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patho.cardio.4

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Question
Answer
glomerular disease   seems to be immunologically  
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azotemia   also called uremia-elevation of blood urea nitrogen (BUN)  
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BUN   blood urea nitrogen  
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prerenal azotemia   hypoperfusion of kidneys  
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retroperitoneal   outside peritoneal cavity  
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fascia   a sheet of connective tissue (as an aponeurosis) covering or binding together body structures; also : tissue occurring in such a sheet  
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hilum   1 : a scar on a seed (as a bean) marking the point of attachment of the ovule  
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kidneys have how much blood?   20% total blood-to be cleaned  
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cortex   "bark" or "rind" [kidneys, adreanl gland, & brains have a cortex or bark around it]  
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medulla   inner portion of kidney  
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pyramids - kidney   triangular divisions of the medulla of the kidneys  
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papilla   narrow, innermost end of the pyramid  
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glycosuria   glucose in the urine  
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diabetes mellitus   (blank)  
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anuria   absence of urine  
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oliguria   scanty amounts of urine  
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polyuria   large amounts of urine  
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nephron   functional unit of the kidney  
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renal corpuscle   composed of Bowman's capsule & Glomerulus  
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uremia fibrinous pericarditis   cardiovascular manifestations of elevated BUN  
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nephritis   acute or chronic inflammation of the kidney affecting the structure (as of the glomerulus or parenchyma) and caused by infection, a degenerative process, or vascular disease  
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glomerular diseases   immune system-nephritic  
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nephritic disease   hematuria (red blood cells in urine)  
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albumin   any of numerous simple heat-coagulable water-soluble proteins that occur in blood plasma or serum, muscle, the whites of eggs, milk, and other animal substances and in many plant tissues and fluid  
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hema turia   blood in urine  
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pyurea   the presence of pus in the urine; also : a condition (as pyelonephritis) characterized by pus in the urine  
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cystitis   inflammation of the urinary bladder  
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proteinurea   the presence of excess protein in the urine  
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proteinurea   urine foaming  
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acute renal failure   oliguria (low urine) or anuria (no urea)  
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nephrolithiasis   : a condition marked by the presence of renal calculi  
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mesangium   a thin membrane that gives support to the capillaries surrounding the tubule of a nephron  
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glomular lesions   sclerotic - scarring  
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interstitial fibrosis   scarring between glomerules  
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immunoflourescence   diagnose glomular disease  
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amyloid   a waxy translucent substance consisting primarily of protein that is deposited in some animal organs and tissue under abnormal conditions (as in Alzheimer's disease)  
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nephrotic   an abnormal condition that is marked by deficiency of albumin in the blood and its excretion in the urine due to altered permeability of the glomerular basement membranes (as by a toxic chemical agent)  
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nephrotic syndrome   PROTEINUREA  
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glomerulonephritis   deposition of antigens-antibodies attack basement membrane(filter) -streptococci release  
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nephrotic syndrome   proteinurea - hypoalbuminemia-edema  
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edema   an abnormal excess accumulation of serous fluid in connective tissue or in a serous cavity -- called also dropsy  
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minimal change disease (lipoid nephrosis)   corticosteriods  
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focal & segmental glomulerulosclerosis (FSGS)   podocyte injury-progresses to complete renal failure  
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membranous nephropathy   deposits along GBM--sometimes secondary to infection-may be benign or progress to renal failur  
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membranoporligerative glomerulonephritis (MPGN)   Type I - immune complex Type II dense deposit  
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nephritic syndrome   HEMATURIA - oligurea - BUN (blood urea nitrogen) hypertension -  
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hereditary nephritis   Alport syndrome-affects eyes "port" to soul - cataracts  
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Crescentic (Rapidly progressive) glomerulonphritis   rapid loss of renal function-oliguria-bowman's capsule cells proliferate  
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Goodpasture syndrome (anti-glomerula Basement Membrane disease   Deposits of IgG  
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Immune Complex (Type II) Crescentic glomerulonephritis   complication of immune complex nephritides  
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Berger's Disease-IgA nephropathy   dr. Berger-peds dr-usually affects young children - loin pain - hematuria -deposits of IgA in mesangium-progress to renal disease  
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Alport Syndrome   eyes in Males, (not all!)-corneal dystrophy - X-linked  
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chronic glomerulitis   end-stage renal failure-hemodialysis patients have this - proteniuria  
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cysts on kidney   benign; sometimes from dialysis  
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autosomal dominant (adult) polycystic kidney disease   mutated gene, slow-acting-death from renal failure  
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medullary cystic disease   sponge kidney - or nephronophtis -medullary cystic disease complex  
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medullary cystic disease   morphology-small kidneys; tubular atrophy -renal failure in children  
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renal tumors - benign   p53 tumor suppressor gene  
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wilms tumor   proliferation of abnormal renal stem cells  
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arterial walls are thicker (2)   accommodate pulsatile flow and higher blood pressures  
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pathologic lesions in vascular tree   affect certain parts of circulation  
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atherosclerosis affects mainly   elastic and muscular arteries  
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hypertension affects   small muscular arteries and arteriolds  
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vasculitis involve   only vessesl of acertain caliber  
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vessel wall cellularity   endothelial cells (EC) and smooth muscle cells (SMCS)  
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endothelial cells extracellular matrix (ECM)   elastin, collagen and glycosaminoglycans  
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endothelial cells vessels walls 3 layers   intima, media, and adventitia  
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internal elastic lamina   dense elastic membrane in arteries (especially)  
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lamina   thin plate or flat layer (L. layer, stratum)  
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fenestrations   having windowlike openings (L. window)  
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how do smooth muscle cells in epithelium receive oxygen and nutrients?   in small vessels, directly from blood flow through fenestrations  
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vasa vasorum   in larger vessels, small arterioles supply smooth muscle cells (SMC) with oxygen and nutrients  
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arteries 3 types   Large/elastic - medium-sized/muscular - small/arterioles  
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large/elastic arteries   aorta & large branches - innominate, subclavian, common carotid and iliac and pulmonary arteries  
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small arteries and arterioles   pressure and velocity of blood flow are both sharply reduced; flow is steady rather than pulsatile -  
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what happens with even small changes in arteriolar lumen size?   blood flow is profoundly affected  
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capillaries   approximately the diameter of a single red blood cell - have endothelial cell lining but no media  
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veins   larger diameter, larger lumen, thinner walls  
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lymphatics   thin-walled, endothelium-lined channels - drain excess interstitial tissue  
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lymphatics - infection & tumor   lymphatics check for infection; also tumor cells from distant sites can be transported by lymphatics  
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intima   L. innermost  
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tunica intima   innermost coat of a blood or lymphatic vessel; consists of endothelium, thin fibroelastic subendothelial layer, inner elastic membrane  
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what happens with vascular injury?   SMC growth is stimulated; associated matrix is synthesized  
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vessel wall is injured   intimal wall will thicken  
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phenotype of neointimal SMCs   cannot contract (fewer contractile filaments) have more organelles for protein synthesis  
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arteriosclerosis   arterio g. sclerosis (hardening)  
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atherosclerosis   athero Gr. (gruel, porridge) combining form meaning greullike, soft, pasty materials  
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atheroma   lipid deposits in intima of arteries, producing a yellow selling on endothelial surface (gr. atheree, gruel + oma - tumor)  
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atherogenesis   "response to injury" hypothesis - a chronic inflammatory response of the arterial wall to endothelial injury  
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chronic endothelial injury (lesion progression)   can be due to hypertension, smoking, toxins, viruses & immune reactions - causes lesion progression because of increased permeability, laukocyte adhesion and thrombosis  
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accumulation of lipoproteins (lesion progression)   LDL and its oxidized forms - cause lesion progression in the vessel wall  
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monocyte adhesion (lesion progression)   moncytes adhere to endothelium-then migrate into intima and transform into macrophages and foam cells  
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platelet adhesion (lesion progression)    
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factor release from activated platelets   inducing SMC recruitment  
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endothelial dysfunction (2 causes)   hemodynamic disturbances and hypercholesterolemia  
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hemodynamic disturbance   plaque tend to occur at ostia of exiting vessels, branchpoints, and posterior wall of adbominal aorta, - where there are disturbed flow patterns  
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nonturbulent laminar flow   leads to induction of endothelial genes whose products protect against atherosclerosis  
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atheroprotective genes   protect against atherosclerosis - superoxide dismutase  
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nonrandom localization of early atherosclerotic lesions   where there is nonturbulent laminar flow, there is protection against plaques  
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dyslipoproteinemias   Factors affecting the circulating levels of lipids -  
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"bad" cholesterol   increased LDL  
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"good" cholesterol   HDL  
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significant correlation between LDL   total plasma cholesterol & severity of atherocslerosis  
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chronic hyperlipidemia   lipoproteins accumulate in intima-then oxidized-then ingested by macrophages-then form "foam" cells  
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oxidized LDL   important to atherogenesis - present at all stages of plaque formation  
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