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CardioPulmonary Physiology - Final - SPC

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Question
Answer
Larynx   Transitional. Control Speech, prevent Aspiration.  
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Single Cartilages of Larynx   Epiglottis(Attaches to medial surf of Thyroid), Thyroid(ADAM's Apple) sits cephalad to cricoid and gives Larynx triangular shape. Cricoid is a complete ring and singlet shaped.  
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Paired Cartilages of Larynx   ARYTENOIDS - vocal chords attache dto vocal process@base, CORNICULATES - posterior wall of larynx on top arytenoids, CUNEIFORMS - anterior and lateral to corniculates  
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Conducting Airways UPPER   Nose, Mouth, Pharynx, Larynx  
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Conducting Airways LOWER   Trachea 1, Mainstem 2, Lobar 5, Segmental 18/19, Subsegmental, bronchioles, Terminal bronchioles  
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Conducting Airways GAS EXCHANGE   Resp. Bronchioles, Alveolar Ducts, Alveolar Sacs, Avleoli  
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Airway Landmarks   Trachea from C5-T5, cricoid to carina, Carina 21-23cm from lips, 11-13 cm long with 15-20 C shaped carts, 1.5-2.5 wide  
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A-C Membrane   Alveolar epithelium, Alveolar Basement Membrane, Interstitium, Cap Base Membrane, Cap ENDOTHELIUM  
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Lobes and Segments RIGHT   UPPER 3 - apical, posterior, anterior MIDDLE 2- Lateral and medial and LOWER 5 - Superior, Med. Basal, ant basal, lat basal, post basal  
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Lobes and Segments LEFT   UPPER 4 - apical-post, anterior, Superior Lingular, Inferior Lingular. LOWER 4 - Superior, ant. medial, Lat medial, post medial  
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Passive Inspiration Muscles   Diaphragm via Phrenic Nerve, External Intercostals  
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Accessory Muscles INSPIRATION   Scalenes, sternocleidomastoid, Pectoralis major, trapezius  
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Accessory Muscles EXPIRATION   Rectus AB, Transverse AB, External Oblique, Internal Oblique, Internal Intercostals  
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Normal Compliance Value   0.2L/cmH2O  
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Normal Specific Compliance Value   0.080  
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Normal Elastance Value   5 cmH2O/L. Low indicates disease as emphysema  
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Low Compliance Diseases   PA, PE, Pulm Fib, Atelectasis  
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P-V curve Horizontal   Horrible Compliance as in PA, PE, atelectasis, Pulm Fibrosis(Silicosis)...Little V for lots of P  
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P-V curve Vertical   Compliance goes up like Emphysems...Lots of V for little P  
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Normal Airway Resistance (Raw) Value   1-2 cmH2O/L/S High values indicate disease like ASTHMA, CHRONIC BRONCHITIS  
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Normal Conductance (Gaw) Value   0.5-1.5 L/Sec/cmH2O LOW values indicate disease like ASTHMA, CHRONIC BRONCHITIS  
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Airway Resistance Factors   Bronchospasm, Secretions, Mucosal Edema, Low Elastance, Artificial Airways  
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Poiseuille's LAW - Laminar Flow   Pr to the 4th power P=pressure, r=radius...16 fold jump in P as r decreases 50%. Radius is biggest factors affecting FLOW  
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Reynold's Number - Turbulence   2rvd/n, r=radius, v=velocity, d=density, n=viscosity. >2000=turbulent flow.  
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Normal Time Constant Value (Kt)time for lung to empty 65% Vt   .2 sec. Kt= compliance x Raw  
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Long Time Constant   2.0 = ASTHMA, CHRONIC BRONCHITIS or 4.0=EMPHYSEMA  
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SHORT Time Constant   .1 = PA, IRDS, ARDS, Pulm Edema  
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Normal STROKE Volume   60-130  
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STROKE V   CO/HR, increase HR = decrease SV, increase SVR/PVR= decreased SV(BAD), Increase Ventrical Preload or Venous return = increase SV  
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Coronary Circulation   Originates at AORTIC SINUS @ base of aorta, terminates at COONARY SINUS @ junction of SVC and R atrium  
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RIGHT Coronary Circulation   Think RPM - RIGHT, POSTERIOR DESCENDING (serves R and L ventricles), and MARGINAL R atrium and ventricle  
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LEFT Coronary Circulation   LEFT ANTERIOR DESCENDING (serves R and L Vetricles), CIRCUMFLEX ( L atrium and Vent)  
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SVR calculation   [(MAP-CVP)/CO]x80  
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Normal SVR   800-1500 dynesxsecxcm  
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Factors to INCREASE SVR   Vasoconstrictors - Dopamine, Norepi, Epi  
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Factors to DECREASE SVR   Vasodilators - Morphine, Nipride, Apresoline, Hyperstat  
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PVR Calculation   [(Mean PAP- PCWP)/CO]x80  
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Normal PVR   120-240 dynesxsec.xcm  
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Blood Chem of increased PVR   ACIDIC, Hypercapnic, Hypoxemia, fix w/PEEP CPAP, all due to constriction  
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Diseases assoc w increaed PVR   Pulm. Emboli, Emphysema, Interstitial Pulm fibrosis  
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Drugs to decrease PVR   O2, Nitric Oxide, Aminophylline causes Alkalemia  
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Capillary Fluid Balance   Hydrostatic Pressure (B/P) pushes out. Osmotic Pressure(electrolytes, Prteins, glucose, solutes) push in oin cell  
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Capillary Balance factors in Pulm Edema   Increased Hydrostatic P(CHF), Decreased Osmotic P(Ca), Increased Cap Membrane Perm(ARDS)  
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High V/Q diseases >.8   Pulmonary Emboli and Circulatory Shock = Deadspace. Responsive Hypoxemia. ABG = ALK, O2 above 100  
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Low V/Q diseases <.8   Atelectasis, PA, Pulmonary Edema = SHUNT/Refractory Hypoxemia ABG = ACID, O2 - under 100  
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Fick's Law   Diffusion over A-C Membrane, directly proportional to SA, Press Gradient, and Diffusion Coefficient...INDIRECTLY to THICKNESS  
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OxyHb curve p50   Normal 27  
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Decreased p50 LEFT   LEFT=LOAD O2 is Easy in Lung and harder to unload in tissues  
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Increased p50 RIGHT   RIGHT=RELEASE O2 in tissues but loading in Lung id harder  
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LEFT shift factors   LEFT-L-aLkalemia, decreased CO2, decreased 2,3,DPG, Hypothermia  
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RIGHT shift factors   ACIDEMIA, increased CO2, Increased 2,3,DPG, HYPERTHERMIA/FEVER, since O2 is RELEASE in tissues, that's why Fever is hot to touch  
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Total O2 delivery calculation   CO x (CaO2x10) Normal is 5 x (20x10)=1000ml...if given dl, multiply by 10  
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O2 Consumption   CO x [C(a-v)O2x10], normal = 250ml.min  
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Increased O2 consumption...common sense   FEVER, Exercise, Seizures, Shivering all >250  
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Decreased O2 consumption   Peripheral shunting, bllod not reaching limbs, Cyanide poisoning-block mitochondria from processing O2, Hypothermia-MET rate goes down, ,250  
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Normal a-v gap   20-15 = 5ml/min  
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Factors increased a-v gap   Exercise, Shivering, Fever, DECREASED CO, tissues are asking for more  
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Decreased a-v gap   Peripheral shunting, cyanide poisoning, hypothermia, INCREASED CO  
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O2 Extraction Ratio   O2ER = (CaO2-CvO2)/CaO2 NORMAL is (20-15)/20= 25%  
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Shunt Calc (QS=shunted bl, QT=total bl flow, CCO2=content O2 in Pulm. Cap . Bed)   QS/QT = (CCO2-CaO2)/(CCO2-CvO2)  
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Severity of shunt   <10%=NORMAL, 10-20%=MILD, 20-30%=MODERATE, >30%= SEVERE  
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Control Centers for Ventilation - Cerebral Cortex   Conscious Control like singing and speech  
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Control Center Vent. - DRG, VRG   DRG maintain norm breathing, VRG when exercise  
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Control Center Vent - Apneustic Center   GASP, boosts INP effort, located in caudal PONS  
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Control Center Vent. - Pneumotaxic Center   PANTING, increase rate and decrease Vt, located in cePhaled PONS  
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Peripheral Chemoreceptors   Located at Aortic Arch(VAGUS) and Carotid Sinus(glossopharyngeal), PaO2 <60 and LOW pH makes them go crazy  
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LOWEST Minute Ventilation (Ve)   LOW CO2, HIGH PO2, HIGH pH  
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HIGHEST Minute Ventilation (Ve)   HIGH CO2, LOW PO2, LOW pH  
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Vagal Reflexes 3   Pulmonary Stretch Receptors, Irritant, and "J"  
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Pulmonary Stretch Receptors   in Smooth Muscle of Conducting Airway, respond to Increased Lung V, Decreased intrapleural P...Inhibit INSP, BROCHODILATE, INCREASED HR  
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Irritant Receptors   Between epithelial cells of large conducting airway. Respond to smoke, dust, chlorine, ammonia or froeign bodies. COUGH, HYPERNEA, BRONCOCONSTRICTION, EXP GRUNT  
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"J" Receptors   in Pulmonary interstitum, respond to interstital edema and pulmonary emboli. HYPONEA, TACHYPNEA, EXP GRUNT  
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Fetal shunts 3   Ductus Venosus, Foramen Ovale, Ductus Arteriosis  
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Ductus Venosus   Umbilical chord with IVC  
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Foramen Ovale   Fetal Atria ( R and L atrium) one way valve  
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Ductus Arteriosis   Pulmonry Artery with Descending Aorta  
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Effect of Exercise Pulmonary   Increases Ve, increases diffusion capacity x 3, increases alveolar Ventilation(up to 65% of Max breathing Cap)  
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Effect of Exercise Cardio   Increased O2 consumption, Increase Extraction Ratio, Decreased SvO2, Increased a-v gap,  
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Normal Work of Breathing   0.5 joules/L  
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Increased WOB   INC Raw, LOW Gaw, LOW Compliance, LOW elastance  
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O2 cost   is the total O2 consumption of RESP muscles = less than 5%(12ml/min out of 250)  
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O2 cost increases with   INC Raw, Decrease Gaw, Decrease Compliance, Decrease Elastance...Emphysema >120 ml/min  
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Tubular Structures   Glomerulus, Bowman's Cap, Proximal Tubule, Loop of Henle, Distal Tubule, Collecting Duct  
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Monitor Renal Function   Creatinine is best indicatorn 16mg/min  
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ADH   secreted by POST Pituitary Gland, infuenced by Serum Osmolarity, increased Osmolarity triggers ADH= decreased urine ourput=Water Retention  
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Plasma Cations   Na -140, K-5, Ca-5  
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Plasma Anions   Cl-105, HCO3-24  
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Aldosterone   Adrenal hormone, releases in response to Hyponatremia, HyperKalemia, Hypovolemia, Decreaed CO...increases Na reabsorbtion and K secretion  
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