertrophy. Pulmonic Stenosis
🗑
|
||||
| What component of the Tetrology of Fallot determines severity | Pulmonary Stenosis
🗑
|
||||
| In Great Vessel transposition, what is the main determining factor of prognosis | How well the R. Ventricle can pump and whether there is sufficent PAD occuring
🗑
|
||||
| What myopathy occurs when the spiral ridges fail to decend | trunctus arteriosis
🗑
|
||||
| What always accompanies a trunctus arteriosus | Venticular septal defect
🗑
|
||||
| What malformation cuases one vessel to recieve blood from both ventricles | trunctus arteriosis
🗑
|
||||
| With which R-->L shunt disrder do you have r. ventricular hyoplasia and cyanosis from birth | Tricuspid Atresia
🗑
|
||||
| cardimegaly due to chronic pressure in the left ventricle can cause what | Coarctation of the aorta
🗑
|
||||
| Where do most coactations in the aorta take place | ust distal to the ductus or ligamentum arteriosus
🗑
|
||||
| When coarctation develops here, it is usually asymptomatic unless severe | postductal
🗑
|
||||
| Does collateral flow around a coarctation that is post ductal usually develop | yes
🗑
|
||||
| What can occur around a coarctation that can lead to or predispose someone to athlerosclerosis | Turbulent blood flow
🗑
|
||||
| In what condition would you see right ventricular hypoplasticity with an ASD and PDA | Pulmonic valve atresia
🗑
|
||||
| True or False: Neonates are capable of surviving a complete aortic valve atreia | FALSE
🗑
|
||||
| Which type of shunting takes place in a truncus arteriosus | R-->L
🗑
|
||||
| What must be present for a trunus arteriosus to occur | VSD
🗑
|
||||
| What must be presnet for survival in a transposition of Great Vessels | VSD or ASD with PSA (R-->L shunting)
🗑
|
||||
| Ishemic Heart disease makes up what % of cardiac deaths | 80%
🗑
|
||||
| Angina, syncope and CHF are all clinical signs of what | Degenerative Calcific Aortic Valve disease
🗑
|
||||
| At what age do people with degenerative calcific aortic valve disease have symptoms | 70-80
🗑
|
||||
| What valve is prone to NOT get infective endocarditis when it becomes clacified | Mitral
🗑
|
||||
| Which valve, when calcifed, will present with regurgitation with secondary arthymias | Mitral
🗑
|
||||
| Prolase of the mitral valve may be secondary to abnormalities in which tissue type | Marfans
🗑
|
||||
| In what age range does mitral valve prolapse occur | 20-40
🗑
|
||||
| A MIDSYSTOLIC CLICK, with dyspnea, fatigue, phsychiatic problems and chest pain may be caused by what | mitral valve prolapse
🗑
|
||||
| Elogated or ruptured chordae tendinea would be a finding in which disorder | mitral valve prolapse
🗑
|
||||
| How is endocarditis diagnosed | blood culture
🗑
|
||||
| Where do emboli from endocarditis move to | spleen, kidney, brain and heart
🗑
|
||||
| Vegetations present on one or more vlaves would clue you into | endocarditis
🗑
|
||||
| erosions or perforations are indicative of what type of endocarditis | acute
🗑
|
||||
| What organism is usually the cause of endocarditis | Staph
🗑
|
||||
| Non-bacterial thrombi are seen in patients suffering from what? | Cancer, any condtion leaving them bed-ridden with a hypercoaguable state
🗑
|
||||
| Do non-bacterial thrmobi produce inflammation or valve damage | Nope
🗑
|
||||
| mitral and tricuspid fibrinoid necrosis, mucoid degeneration, and small/fibrinous sterile vegetations on leaflets are seen in which disease | Endocarditis via SLE
🗑
|
||||
| Vegetations from SLE endocardidtis have a tendency to embolize, true or false | false
🗑
|
||||
| What are the two types of artificial valves | Mechanical and bioprothestic (animal)
🗑
|
||||
| What percentage of patients experience endocarditis due to vlave replacement | 5%
🗑
|
||||
| valve occulsion due to over growth and hemolysis are complications due to what | valve replacement
🗑
|
||||
| You suspect endocarditis, but upon examination you find fibrinoid necrosis and mucoid degeneration... what did this patient have | SLE
🗑
|
||||
| What is the recommmended HDL for males/females | > 35/45
🗑
|
||||
| Who's more likely to develop athlerosclerosis, men or women | MEN
🗑
|
||||
| The pooling/damaging effect of blood on the low pressure of a valve is refered to as... | Venturi Effect
🗑
|
||||
| Onionskin appearance | arteriolosclerosis
🗑
|
||||
| Ischemia without infarct is definitive of which disease | angina pectoris (stable, unstable, prinzmetal)
🗑
|
||||
| What are 90% of transmural MI's caused by | 90% occulsion in the coronary arteries
🗑
|
Review the information in the table. When you are ready to quiz yourself you can hide individual columns or the entire table. Then you can click on the empty cells to reveal the answer. Try to recall what will be displayed before clicking the empty cell.
To hide a column, click on the column name.
To hide the entire table, click on the "Hide All" button.
You may also shuffle the rows of the table by clicking on the "Shuffle" button.
Or sort by any of the columns using the down arrow next to any column heading.
If you know all the data on any row, you can temporarily remove it by tapping the trash can to the right of the row.
To hide a column, click on the column name.
To hide the entire table, click on the "Hide All" button.
You may also shuffle the rows of the table by clicking on the "Shuffle" button.
Or sort by any of the columns using the down arrow next to any column heading.
If you know all the data on any row, you can temporarily remove it by tapping the trash can to the right of the row.
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.
Normal Size Small Size show me how
Normal Size Small Size show me how
Created by:
lowryc